Approach to emergency management of a stroke patient
(Prof Kakaza tutorial)
I. Resuscitation (ABC)
a. Airway and breathing
i. LOC | GCS < 8 Intubation
ii. Aspiration risk
NGT
NPO until speech therapist/OT tests them
b. Circulation
i. If compromised:
IV line with normal saline
a. HT is the most common cause of stroke therefore
sodium is not good for the patient
No dextrose or Maintelyte
Monitor glucose in DM and place on sliding scale
NO GLUCOSE!
Metabolism Penumbra ischaemic Anaerobic respiration Lactic acid
accumulates Damage to cell Oedema
II. History & examination
a. History in keeping with a stroke
b. Examination
i. Neurological
NB Power
Gives vascular distribution/global picture
ii. Cardiovascular
Pulses (rate, rhythm and volume)
NB to feel all pulses because of pulselessness that can occur
in a random fashion Common in females with autoimmune
disease Takayasu
iii. Blood pressure (usually )
</= 220/120 Monitor
a. Want to keep it high to ensure brain perfusion
b. Can be part of cushing reflex response to raised ICP
i. Raised BP
ii. Irregular breathing
iii. Bradycardia
> 220/120
a. IV Labetolol OR
b. Captopril (ACE-I) stat dose
c. NB Do not use Nifedipine
iv. Respiratory
Aspiration risk
Fever/infection detrimental to penumbra
a. If raised Panado
III. CT scan
a. Ischaemia (more common) vs. haemorrhage
i. No CT scan available Give everyone aspirin
b. If ischaemic infarct < 4.5 hours TPA 0.9mg/kg
i. If no TPA available Aspirin 300mg stat | 150mg daily
c. If haemorrhagic
i. Decrease BP, no medication
IV. Admission
a. Treatment (script)
i. Stroke prevention
Anti-platelet
a. Aspirin/Disprin
Anti-hypertensive
Statin (regardless of cholesterol, to stabilise the plaque)
ii. Clexane +/- 3 days later
iii. Definitive anti-hypertensive treatment
Day 4
a. ACE-I (promote vascular remodelling)
b. HCTZ (diuretic) to all patients
iv. Rehabilitation (ASAP) to promote plasticity
Physio
OT
Speech therapy