PRIMARY EAR, NOSE & THROAT CARE
A TRAINING MANUAL FOR
PRIMARY HEALTH CARE WORKERS AND PRESCRIBERS
DR. DANIEL ASARE MD,MA,DLOWACS MEDICAL SUPERINTENDENT ENT SURGEON REGIONAL HOSPITAL SUNYANI
OUTLINE OF LECTURES
LECTURE 1
Anatomy, physiology,
Signs and symptoms of ear disease
Management of common ear diseases
LECTURE 2
Hearing is a Public Health and a social issue
Prevention of deafness
Causes of hearing Treatment of hearing loss
LECTURE 3
Anatomy physiology of nose and paranasal sinuses Signs and symptoms of nose and paranasal diseases Sinusitis, rhinitis, epistaxis Treatment of common nasal diseases
LECTURE 4
1.Practical session Otoscopy Rhinoscopy Technique of syringing Examination of buccal cavity 2.Radiology Interpretation of x-rays of common ENT emergencies 3.Quiz end of session exams on E.N.T Disease
LECTURE 1
INTRODUCTION TO
OTORHINOLARYNGOLOGY
FOR NURSES
THE FIELD OF OTORHINOLARYNGOLOGY COVERS DISEASES OF THE
1. Ear
2. Nose
3. larynx
4. Pharynx
5. Buccal cavity
6. Oesophagus (upper)
Most of the Ear, Nose Throat (ENT) area is
accessible to direct visualization and a good part can be examined normally and digitally. Certain structures such as pharynx larynx nasopharynx requires light for clear visualization, head lamp. Both hands are needed to expose and visualize these
structures with appropriate instruments. For
these reason a reflected light using the head
mirror and light source is used.
INSTRUMENTS
Examination of the ear can be done with otoscope
ROUTINE EXAMS
A routine examination covers the following regions: Oral and buccal cavily and oropharynx nasophrynx and posterior part of nose Hypopharynx and larynx. Nose Ears Neck
Examine as well adjacent structures. Tongue depressors are used in the buccal cavity the pharynx and posterior nasal pharynx
Nasal speculum Ear specula Nasopharyngeal and laryngeal mirrors
Cotton applicators
Tuning fork
THE EAR
A brief outline of the anatomy and physiology
ANATOMY
The ear can be divided into the following 3 parts: 1. External Ear Consisting of the pinna (Auricle),Tragus, Antetragus, External Auditory Canal/Meatus 2. Middle Ear 3 Ossicles - Incus - Malleus - Stapes
Eustachian Tube Connects The
Middle Ear To Pharynx Mastoid Air Cells
Facial Nerve
Muscles (Stapedius/Tensor Tympani)
3. INNER EAR:
Consisting of the three semi circular canals The Cochlea The vestibula containing the utricle and saccule
Nerves connecting the inner ear to the central
nervous system.
THE EXTERNAL EAR
Pinna External Auditory Canal The outer third of the external auditory canal is cartilagenous and the medial 2/3 bony. Average length (31m and 25mm respectively). The canal has a slight curve, directed forward and a bit downward in the Adult.
In a child the curve is more in a
forward direction. Hence to visualize
the ear drum one must pull the pinna
upward and a bit backward in the adults whereas in the child the pinna
needs to be pulled backward and
slightly downward.
The Cartilagenous part of the ear canal contains hair follicles and sebacious and ceruminous glands and hence is a site for furunculosis and other skin disease. WAX is an essential part of the ceruminous glands.
It has Bacteriocidal - Anti fungal
- Water proof properties
And also serves as a fly paper for flies. Dont enthusiastically clean
your ear you will be predisposing the
ear to infections and insection.
QUESTION In case insects enter some ones
ear what will you do at home?
The pinna is highly visualized. So
if it is cut off it can survive when sutured back even after 48 hours.
THE MIDDLE EAR Starts with the Tympanic membrane which reflects the situation in the middle ear cavity. The three ossicles
Incus Malleus Stapes
Are joined in an ossicular chain. The
stapes footplate covers the oval
window. The tympanic membrane
has two parts the parts tensa and flaccida.
Attached to the tympanic
membrane is the handle of malleus.
Flaccida has two layers one
devoid of fibrous layer. Tensa has three layers.
The middle ear is connected to the
Nasopharynx by way of the Eustachian
tube approximately 38mm in length
consist of lateral third which is bony and
medial two thirds which Cartilaginous.
