Protocol Manualfinal 2011 Word
Protocol Manualfinal 2011 Word
PROTOCOL MANUAL
2011
1
TABLE OF CONTENTS
U
SECTION 1: INTRODUCTION
Introduction 6
SECTION3: ED PROTOCOLS
ED Triage Algorithm 45
Termination of Resuscitation 46
ED Resuscitative Thoracotomy 47
I: Airway
ED Rapid Sequence Intubation 50
II: Head & Face
Indications for Head CT 56
Initial Management of Head Injury 57
Severe Closed Head Injury Algorithm 60
Facial Bone Fracture 61
III: Spine Evaluation
Spine Evaluation and Clearance 62
Cervical Spine Clearance Algorithm 63
Initial Management of Spinal Cord Injury 64
Critical Pathway of Removal of Backboard and Cervical Collar 65
IV: Blunt and Penetrating Neck Trauma
Blunt Cerebrovascular Injury Algorithm 66
Penetrating Neck Injury Evaluation 67
Penetrating Neck Algorithm 69
V: Thoracic Trauma
Blunt Cardiac Injury 70
Blunt Cardiac Injury Algorithm 72
Blunt Injury to the Thoracic Aorta 73
Blunt Thoracic Injury with Suspected Injury to Thoracic Aortic Arch
Or Arch Vessels 77
Suspected Thoracic Aortic Injury Algorithm 78
Penetrating Mediastinal Wound Algorithm 79
VI: Blunt and Penetrating Abdominal Trauma
Thoraco-Abdominal Stab Wound Algorithm 80
Diagnostic Evaluation of Blunt Abdominal Trauma 81
Blunt Abdominal Trauma Algorithm 83
GSW to Abdomen, Flank, or Low Back Algorithm 84
Evaluation of Genitourinary Trauma 85
Genitourinary Trauma Algorithm 86
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VII: Pelvis
Pelvic Fracture Radiologic Evaluation Algorithm 87
Pelvic Fracture Treatment Algorithm 88
VIII: Blunt and Penetrating Extremity Injury
Traumatic Peripheral Vascular Injury 89
Extremity Blunt Trauma Vascular Injury Algorithm 90
Penetrating Extremity Vascular Injury Algorithm 91
Open Long Bone Extremity Fracture Protocol 92
IX: Miscellaneous
Management of Injury in Pregnancy 93
Initial ED Management of the Pregnant Trauma Patient Algorithm 95
ED Evaluation of Burn Patients 96
Burn Service Care Algorithm 99
Burn Adult Resuscitation Algorithm 100
Massive Transfusion Protocol 101
Trauma Early Glutamine Administration & Enteral Feeding Algorithm 102
The Anti-Coagulated Trauma Patient 103
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Revised 10/08
Preface
The Faculty and Staff of the Trauma Program at the University of Kentucky Hospital are pleased to
present the new Trauma Protocol Manual 2011. This latest revision has been updated and
expanded to include more resource material relevant to the variety of disciplines involved in the
care of the multi-injured trauma patient. The UK Trauma Program strives to deliver timely and
effective care to the injured patient utilizing evidence-based guidelines and protocols. This manual
outlines expectations and standards of care appropriate for Level 1 Trauma Center designation.
To further enhance trauma care internally and in our service region, the Trauma Protocol Manual
will be published on-line via the Department of Surgery Website, the Trauma Program Website and
the University of Kentucky CareWeb. These sites may be accessed at the web addresses listed
below. New and/or updated protocols/guidelines will be posted on the website. The UK Trauma
Program recommends periodic review of the site to monitor for latest revisions. The
protocols/guidelines are formatted so that they may be downloaded and printed. Hardcopies are
available in the Trauma Program Office (H213) or by calling 859-323-5022. Faculty and house staff
may obtain CD version by contacting Trish Cooper (3-5037) in the Trauma Program Office.
Please direct inquiries to Lisa Fryman, RN, Trauma Nurse Coordinator/Program Manager at 859-
257-1231 or [Link]@[Link]. Specific protocol discussion may be directed to Faculty by
calling 859-323-6346.
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SECTION 1: INTRODUCTION
BLUE SURGERY (TRAUMA/EMERGENCY) SERVICE
INTRODUCTION:
The Blue Surgery (Trauma/Emergency) rotation is sponsored by the Division of General Surgery,
Section of Trauma and Critical Care. The length of the rotation depends on the year of post-
graduate training. The rotation is designed specifically to provide all residents with experience and
didactic knowledge in comprehensive care of the injured adult (> 15 years of age) and adults
requiring emergent general surgical intervention.
ROTATION OBJECTIVES:
3. Provide clinical experience, instruction, and knowledge in the management of critically ill
patients.
ROTATION REQUIREMENTS:
A. Trauma Patients*
The majority of trauma patients are admitted via the Emergency Department (ED). There will be
direct inter-facility transfer of injured patients from referring the referring hospital to the ICU or floor.
Occasionally, direct admits to the OR will bypass ED evaluation. Trauma patients can present as a
referral from another hospital and physician, direct from the accident scene via helicopter (scene
call), or unannounced by ground ambulance from Fayette County or the surrounding county EMS.
*Refer to attached "Trauma Admission Policy"
Trauma Expect: Patients referred and accepted by the trauma service from another hospital
(ground or air transport) are considered trauma expects. Trauma expects can be referred for EM
evaluation. Unless referred for EM evaluation, the Trauma/emergency surgery residents are
immediately responsible for supervision of trauma expect patient care upon that patient's arrival in
the ED.
Local EMS Transports: Patients transported by local EMS providers become the responsibility of
the Trauma/emergency surgery residents by one of two mechanisms:
1. Trauma consult called by the ED
2. Trauma Alert* called by ED.
*The Trauma Alert system is discussed below under a separate heading.
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Initial assessment and evaluation of the multiply injured patient should proceed according to ATLS
protocol. A review of your ATLS provider manual is highly recommended. Resident roles and
responsibilities during the initial evaluation are outlined in the attached documents. Role
assignment is pre-designated depending upon experience, skill proficiency, and resident knowledge
base. The chief surgical resident in house (PGY4 or PGY5) assumes responsibility for the timely
evaluation, management, and disposition of the trauma patient. This responsibility also includes the
timely notification of the attending physician. Patient disposition should be determined within 60
minutes of ED admission. The entire diagnostic evaluation/disposition should not exceed 120
minutes. Should it become obvious at any point during the initial evaluation that the patient will
require surgical intervention, it is imperative that the OR be contacted immediately. A surgical
resident will accompany hemodynamically unstable patients outside the ED for all diagnostic
procedures (i.e., CT scan, angiography, etc.). Physicians are not required to accompany "stable
patients". It is the responsibility of the ED nursing staff to insure that all trauma patients will be
accompanied by an RN during procedures done outside the ED. There is a policy that governs the
RN responsibilities for transport.