The Eustachian tube is normally closed and opens only during swallowing and yawing or pronouncing koka, kuku etc
THE INNER EAR
Situated in the petrous part of the temporal bone and consist of three semicircular canals One horizontal Two vertical The vestibule containing the utricle and saccule and the cochlea. The Cochlea contains the organ of corti which is the organ of hearing. The semicircular canals is responsible for signal charges in position of the head balancing.
THE FINAL PART
Nerves connecting the central nervous
system from the inner ear through the internal auditory canal. The Center of
Hearing is the Auditory Cortex.
PHYSIOLOGY
THE PHYSIOLOGY OF HEARING
Sound is conducted through the Ear
Canal, the tympanic membrane and the
ossicles to the cochlea. From here
impulses are carried in the Cochlear nerve
and its Central Connections.
The Ear Canal the tympanic membrane
and the ossicles form the conductive
component of hearing and the cochlea
and its neural connections from the
sensory neural mechanism of hearing.
Conductive hearing loss
Sensoneural hearing loss
Mixed hearing loss
THE PHYSIOLOGY OF BALANCE
The sense of position is obtained from several sensory mechanisms: visual, vestibular and proprioceptive. The three are important for maintenance of balance. Loss of one can be compatible with adequate maintenance of posture. Loss of two greatly impairs posture and locomotion. The cerebellum is closely related to the vestibular system. Vertigo spinning result from loss of vestibular function.
SIGNS AND SYMPTOMS OF EAR DISEASE
Otalgia - Primary - Secondary Otorrhea/Otorrhagia Tinitus Hearing loss Vertigo Facial paralysis Itch Congenital deformities
Otalgia or painful ear, results from
Involvement of ear structures or
In the absence of ear disease, pain referred from other structures (referred otalgia)
REFERRED OTALGIA
Parts of the ear and several structures on the head and neck have a common source of sensory supply GTVF C2, C3 The commonest sources of referred otalgia are:
1. The teeth
2. The temperas mandibular joint 3. The tongue 4. The pharynx 5. The Nasopharynx and the hypophaynx and neck.
Otalgia due to disease of the ear primary otalgia are due to
Trauma/Foreign Bodies
WAX Obstruction Otitis Externa
Otitis Media Acute
CA of Ear Canal
WAX OBSTRUCTION
The Ear Canal may be occluded with WAX and cause pain. Small amount
of wax can be easily removed with
WAX curette under direct vision. No
maneuver should be carried out
blindly in the Ear.
WAX can be syringed with tap water at body temperature. Cold or hot water will cause caloric stimulation vertigo.
Technique of syringing syringing that causes pain must be stopped. Drape patient Direct stream posteriorly
INDICATION FOR SYRINGING
WAX Obstruction
FB in Ear (inanimate objects)
Debris in Ear CONTRAINDICATION Perforation of T.M Acute Ottitis Media Chronic Ear Discharge
Sometimes WAX needs to be
softened with olive oil or glycerine warm to body temperature 3x daily for 3 days after which syringing is repeated.
Acute ottis external is the inflammation of the skin linning the external auditory canal it can be acute or chronic. Acute external ottis usually presents as a very painful ear while chronic external ottis is often characterized by itchness and discharge.
AOE
May be localized (circumserbed ottis ext. furuncle. Or diffuse otitis ext.
PREDISPOSING FACTORS
Wet ears swimmers ear Ear trauma caused by pricking of the ear
Or by use of cotton swabs or features
Or somehow as a result of systematic diseases eg. Diabetes mellitus.
ORGANISMS
1. Bacteria eg. Staphylococci Steptococci Pseudomanas
Fungi otomycosis due to
aspergillus Niger or Canada Herpetic eruptions Herpes zooster oticus Ramsay-hunt-disease
Clinical features commonest symptoms
Pain in the ear Swelling of the ear
There may be hearing loss.
Pre-or-post auricular lymph needs enlargement. Fever may be present.
ON EXAMINATION
Movement of pinna painful, tragal
tenderness, stenotic canal.
Tympanic, membrane normal, canal
red or with pus/erythema.There may be a discharge.
COMPLICATION
Extension to perichondrium as
gabbage ear in older people or
diabetic may lead to malignant
ottis, ext. destruction of bone sets in.
TREATMENT
Antibiotic Local treatment wick ribbon gauze
Topical ear drops (neomyxin
polyxin)
Not CED/Gentamycin Ear drops
Anti inflammatory drugs
Ear drops should not be kept in a
refrigerator. Some of the topical preparations have steroid base anti inflammatory Properties. In otomycosis use antifungal drugs - Thorough cleaning of the ear
In chronic otis ext the skin involvement is pronounced eg. Eczematous or seborrheic
dermatitis from irritation by the
discharge. The causative agent is pseudomonas aeruginosa.
TREATMENT
Local treatment Swabs for C/S Tropical ear, ear drops polymyxin Neomycin Cream of these topical drugs and steroid base for the skin condition. Surgery for canal stenosis
TRAUMA
Haematoma - Bony - Blow - Dress Foreign Bodies
Cutlass Cut/Bite
Suture
ACUTE OTITIS MEDIA AOM
Acute infection of the middle ear include acute viral otitis media,
acute supurative Otitis media (ASOM) acute serous
otitis media.
ETIOLOGY
Children by virtue of shortness of Eustachian tube, feeding cultures, cold Blockage of the eustachian tube exudation of serous fluid in middle ear bulging of tympanic membrane Resolution or natural spontaneous rupture discharging blood and mucous or chronic otitis media hearing loss other complications.
SYMPTOMS
Fever Pyrexia Chills Convulsion Ottalgia Cattarrh Discharging Ear Mucoid General Malaise
- All signs of Malaria except few.
OTOSCOPIC FINDINGS
Reddened Tympanic Membrane Bulging and injected tympanic
membrane
Ruptured T.M with perforation
Canal normal
In ASOM Mucopurulent discharge
TREATMENT
Depending on stage
Myringotomy
Antibiotic Nasal decongestant Analgesics Clean pus
Treat other diseases adenoids sinusitis.
CHRONIC OTITIS MEDIA/CSOM
Persistent of discharge on/off from 6
weeks onwards to years
SYMPTOMS Tinitus
Hearing loss
Discharge off/off
FINDINGS
Perforation in T.M central or peripheral with or without pus. Very difficult to treat Antibiotics Antihistamines Keep ear dry Clean ear of discharge Tympanoplasty reconstructive surgery
COMPLICATIONS OF AOM/COM
Meningitis Otitis hydrocyphalus Chronic otitis media Serous otitis media Deafness Facial nerve paralysis Cerebellar asscess Lateral sinus thrombophlebitis Temporal lobe abscess larbyrinthitis
LECTURE 2
HEARING LOSS
May be a symptom of its own
or associated with tinitus, vertigo (triad of symptoms meneres).
A.
TYPES OF HEARING LOSS
Conductive Sensomanual Mixed hearing loss The auditory and vestibular nerves (VIII) are intimately related to CN VII. In the internal auditory meatus
B.
Acquired Congenital H.L
The following can cause hearing loss
Lesions of the external auditory canal
Congenital atresia
WAX, foreign body
Otitis ext
Trauma Tumours Stenosis Exostosis
LESION OF THE MIDDLE EAR
AOM, COM, CSOM, ASOM Trauma
Tumors
Glue ear The above two produces conductive H.C
The following lesions produce sensoneural hearing loss
Familial
Congenital
Presbycusis
Noise induced H.L
Ototoxic drugs Head injury labyrinthitis
Commonness infectious disease cause hearing loss leading cause in Ghana
Meningitis CSM Measles Febrile Convulsion
Parotitis unilateral H.L
CSOM
MANAGEMENT OF H.L
Examine ext, middle ear
Text of hearing turning fork test.
Audiometry Treat disease
Rehabilitation. Hearing Aid
Cochlea implant
OTOTOXIC DRUGS
Aminoglycosides antibiotic Quinine Salicylates With some drugs the damage is corrected if withdrawn early others progresses. A patient with renal failure on Aminoglycoside is susceptible to ototoxicity Hearing loss is usually bilateral and symmetrical.
NOISE INDUCED H.L
Acoustic trauma sudden exposure Nose induced H.L gradual over a long period in a noisy environment.
Noise damages the cochlea hair cells can be due to
sudden, sharp, laud noise or from prolonged exposure to noise. Eg. Rifle fire, expolosion or blast. Prolonged noise occurs in industrial setting Airports, Sawmills, Dickos
PREVENTION OF DEAFNESS/TREATMENT
EPI Primary prevention Secondary prevention Use of ear protection
Minimizing industrial and domestic noise
Screening with hearing test for all such workers
School screening to detect early treatable causes
New born screening
Hearing AIDS are valuable in the
treatment of many types of Hearing loss
Surgical treatment for many types
Hearing AID has a Microphone, an amplifier, and receiver.