Trauma Alert
A trauma alert will be called based on the outlined mandatory and/or potential criteria (see attached
document). Patients receiving a trauma alert may be arriving via ground ambulance, air medical
transport, or could be present in the ED and experience an acute deterioration in condition.
2. Trauma Labs
There is a document outlining laboratory values that will be ordered when ordering trauma labs. A
trauma panel is available. The labs ordered are based on the severity of the injuries. Blood Alcohol
and urine drug screens are mandatory. Any questions regarding the necessity for additional lab
values should be clarified with the chief surgical resident and communicated to the nurse. Refer to
policy on trauma labs in the ED Policies/Procedures Section.
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3. Documentation
The trauma admission form, upon completion, will be added to the medical record. This precludes
writing an admission narrative H&P. The original goes to the patient's medical record. The yellow
copy should be placed in the Trauma Coordinator's mailbox in the general surgery corridor. Any
missing trauma admit forms are the responsibility of the chief surgical resident on-call that day. Any
trauma admission form submitted incomplete will be returned to the chief resident for completion
within 24 hours. Missing data elements will be noted for completion. THE TRAUMA ADMIT FORMS
SHOULD BE COMPLETED IN LEGIBLE ENGLISH.
Documentation should not stop with the completion of the trauma admission form. Any and all
significant changes in patient condition while in the ED should be documented completely and
legibly in the medical record.
Daily Census
A daily census will be the responsibility of the off-going chief resident and his/her team. Updated
census information should be complete for morning rounds. All patients admitted to or consulted by
the service should be represented on the census. ED and OR mortalities should be listed on the
weekly M&M list.
Procedure Documentation
All procedures (deep lines, chest tubes, arterial lines, intubation, DPL, LP, cutdown, etc.) should
have a procedure note completed in SCM in detail.
b. Procedure notes should be completed for all procedures regardless of whether the
attending is present or absent. Procedures such as Intubation, bronchoscopy,
Groshong catheter removal, suture of lacerations, etc. should be documented. These
procedure notes are used to provide necessary and complete documentation in the
medical record for procedures performed.
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c. All procedures performed in the ED by trauma service residents should have a note
completed. There has been some confusion about procedures performed in the
Emergency Department after hours and on weekends. The supervising attending
physician for emergency department patients is the attending surgery physician
listed on the call schedule not the blue surgery attending on the service. There are
occasions when the blue service attending is present after hours and on weekends
and should be listed as the supervising physician. The latter circumstances should
be obvious.
A brief written note should appear in the progress notes that documents the procedure and
indicates that a more detailed note will follow. For all procedures the following information must be
provided:
Name: Diagnosis:
Reg Number: Indication:
Date of Procedure: Resident Surgeon:
Location: Attending Surgeon:
Service: (performing the procedure) Preparation:
Anesthetic:
1. A referral from another hospital and physician. *Trauma/Emergency Surgery residents are
immediately responsible for supervision of general surgery referrals accepted from another hospital
upon that patient's arrival in the UK ED.
* Patients accepted in transfer by other general surgery services (Green and Gold) or Green and
Gold patients that present in the ED for evaluation are and remain the primary responsibility of the
Green or Gold Surgery service chief resident.
2. A consult from the ED attending. The Trauma/Emergency Surgery service is responsible for the
evaluation of ALL general surgery UK ED** consults.
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**The Trauma/Emergency service should and will evaluate all ED general surgery consults. After
6:00 am and before 5:00 pm, it is permissible to triage appropriately to other general surgery
services (Green and Gold) but only after appropriate evaluation and reasonable diagnostic
possibilities have been established. The Blue surgery attending must approve the transfer. The
triage or transfer of service should be arranged between the chief surgical residents and/or between
service attendings not between junior house officers.
During regular working hours (8:00 am to 5:00 pm, Monday through Friday), all ICU surgical
consults and in house UK emergency consults (including emergent Kentucky Clinic consults) are
the responsibility of the Trauma/Emergency Surgery service. After regular working hours and on
weekends, in-house UK or in-house VA emergency general surgery consults and VA ED consults
are not the responsibility of the Blue Surgery service residents. These patients are the responsibility
of the General Surgery ESS resident. The only exception to these rules is elective general surgery
consults directed specifically to one of the Blue (Trauma/Emergency) Surgery attendings.
The chief surgical resident in house (PGY4 or PGY5) assumes ultimate responsibility for the timely
evaluation, management, and disposition of all general surgery emergency patients. This
responsibility also includes the timely notification of the attending physician.
NOTIFICATION OF CONSULTANTS
Consultant(s) evaluation is frequently required for the complete evaluation and treatment of the
multiply injured patient. Timely consultant notification and patient evaluation are necessary to
minimize emergency department length of stay and to insure high quality patient care. The Section
of Trauma and Critical Care has established the following guidelines. We expect the
Trauma/Emergency Surgery service residents to adhere to these guidelines. Consultants should be
notified promptly following completion of the secondary survey (<20 minutes after patient arrival) or
sooner if their services are required (acute neurosurgical, face team, cardiothoracic, or orthopedic
intervention). Consultants should respond to a page within 10 minutes. Consultants should be
present for patient evaluation within 20 minutes of notification. Consultation should be performed by
an upper level resident (PGY2 or higher) or faculty. Interns should not be notified for ED patient
evaluation unless all other members of the consultant team are involved in priority patient care that
precludes their presence.
physician. Admitting office notification can be accomplished in one of two ways: 1. You may give a
verbal order to the ED nurse caring for the patient; or 2. You may enter the information directly in
the computer. Do not call admitting because this is time-consuming and inefficient!
Call coverage teams will consist of Senior Surgical Resident (PGY 4 or 5), a midlevel surgery
resident (PGY2 or 3), and an intern. Night call and work hours will conform to the ACGME work
hours and night call standards. During some months additional senior, midlevel and first year
residents (EM, Pulmonary, Anesthesia, OB/GYN, PM&R, Family Medicine) will rotate on the
service. These additional resident resources will be integrated into the service to provide additional
coverage in compliance with ACGME work hour standards.
After accepting patients for transfer*, the chief surgical resident will notify ED triage and relay the
following information on all expects:
1. Patient name/age/sex
2. Referring Physician and Hospital/ETA
3. Residents to be paged on arrival and pager number
4. Patient history/chief complaint
5. Specific diagnostics/interventions to be prepared
*The air medical dispatcher will contact ED triage for all patients being transported by the air
medical service.
Referring physicians should be treated in a polite and courteous manner. REMEMBER THAT THEY
ARE ASKING FOR OUR ASSISTANCE. MANY REFERRING HOSPITALS DO NOT HAVE THE
RESOURCES TO CARE FOR THESE PATIENTS. ALL patients referred by an outside physician
are to be accepted in transfer by the chief surgical resident unless otherwise instructed by the
Trauma/Emergency surgery service attending on call (NO EXCEPTIONS).
B. ICU Call
Primary ICU calls for Blue Surgery Service patients are the responsibility of the PG2 or PG3 on call.
The Critical Care Nursing Staff have been instructed to direct all calls to the PG2 or PG3 on call.
Interns receiving ICU calls will refer them to an upper level resident.
C. Floor Call
Primary floor calls are the responsibility of the Blue Surgery Intern on call. Questions or problems
regarding floor patients should be directed to the chief surgical resident on call.
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Patients rounds should occur twice daily on all Trauma/Emergency Surgical Service patients and
consults.
A. Morning Rounds
The chief surgical resident assumes primary responsibility for the timing and conduct of rounds. In
general, daily morning rounds begin at 6:00 am (7:00 am on weekends) in the Trauma ICU unless
otherwise notified. Residents from each call team are to be present for rounds. Given that this is not
always possible; a resident from each of the call teams should be present so that information
transfer occurs in an orderly fashion. The intensivist (Surgical Critical Care) residents will make AM
ICU rounds with the Blue Surgery Service.
B. Attending Rounds
Daily attending rounds will arranged between the faculty on service and the senior surgical
residents. Residents should present patient information in a clear, concise, and detailed format
facilitating the completion of rounds in a thorough but timely fashion.
A. Hospital Discharge*
Patient discharge from the hospital should be timely and efficient. This process is facilitated by
discharge planning rounds. Timely and cordial interaction with the nursing staff that provide
discharge teaching and with the social worker who arranges extended care (i.e. Nursing Home,
Rehabilitation, Coma Center, Home Health, etc.) is critical. The Trauma Case Manager will be
responsible for coordinating discharge of the multiply injured patients. When patients are identified
for discharge, the nursing staff should be notified on the day prior to discharge. Discharge orders
and prescriptions will be completed the evening before or by 8:00am on the day of discharge. The
following critical errors are often made in patient discharges:
1. Patients are not scheduled for subspecialty appointments prior to discharge (i.e.
Neurosurgery, Orthopedics, ENT, Plastics, etc.)
2. Patients are not given adequate supplies or medication**. This is poor patient care, results
in unnecessary patient calls, and is unfair to the patient and their family. Please make sure
that patients are given adequate medication and supplies to make it to their first clinic
appointment. Pre-printed prescriptions should be used.
3. Appropriate labs and X-rays are not being ordered for the first clinic visit.
4. The first blue surgery clinic appointment should be scheduled 2-4 weeks following discharge
unless otherwise specified. Not all patients require a Trauma/Emergency Surgery service
clinic appointment. Please check with chief surgical resident, attending physician, or case
manager before scheduling a follow-up appointment.
5. The attending physician of record for the discharge summary is the attending on service
when the patient is discharged from the hospital.
*Please refer to the attached discharge documents and policies in this manual.
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**Trauma service residents will not prescribe narcotic pain medications after hours or on
weekends. Patients should be instructed to contact the clinic during regular working hours
for narcotic pain medications.
V. Clinic Responsibility
VI. OR Scheduling
The Trauma/Emergency Surgery service has elective operating room time every Thursday and
Friday. Cases must be booked no later than 24 hours prior to Surgery. Do not wait until the last
minute to book elective cases. The chief surgical resident is responsible for checking, verifying, and
establishing case order. This should be accomplished along with the surgical attending on service.
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A. Emergent
The booking of emergent surgical cases is the primary responsibility of the chief surgical resident.
B. Elective*
The booking of elective surgical cases on Trauma/Emergency Surgery inpatients is the primary
responsibility of the chief surgical resident. *The clinic personnel must be notified about elective
inpatient booking so they can keep our schedule correct.
*Scheduling for clinic patients is covered under clinic responsibilities.
PERSONNEL
Trauma Nurse Coordinator (TNC)
Name: Lisa J. Fryman, RN
Phone: 859-257-1231
Pager: 330-6421
The TNC is responsible for program administration, quality assurance activities, and systems
problem solving. The TNC maintains and facilitates the trauma registry for the purpose of research
and quality assurance. Trauma admission forms should be forwarded to the TNC. The TNC also
coordinates ATLS and other education related activities. Clinically, the TNC evaluates quality of
care, especially during the initial assessment and resuscitative phase.
TEACHING CONFERENCES
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LEGAL RESPONSIBILITY
Many trauma patients become involved in civil or criminal cases. Consequently, residents often
receive a subpoena to testify in these cases. Under most circumstances, the responsibility to testify
in court belongs to the attending faculty member that supervised the case. Please bring all
subpoenas involving testimony in these cases to the immediate attention of the Chief, Section of
Trauma and Critical Care or to the Chief, Division of General Surgery. All other medical
practice/legal issues should be brought to the immediate attention of the Chief, Division of General
Surgery.
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This policy is designed to clarify protocols for trauma patient admissions to the University of
Kentucky Hospital. A major trauma patient is any patient with significant injury to two or more
systems or who, on the basis of mechanism of injury, has a high potential for injury to two or more
systems. Major trauma patients also include those with evidence of physiological compromise that
cannot be attributed to only or organ system.
All trauma patients with injury to more than one system, or who are otherwise included under the
definition of major trauma, will be admitted to the Trauma Service for a period of no less than
twenty-four (24) hours. Admission to Services other than the Trauma service does not preclude
close consultation with the Trauma Service; alteration in the condition of the patient will require
evaluation by the Trauma Service and any appropriate consultative service.
If after twenty-four hours the patient has a single system injury without injury to another system,
transfer to the appropriate service will be instituted. The Trauma Service may be requested to
function as a consultative service on the patient after transfer of the patient to another service.