FACIAL PARALYSIS
LMN paralysis of all half of face UMN intact emotional movements
Bells palsy
CSOM as a complication Ramsay hunt disease
TINITUS
Noise
Ototoxic
Laribynthitis
WAX
Treat cause
Prevent offending cause
Maskers of noise
PRESBYCUSIS Sensoneural H.L due to the aging
process is referred to a
presbycusis.
LECTURE
3
THE NOSE AND PARANASAL SINUSES
The nose and the paranasal sinuses lie in the
upper part of the upper respiratory tract. The
paranasal sinuses connect with the nose
through various ostia. The nose is continuous
posteriorly with the nasopharynx and is
connected to the eustachian tube and the
middle er by way of the nasopharynx.
The anterior and middle cranial
fossa, the orbit and the roof of the mouth together with the
teeth are closely related to
certain parts of the nose and the sinuses.
The function of nose and paranasal sinuses are
Cosmetic
Upper part of respiration
Makes (the sinuses) the head lighter
Take part in resonance Nose filters, warm air Olfaction
THE NOSE
The external nose it formed by bones and cartilages. The anterior and posterior apertures of the nose are called anterior and posterior choans, respectively. The lateral wall of the nose contains the opening of the paranesal sinuses. It is marked by three turbinates. The interior (independent bone), the middle and superior conchae or turbiantes.
The area below the turbinate is called
meatus. The nasolacrimal duct opens into the inferior meatus. The middle
meatus contains the openings of the
frontal, maxillary and anterior enthmoid sinuses.
The superior meatus contains the
opening of the posterior ethmoid
cells. The sphenoid sinus opens
posteriorly in an area called the splenoethmoidal recess. The
mose contains olfectory cells and
nerve.
BLOOD SUPPLY
The turbinates are erectile tissues. The nasal septum contains many blood vessels and is called littles area a
frequent spot for epistaxis.
There is an area around the nose
known as the danger zone. Where internal carotid and external carotid branches meet and any small infection can result in
extension of infection to the brain
or cavernous sinus.
SYMPTOMS DUE TO NOSE INVOLVEMENT
Nasal obstruction
Nasal discharge increased
Loss of smell anosmia Sneezing excessively
Symptoms due alteration of the nasonator of the nose NASAL
speech.
Dryness to crust formation Pain in the nose
Nose bleeding
Trauma: Fracture Nasal Bone
SINUSE SYMPTOMS
Halitosis Sinus headache Tumours of sinus Symptoms of nose and sinus disease can be a part or a manifestation of systematic disease. For example epistaxis can be due to a bleeding diatesis nasal and sinus allergy may occur on a patient with bronchial asthma.
Polyp formation in a child may be due to cystic fibrosis: facial
and nasal deformity and asymmetry may be to congenital or familial.
Read about common cold/coryza
Pharyngitis Acute chronic pluritis
Unilateral offensive smell in a child is a foreign body in the
nose unless proven otherwise.
EPISTAXIS
Nose bleed is common. Minor instances are easily treatable or
controlled at home.
Bleeding is unilateral, or bilateral anterior or posterior.
CAUSES
Local and systemic causes local attributable to nose and its structures Trauma Nose prick to littles area
FB
Tumors
Infection Rhinitis, Sinusitis
Vicarious Menstruation Congenital - teleangioectasia
GENERAL CAUSES
SCD
Bleeding diathesis Leukemia Arterial hypertension Climatic condition such as harmattan
Altitude
The common cause is nose prick at littles area.
MANAGEMENT
At home pinch nose for 5 mins, sit upright apply ice pack. IN THE HOSPITAL FIND CAUSE Canterize bleeding part littles area
- Chemical
- Electrical Cantery
Anterior Nasal Packs with Gauze
Treated BIPP (Vaseline Gauze)
Posterior Packing
Catheter in Posterior Nasal Space Sedation + Rest
Replacement of Blood Loss
Ligation of Vessels
ACUTE/CHRONIC SINUSITIS
Acute sinusitis can involve all the sinuses in one or both sides pansinusitis all. Abology often 20 nasal infection following acute viral infection. catarrh