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University of Kentucky / UK
HealthCare Policy #ED08-50
Policy and Procedure
Purpose: To effectively manage major trauma patients, the Trauma Alert Team must
assemble quickly and mobilize the resources essential to diagnosis and treatment.
Policy
Procedure
Persons and Sites Affected
Policies Replaced
Effective Date
Review/Revision Dates
PROCEDURE: To effectively manage major trauma patients, the Trauma Alert Team must
assemble quickly and mobilize the resources essential to diagnosis and treatment.
I. Criteria for Adult Trauma Alert
In order to ensure that critically injured patients receive appropriate medical care, the
Trauma Service has developed criteria to guide medical professionals in rendering trauma
care.
A. Mandatory Criteria
1. Trauma Alert Red
2. Trauma Alert
A Trauma Alert will be issued if a patient exhibits one or more of the following
criteria:
The characteristics of the accidents or injuries listed below indicate that patient
condition may necessitate a Trauma Alert.
Only the Senior Trauma/Emergency Physician can deactivate a Trauma Alert and dismiss
Trauma Alert personnel from the Emergency Department. The Emergency Department
Charge Nurse will document Trauma Alert deactivation on the Nursing Care Record and will
ensure appropriate documentation on the Trauma Alert Log completed by the clerical staff in
order to prevent unwarranted patient billing.
A. Notification
2. The ED PRA will call the paging operator via STAT number and instruct
him/her to issue a Trauma Alert or Trauma Alert Red supplying estimated time
of arrival (ETA) and brief patient descriptors for mechanism of injury.
Notify the Blood Bank by phone (Trauma Alert Red/Trauma Alert only)
Document all notifications in the Trauma Alert Log and denote Trauma
Alert Red or Trauma Alert.
3. When the paging operator receives instructions from the ED PRA, he/she will
activate the Trauma Alert pager system to notify Trauma Team members.
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When physicians assigned to the Trauma Team are notified that a Trauma Activation
is in effect they will:
The Trauma Service Primary Call Resident will notify capacity command center staff
of potential admission.
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In the event of a Trauma Alert Red, an operating room will be designated and held
for 30 minutes in anticipation of emergency operative intervention. The Attending
Anesthesiologist will respond to the Emergency Department within 5 minutes of
Trauma Alert Red notification to assist with airway management if necessary and to
evaluate the patient for pending operative intervention.
F. Laboratory Personnel
When the laboratory is notified by the ED clerk that a Trauma Activation has been
issued laboratory personnel will:
Expedite analysis of all trauma labs.
Call Emergency Department to report all panic level laboratory results. All other
laboratory results will be reported to the ED via the computer.
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A. The Trauma Coordinator will review all Trauma Alert Red/Trauma Alert records for
completeness of information and for details of the response efforts (trauma
indications, arrival times of Trauma Alert Team members).
B. The Trauma Coordinator will submit a quarterly report and summary of all Trauma
Alerts to the Trauma Patient Care Committee.
C. The Trauma Coordinator will present specific issues or concerns related to a case(s)
for discussion and action planning.
If the committee determines that response was sub-optimal, the Chairman will notify
the department director or service chief of the area that delivered sub-optimal
service.
D. All Trauma Alert Red activations will be reviewed utilizing the following criteria:
Formerly ED08-101
Policies Replaced
Chandler HP Good Samaritan Kentucky Children’s CH Ambulatory KC Other
Effective Date: 12/89 Review/Revision Dates: 02/10
Approval by and date:
Signature Date_______________
Name Penne Allison, RN, BSN, MSOM, Director
Signature Date_______________
Name Roger Humphries, MD Medical Director
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Potential Criteria
Age > 55 with significant mechanism of injury
Falls > 20 feet
Rollover MVC
Ejection of patient
Response/Resources activated:
Extrication > 20 minutes
Trauma Surgery Attending Motorcycle crash speed > 20 mph & separation of rider
Trauma Surgery Chief Resident Motor vehicle crash speed > 40 mph
Anesthesiology Attending Same vehicle occupant fatality
Emergency Medicine Resident Pedestrian struck by motor vehicle
ED Nurses Intrusion into vehicle > 12 inches
ED Technician Blast injury
ED Paramedics Multiple system trauma involving
more than 1 surgical specialty
Ultrasound Technologist
Radiology Technologist
CT Scan Technologist Response/Resources activated:
Respiratory Therapist
Blood bank cooler of uncross-matched blood Emergency Medicine Attending
Operating Room Charge Nurse notified Trauma Surgery Chief Resident
Operating Room made available Emergency Medicine Resident
Chaplain OB Chief Resident *if applicable
ED Nurses
ED Technicians
ED Paramedics
Radiology Technologist
Ultrasound Technologist
Respiratory Therapist
CT Scan Technologist
Blood Bank cooler of uncross-matched blood
Chaplain
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In order to ensure that critically injured pediatric patients receive appropriate medical care, the
Trauma Service has developed criteria to guide medical professionals in rendering trauma care.
A. Mandatory Criteria
2. Trauma Alert
A Trauma Alert will be issued if a patient exhibits one or more of the following criteria:
Pediatric trauma score <13
Respiratory rate <10 or >30
Glasgow Coma Scale (EMV) < 10
Any intubated trauma patient
Penetrating trauma to head
Combination of 2nd or 3rd degree burn > 15% BSA and multiple trauma
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B. The characteristics of the accidents or injuries listed below indicate that patient condition
necessitate a Trauma Alert.
A. The Trauma Service authorizes the following individuals to initiate a Trauma Activation if any
criteria are met during transport or upon arrival:
A patient who exhibits one or more criteria is scheduled to arrive at hospital <
15 minutes.
A patient who exhibits one or more criteria arrives without previous notification.
A patient’s condition deteriorates acutely while in the Emergency Department.
Only the Senior Trauma/Emergency Physician can deactivate a Trauma Alert and dismiss Trauma
Alert personnel from the Emergency Department. The Emergency Department Charge Nurse will
document Trauma Alert deactivation on the nursing care record and will ensure appropriate
documentation on the Trauma Alert Log completed by the clerical staff in order to prevent
unwarranted patient billing.
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A. Notification:
1. When authorized personnel request a Trauma Alert Red/Trauma Alert the ED charge
nurse will:
2. The ED PRA will call the paging operator via STAT number and instruct him/her to issue
a Trauma Alert/Trauma Alert Red supplying estimated time of arrival (ETA), and brief
patient descriptors for mechanism of injury, and:
Notify the Blood Bank by phone (Trauma Alert Red/Trauma Alert only)
Document all notifications in the Trauma Alert Log and denote Trauma
Alert/Trauma Alert Red.
3. When the paging operator receives instructions from the ED clerk, he/she will activate
the Trauma Alert pager system to notify Trauma Alert Team members.
The Pediatric Chief Surgery Resident and intern will respond within five minutes (15 minutes if out
of house). Because the Pediatric Surgery Chief Resident may take call from home, the Trauma
Service Chief Resident also responds to the Pediatric Trauma Alerts to provide direction of the
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resuscitation until the Pediatric Surgery Chief Resident arrives, or to provide assistance to the
Pediatric Surgery Chief Resident. Care of the patient and direction of the resuscitation will be
assumed by the Pediatric Surgery Chief Resident upon his/her arrival.
As the Trauma Charge Physician, the Trauma Chief Resident/Senior Emergency Medicine
Physician will coordinate response activity. The Trauma Service Primary Call Resident will notify
capacity command center staff of potential admission.
CT Scan technologists will respond within five minutes, supply contrast for potential scanning,
ascertain scanning needs, and ensure CT Scan and abdominal ultrasound availability for the
patient.
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In the event of a Trauma Alert Red, an operating room will be designated and held for 30 minutes
in anticipation of emergency operative intervention. The Attending Anesthesiologist will respond to
the Emergency Department within five (5) minutes of Trauma Alert Red notification to assist with
airway management if necessary and to evaluate the patient for pending operative intervention.
F. Laboratory Personnel
When the laboratory is notified by the ED clerk that a Trauma Alert has been issued, laboratory
personnel will:
A. The Pediatric Trauma Coordinator will review all Trauma Alert Red/Trauma Alert
records for completeness of information and for details of the response efforts
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(trauma indications, person who initiated alert, arrival times of Trauma Alert, and
ancillary personnel).
B. The Pediatric Trauma Coordinator will submit a quarterly report and summary of all
Trauma Alerts to the Pediatric Trauma Patient Care Committee.
C. The Pediatric Trauma Coordinator will present specific issues or concerns related to
a case(s) for discussion and action planning.
If the committee determines that response was sub-optimal, the Chairman will notify
the department director or service chief of the area that delivered sub-optimal
service.
D. All Pediatric Trauma Alert Red activations will be reviewed utilizing the following
criterion:
All Pediatric Trauma Alert activations quality monitoring results will be forwarded to the Chief
of Pediatric Trauma.
Formerly 08-23
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Criteria Criteria
Response/Resources Activated:
Pediatric Surgery Attending
Pediatric Surgery Chief Resident Emergency Medicine Attending
Trauma Surgery Chief Resident Pediatric Surgery Chief Resident
Anesthesiology Attending Pediatric Surgery Attending -notified
ED Nurses Trauma Surgery Chief Resident
ED Technicians Emergency Medicine Residents
ED Paramedics ED Nurses
Ultrasound Technician ED Technicians
Radiology Technician ED Paramedics
Respiratory Therapist Radiology Technician
CT Scan Technician Ultrasound Technician
Blood Bank cooler of uncross-matched blood Respiratory Therapist
Operating Room Charge Nurse CT Scan Technician
Operating Room made available Blood Bank cooler of uncross-matched blood
Chaplain Chaplain
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PROCEDURE:
C. Responsible for:
D. The Trauma Team Leader may assume or delegate the role of evaluation and
management depending on patient acuity.
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Emergency Medicine MD or MD #2
Primary MD or MD #3
MD #4
A. Pre-notifies physicians, primary patient care nurse, circulating nurses and financial
counselor of impending arrival.
B. Responsible for:
A. Prepares Trauma Resuscitation area prior to patient arrival, and ensures that needed
equipment/supplies are readily available.
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C. Directs nursing team members as needed and assumes overall nursing responsibility
for patient to include:
A. Responsible for:
A. Responsible for:
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A. Responsible for:
Documents Resuscitation.
Assists with crowd control.
Assists with procedures as needed.
Financial Counselor(s)
A. Pre-notifies Radiology, Respiratory Therapy, Operating Room, Blood Bank etc. prior
to patient arrival.
B. Identifies patient from EMS run sheet, Air Medical records, and/or patient’s family
and updates computer when possible.
C. Prepares lab slips, X-Ray requests, EKG requests and ID bracelets.
D. Secures patient valuables in ED safe.
Respiratory Therapy
Radiology Technologist
Ultrasound Technologist
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PURPOSE: Describe a standard set of laboratory studies obtained for each trauma patient, and
to list specific additional studies for frequently encountered situations. We
understand certain cases may require additional studies, to be obtained at the
discretion of the treating physicians.
PROCEDURE:
Lab specimen should be obtained via a femoral arterial stick in patients that are:
(a) Unstable, (b) intubated, or (c) have major mechanisms of injury. Venous samples
are appropriate for patients that are: (a) Stable, (b) non-intubated, (c) have minor
mechanisms of injury, and (d) ALL pediatric patients.
Standard Lab Panel for Trauma Alert Red and Trauma Alert Patients
* ABG Panel (ABG, Hct, Na, K, iCa, Glu)
* Type and crossmatch (4 units packed red blood cells)
* Coags (PT, PTT, INR)
1. Altered mental status is defined as GCS ≤ 14, not readily explained by pre-hospital
medication administration or shock.
2. Female trauma patients without history of prior hysterectomy (Refer to ―Evaluation and
Management of Injury in Pregnancy,‖ located in the protocol manual).
____________________________________ ________________________________
Medical Director, Emergency Medicine Trauma Program Director
____________________________________
Emergency/Trauma Services Director
Formerly 08-103
36
- 37 -
SUBJECT: Care of Patients in Need of Immediate Surgery When the Operating Room is at
Maximum Capacity
SEE ALSO: OR Policy, OR 01-09, OR Access for Trauma.
PURPOSE: To provide criteria and procedures to manage the needs of patients who require
access to the OR at times when the Operating Room is at maximum capacity.
CRITERIA:
Patients meeting the following criteria may access the Operating Room under this policy:
PROCEDURE:
In conjunction with the OR response, the staff in the ED will respond in the following manner:
B. Remaining surgeons
1. Responsible for retrieving required carts: i.e. craniotomy, airway, chest,
abdominal or vascular depending on need.
2. Responsible for opening room, setting up sterile field, serving as scrub and
circulating personnel until OR personnel arrive.