PREDISPOSAL
Dusty environ
Excessive dryness Instillation of concussion
BACTERIAL INFECTION
Henophilis
Influenza
Stephylococci
Sometimes fungi
SYMPTOMS
Feeling of fullness on the side of face Dull headache
FINDINGS
Tenderness
Hyperemia over affected sinus
Pus under the meatus in the nose X-ray shows opacity in the sinus
TREATMENT
Treat infection
Decongest nose Most will resolve if note
Anthral lavage - AWO is scheduled six
weeks later
Frontal Trephination Chronic sinusitis from
1. Unresolved Acute Sinusitis or recurrent Sinusitis. Duration 6 weeks or more.
SYMPTOMS
Nasal obstruction Halitosis
Post nasal drip
Headache
FINDING
Polyps Caries tooth (if chronic maxillary sinusitis)
Allergy
X-ray - opacity
TREATMENT
Antibiotic Decongestant
Antilustamine
Anthral Lavege
Intranasal Anthrotomy
Coldwell Luc Operation
COMPLICATION OF SINUSITIS Can come from acute of chronic sinusitis 1. Orbit involvement leads to proptasis,
Ostcitis Osteomylitis
Mucocele
Orbital Cellulitis Oroanthral Fistula Intracranial Spread Cavenous Sinus Thrombosis Chronic Pharyngitis, Laryngitis
LECTURE 4
LARYNX AND
PHARYNX
LARYNX
The Larynx forms the lower part of the upper respiratory tract. Apart from the gradual increase in size as childhood progresses, the major change in the Larynx during adolescence the anterior posterior length of the glottis increases by approximately 1cm in males and 3mm to 4mm in females.
The former increase accounts for the voice change in adolescent males. The thyroid cartilage forms the central and anterior walls of the larynx and produces the prominence in the neck referred to as the Adams Apple. Paired cartilages Thyorid Cricoid, Epiglotis. Form the framework of the larynx.
THE NERVE SUPPLY OF THE LARYNX
Significant nerve supply of the
larynx is derived from the
superior and recurrent laryngeal nerve.
FUNCTIONS OF THE LARYNX
Sphincter action: prevents entry of food and drink into trachea during deglutation
Passage for air
Voice production defecation and parturation
SYMPTOMS OF LARYNGEAL PATHOLOGY
Hoarseness Stridor Nerve Paralysis Asphyxia
Causes Inflammation
Acute Laryngitis
Ltb
Acute Epigloltitis Laryngeal Dyptheria
CHRONIC
Non specific Polyp Singers node
Chronic laryngitis
Hoarseness of voice in an adult of more than 4 weeks is cancer until proven otherwise.
Laryngea paralysis can give rise to Hoarseness of voice respectively difficulty and aspiration of liquids or solids into the trachea and bronchial tree.
Opening into upper trachea as a result of airway obstruction. INDICATION respiratory obstruction
TRACHEOSTOMY
To bypass obstruction
Lung toileting Reasons accumulation of secretion in tetanus Ventilation for assisted respiration in coma Poisoning
TYPES
Emergency Elective Intubation
THE PHARYNX 3 PARTS
Oro, Naso, Hypo Pharynx Tonsils Adenoids disappear in adolescents Waldeyers ring formed by palatine tonsils adenoid (pharyngeal tonsil) ligual tonsils and submuscosal follicles
FUNCTIONS OF PHARYNX Deglutition
Speech Airway
Taste
SYMPTOMS
Sore Throat FB throat Tonsillitis Common cold
Leukemia and tumour
diptheria
DYSPHAGIA CAUSES
FB Tumour
Infection
Trauma Ulceration
SORE
Tonsillitis Inflammation of tonsils Bacterial infection
Beta haenolytic streptoco
SYMPTOMS
Fever, Malaise, Odynophagia, Ottalgia
TREATMENT
Antibiotic Analgesic
COMPLICATIONS
Peritonsillar Abscess (Quinsy) Parapharyngeal Space Infection Chronic Tonsillitis Aom Glomerulonephritis Pericarditis Rheumatic Fever
SURGERY
TONSILECTOMY
Absolute indications Repeated attack 3 x a year Rec. tonsillitis Huge tonsils causing airway obstruction Snoring History of quinsy
ADENOIDECTOMY
If adenoids so hypertophied, that causing airway obstruction or feeding problems. Snoring and sleeping apnea.
1. Practical Exercises
Otoscopy
Radiology Interpretation
Turning Fork Test Discussion
2. Quiz