C. Prior to entering OR room, all staff will apply head covering, mask, and shoe covers.
A. ED Charge Nurse
1. Responsible for determining need for Trauma Alert Red and for anticipating
necessity for accessing the Operating Room.
2. If aware of OR maximum capacity, assess ED staff, assignments and current
activities (if time allows).
3. Responsible for reassigning ED patients of primary patient care nurse if patient
going to OR (may also send NA if ED staffing allows).
4. Responsible for determining availability and need for additional resources.
5. Communicates need for respiratory therapist to accompany patient to OR.
6. Responsible for ensuring that blood bank delivers blood to OR.
B. Patient Care RN
1. Assumes nursing responsibility for the patient; delegates responsibility to others,
when available, to accomplish tasks.
2. The ED nurse will prepare the patient for transport and accompany the patient
immediately to the OR.
NOTE: Patients directly admitted to the OR via Air Medical Services will be
accompanied by the Air Medical staff who will continue resuscitation until relieved
by the ED nurse or OR personnel (i.e., Anesthesia).
3. The ED nurse will facilitate continued resuscitation in the OR until the call team
arrives. The ED nurse will be responsible for the following:
The ED nurse will remain in the OR until released by the anesthesiologist and/or the
arrival of the OR call team.
38
- 39 -
Quality Monitoring:
**All cases will be reviewed by the Trauma Coordinator and the Multidisciplinary Trauma
Committee.
____________________________________
Emergency/Trauma Service Director
Medical Director, Emergency Medicine
REVIEWED: 8/97, 6/00; 8/08
39
- 40 -
40
- 41 -
Purpose:
Provision of guidelines for use of the trauma/critical care nursing record will allow a standardized
method for use of the assessment tool resulting in more effective utilization and accuracy of the tool
and the data collected.
Rationale:
Effective and timely management of the multiple trauma or critically ill patient demands a rapid,
organized, systematic approach to assessment, planning, implementation and evaluation. The
trauma/critical care nursing record provides the framework for organization of the approach and
provides a documentation tool to facilitate establishment of a permanent record of comprehensive
baseline and monitoring assessment data as well as facilitating the documentation of the
implementation and evaluation phases of the nursing process. The nursing record organizes
specific data points to assist in determination of patient status and detection of potential or actual
human response to illness.
Considerations
1. The trauma/critical care nursing record is included in the patient's permanent medical record.
2. Use of the assessment tool is initiated by the nurse when deemed appropriate. The tool must
be used with all critically ill or injured patients who require ongoing monitoring and are
classified as acuity Level I or II.
3. As assessments, interventions and/or evaluations are completed the corresponding time and
information will be recorded in the appropriate area.
4. Evaluative notes will be recorded under the Nursing Evaluation section of the nursing care
record.
5. The record must be signed and initialed by all nurses delivering care to the patient including
the recording nurse. Assurance of adequate signatures to document care provision is the
responsibility of the patient care nurse.
6. A mechanism for quality assurance will be implemented to audit charts for completeness.
Staff will receive feedback surrounding quality assurance audit filters regarding form
completion.
41
Requisites
Procedure:
4. Initial Assessment
a. Complete initial assessment section by documenting the primary and secondary
survey findings. Document time assessment of system occurs in space provided.
All sections should be completed in full. If you are unable to assess a particular
area or it is not applicable document accordingly.
b. All trauma patients must have a completed Glasgow coma scores, pupil evaluation
& trauma score and on arrival and on discharge from the ED.
5. After completion of the initial assessment, shade injured areas on diagram using legend
at bottom of page.
6. Document lab draws with time, site, prep and person obtaining labs.
7. Vital Signs
a. Vital signs should have a PTA set documented. Then the frequency of the vital
signs should correspond to the acuity of the patient. Vital signs will include
Temp, BP, HR, RR, SpO2, GCS and pupils.
b. Critical Care monitoring should be documented every hour at minimum e.g. ICP,
CPP, CVP, PCWP, etc.
d. Drips will be documented at regular interval for dosage and rate or when titrated.
9. Intervention
a. Procedures - record time of procedures, size of tubes & lines placed along with
sites and removal of backboard and/or changing of C-Collar in appropriate spaces.
Other procedure data can be entered in the nursing evaluation section.
11. Rhythm strip must be posted and interpreted for all patients with chest pain or a cardiac
event.
12. Medications
Document all medication administration by entering time administered, name of med, dose,
route, your initials and ordering MD.
43
14. Transport
Patient transports to diagnostics should be documented in this space.
16. Notifications
All notifications should be documented by checking the box and inserting time notified in space
provided.
17. Signatures
All nursing staff involved in care of the patient should sign and initial the nursing care record. All
signatures should be legible.
18. Disposition
Document time report is called and last name of RN receiving report.
Time of actual patient movement from the department should be noted as well as destination of
patient. If "floor" is checked, enter room number.
19. Place patient stickers in upper right corner of patient chart on white and yellow sheets.
The white copy is to accompany the patient to the in-patient area and yellow stays in ED.
Formerly ED-07-13
44
SECTION 3: ED PROTOCOLS
45
TERMINATION OF RESUSCITATION
ON THE BASIS OF PREHOSPITAL CRITERIA
Introduction:
1. Blunt Trauma whom arrives pulseless with no signs of life (patients > 15 years old).
2. Penetrating Trauma whom arrives pulseless with no signs of life with prehospital CPR >
15 minutes (patients > 15 years old).
Signs of Life:
1. Reactive pupils
2. Spontaneous movement
3. Organized ECG activity
Trauma Alert Activation should NOT occur initially for patients meeting the pre-hospital criteria.
The Emergency Department faculty will evaluate the patients immediately and determine the
following.
1. Pulselessness - if a pulse or any sign of life are detected, Trauma Alert Activation should
occur immediately.
2. Normothermic (T > 90 degrees F)
If all the above-mentioned physical findings are verified, the patient will be pronounced DOA
and no further resuscitation activity should ensue.
The ED Faculty should ensure completion of the Trauma Admission Form in its entirety,
completing under diagnosis ―Patient pronounced DOA‖ and noting any injuries diagnosed by
gross physical examination (e.g., femur fracture, penetrating head injury, etc.).
The pink copy of the Trauma Admission Form will be forwarded to the Trauma Coordinator for
entry into the Trauma Registry. The original copy will become part of the patient’s permanent
medical record.
References:
American College of Surgeons’ Committee on Trauma: Advanced Trauma Life Support, ed.8,
Chicago, 2008, The College.
Committee on Trauma: Resources for Optimal Care of the Injured Patient 2006. Chicago, 2006,
American College of Surgeons.
Revised 11/08
46
Revised 11/08
Purpose:
Emergency Department resuscitative thoracotomy may be necessary to salvage patients who
present in extremis and may otherwise die without aggressive therapy. Emergency Department
thoracotomy is not indicated in the resuscitation of all trauma patients who present in extremis.
The following protocol is intended to be a guide and is not intended to be all-inclusive or
exclusive. Additional patients not covered by this protocol who might benefit from Emergency
Department thoracotomy will be rare and case-specific. The procedure is performed in
conjunction with other resuscitative efforts and should not be employed in isolation. Under
certain conditions, resuscitative efforts might best be accomplished in the Operating Room. An
Emergency Department resuscitative thoracotomy should only be performed by general surgery
PGY-3, or higher, level residents or attendings.
Indications:
Definitions:
Procedure:
47
and will be postero-lateral to esophagus. Dissect around aorta inferior to
pulmonary hilum and apply aortic cross-clamp.
7) Enter pericardium by longitudinally incising pericardium anterior and parallel to
phrenic nerve. This is best accomplished by grasping pericardium with forceps
and cutting with Metzenbaum scissors. Pericardial incision is carried inferiorly to
diaphragmatic reflection and superiorly to level of superior pulmonary hilum. Care
must be taken to avoid injury to left atrial appendage and phrenic nerve. This is
best accomplished by lifting tip of scissors laterally as incision is made.
8) Manually lift heart from pericardial sac. If hemopericardium present, then
examine for cardiac perforation. Teflon pledgetted 3-0 prolene suture on a taper
needle is present in thoracotomy suture pack for repair of cardiovascular
wounds. If hemopericardium is not present, then begin open cardiac
compression. Aortic cross-clamping, if not previously performed, is indicated if no
hemodynamic response is noted.
9) Additional exposure may be accomplished by extending thoracotomy incision
across sternum into contralateral chest cavity.
Reference:
48
EMERGENCY DEPARTMENT TRAUMA THORACOTOMY TRAYS
TRAY #1
QUANTITY INSTRUMENT
1 - Finochietto – Buford Rib Spreader (10‖ Spread)
1 - Tuffier Rib Spreader
1 - 6 ¾‖ Curved Mayo Scissors
2 - 9‖ Metzenbaum Scissors
1 - #10 Disposable Blade
2 - 5 ½‖ Tissue Forceps
2 - 9‖ DeBakey Forceps
2 - 8‖ DeBakey Needle holders
4 - Vanderbilt Clamps
1 - Medium Satinsky Clamp
1 - 9 ½‖ DeBakey Aortic Clamp
8 - 5 ¼‖ Towel Clamps
1 - Liston Bone Cutter Straight
1 - Lebsche Sternal Chisel
1 - Hammer
4 - Surgeons Towels
1 - Teflon Sheet 6‖ x 6‖
10 - Laparotomy Pads
SUTURE BAG
49
RSI INTUBATION
Julia E. Martin M.D.
Assistant Professor
Department of Emergency Medicine
General information:
Airway control is always the most important objective in the initial resuscitation and stabilization.
It takes the highest priority in primary assessment. The trauma team must be prepared for any
airway emergency.
RSI involves the use of neuromuscular blocking agents and sedatives to facilitate endotracheal
intubation. Rapid Sequence induction technique is used to prevent regurgitation and aspiration
of gastric contents. Requires preoxygenation and denitrogenation by using 100% oxygen via
non-rebreather face mask to prevent apnea related hypoxia during the procedure. Once
paralytic is on board, mask ventilation is not attempted at this point. During induction, a skilled
assistant provides manual in-line axial stabilization of the head while a second assistant presses
the cricoid cartilage to prevent gastric aspiration. Cricoid pressure is maintained until the cuff on
the ET tube is inflated and tube placement is confirmed. Main disadvantage is once anesthesia
has been induced there is no turning back. The only contraindication to RSI intubation is a
practitioner who is not skilled in airway management. Indication for surgical airway is the
inability to intubate the trachea. In neck trauma, intubation may be difficult or impossible and
surgical airway may be required.
Short acting agents are used to allow patient to resume spontaneous respirations and to allow
close monitoring of neurological status. Oral endotracheal intubation is usually the preferred
method. If the head and neck are stabilized by an assistant there is almost no risk of spinal
cord injury by oral tracheal intubation.
Always anticipate vomiting. Even patients, who otherwise seem relatively unresponsive, may
vomit during attempted intubation without RSI. This may result in loss of airway control and
aspiration of gastric contents. Struggling patients increase muscle activity making hypoxemia
worse and increase ICP. As a general rule, presume all trauma patient’s have just eaten. Risk
for aspiration is greatest during anesthesia induction and instrumentation of the upper airway.
This risk is minimized by applying cricoid pressure.
Patients with severe closed head injury are of major concern because intracranial pressure can
rise precipitously during intubation. Rapid sequence induction of anesthesia and oral intubation
are now recommended for patients with head injuries to minimize the rise in ICP.
Remember, rendering patient apneic, when endotracheal intubation is beyond the skill of the
operator, may be rapidly fatal.
50
Burn patients with airway involvement and inevitable airway loss
Class 3-4 hemorrhagic shock
Failure to maintain adequate oxygenation (PaO2 < 60 despite 100% FiO2)
Paralysis due to high spinal cord injury
Need for positive pressure ventilation
Blunt chest trauma with compromised ventilatory effort
Mandibular fractures with loss of airway muscular support
Evaluation:
Respiratory distress associated with trauma to the upper airway is frequently made worse by
blood or gastric contents in the airway and requires prompt action. These patients are often
combative because of hypoxia.
When evaluating an awake patient with severe facial trauma ask them if they are getting enough
air. If they cannot answer, stick out their tongues fairly easily or are hyperventilating, they
should probably be intubated. In unconscious patients, it is probably best to intubate.
Tachypnea may be subtle but an early sign of airway or ventilatory compromise. Tachypnea is
often also associated with pain and/or anxiety.
Agitated and combative patients that are not hypoxic or have a significant head injury are better
managed with Haldol 5-10 mg.
DRUGS:
Sedatives:
Versed:
Benzodiazepine
Rapid onset (1-2 min) and short duration (20 min)
Amnesic
Anticonvulsant
Muscle relaxant
Slight decrease in blood pressure and increase in pulse rate.
No increase in ICP.
Dose: 0.1 mg/kg
Etomidate:
Nonbarbiturate, nonnarcotic sedative-hypnotic induction agent.
51
Good agent in multisystem trauma patient because it evokes minimal change in
HR and CO compared to Thiopental. (ideal agent in any patient in shock
including cardiogenic and septic shock)
Decreases ICP and IOP during procedure
Rapid onset (<1 min) and short acting (5 min)
Vomiting, esp. with combined with a narcotic
Dose: 0.3 mg/kg
Thiopental:
Ultrashort acting barbiturate sedative
Dose: 3-5 mg/kg
Onset 30-40 sec
Last 10 min.
Does not cause increase in ICP but can cause severe hypotension, therefore
avoid in multi-traumatized patients.
Can also induce bronchospasm.
Fentanyl:
Narcotic
Little or no histamine release
Rarely causes hypotension
Consider in head-injured patients as a premedication to prevent increase in ICP
(blunts pressor response)
Rapid injection may cause chest wall rigidity.
Dose: 3-5 mcg/kg
Onset in 2 min with 30-40 min duration.
Paralytic Agents:
Vecuronium:
Nondepolarizing agent
1/3 more potent and pancuronium and duration of action is 1/3 to ½ as long (25-
40 min vs Pancuronium which last 2-3 hours)
Onset 2-3 minutes
Dose not cause the degree of tachycardia seen with pancuronium.
No histamine release.
Defasciculating dose: 0.01 mg/kg
Paralytic dose: 0.1 mg/kg
Succinylcholine:
Depolarizing agent, which causes muscle fasciculations which can be prevented
by pretreatment with a non-depolarizing neuromuscular agent.
Rapid onset (30-60sec) with short duration of action (5-7 min).
Dose:
Adult: 1.5 mg/kg
Pediatric (<10 y.o): 2.0 mg/kg
Contraindications:
Burns > 7 days old
Extensive crush injuries > 7 days old.
Paraplegia > 7 days old.
Narrow-angle glaucoma
52
Neuromuscular Diseases:
Guillain-Barre, myasthenia gravis, Multiple sclerosis, muscular
dystrophy, Parkinson’s disease.
Others susceptible to increased potassium:
Renal failure (no real evidence that RSI increases K+)
Rhabdomyolysis
Rocuronium:
Non-depolarizing agent
Onset < 1 min.
Duration 20-30 min.
Dose: 0.9-1.2 mg/kg
Expensive
Adjunctive:
Atropine:
Succinylcholine will cause bradycardia in infants and children therefore they
should be premedicated with atropine. Also pretreat any adult who is already
bradycardic.
Children < 8 y.o.
Dose: 0.01 mg/kg up to 0.5 mg (minimum dose of 0.1 mg)
Lidocaine:
Dose: 1.5 mg/kg
Some studies recommend intravenous Lidocaine to blunt the pressor response of
increased pulse, increased blood pressure, increased intracranial pressure,
and increased intraocular pressure associated with intubation, its
usefulness is controversial. However, because a single dose of lidocaine is
unlikely to cause harm, it seems reasonable to use in the patient who has a
known or suspected head injury.
Should be administered 2-3 min prior to intubation.
Procedure:
The 5 P’s of rapid sequence intubation:
Preparation
Preoxygenation
Pretreatment
Paralysis (with anesthesia)
Placement (of the endotracheal tube)
1. Preoxygenation with 100% oxygen for 3-5 minutes via NRB mask (or 3 vital capacity
breaths, avoid BVM if possible).
2. Secure IV’s, ECG, pulse oximeter.
3. Prepare intubation equipment: ETT with stylet, suction, BVM, laryngoscope.
4. Premedication:
Lidocaine (head injury) 1.5 mg/Kg
Vecuronium (defasciculating dose) 0.01 mg/Kg
Versed 0.1 mg/Kg
Atropine (peds) 0.01 mg/Kg
Etomidate 0.3 mg/Kg
53
5. Perform Sellick’s maneuver, maintain maneuver until after confirmation of tube placement.
6. Succinylcholine 1.5 mg/Kg (Peds: 2.0 mg/Kg)
7. Wait 30-60 sec, place ETT.
8. Confirm ETT placement by: listening for bilateral breath sounds, chest rise and fall, tube
fogging, & positive ETCO2. Final confirmation by CXR.
9. Release Sellick maneuver.
10. Secure ETT.
Basic RSI
Time (min)
-5.00 Preparation and Preoxygenation
Intubate
Intubate
54
References:
Wilson R, Walt A. Anesthesia for the Trauma Patient. Management of Trauma pitfalls and
Practice, 2nd edition. Baltimore: Williams & Wilkins; 1996: 85-90,249-250.
American College of Surgeons. Airway Management. Advance Trauma Life Support. American
College of Surgeons; 1993:47-59.
55
56
Initial Management of Head Injuries
Brain injury is the most common cause of death in trauma. Brain injury is divided into primary
and secondary forms. Primary brain injury, which occurs immediately upon impact, can be
reduced only through prevention initiatives. Secondary brain injury, which ensues within hours
to days later, results from a cascade of cellular events (intracellular calcium, cell membrane
permeability changes, depletion of cellular energy, free radical generation, cell signaling
molecules) that harms or even destroys neuronal tissue in and around the site of the primary
injury.
Normal ICP is 0-15mmHg. Therapy should be initiated to lower ICP when it reaches 20mmHg.
More important than ICP is cerebral perfusion pressure (CPP), the difference between MAP and
ICP (CPP=MAP-ICP). CPP < 50mmHg must be strictly avoided and recommended CPP is 50-
70mmHg, though optimal CPP is unproven. CPP may be increased by lowering ICP or by
raising MAP.
ICP may be lowered by removal of noxious stimuli and adequate sedation. Morphine is sedative
and analgesic without increasing ICP. Midazolam may reduce MAP and raise ICP. Propofol is
the recommended sedative agent in brain injured patients because it lowers ICP. MAP can be
increased by use of alpha-adrenergic agents.
Alternative measures for brain oxygenation include jugular venous oxygen saturation (SjO2) and
brain tissue partial pressure of oxygen (PbrO2). If measured, interventions to increase cerebral
oxygenation when SjO2 drops below 50% and/or PbrO2 drops below 15mmHg.
Mannitol is a hyperosmolar plasma expander that also functions as an osmotic diuretic. Mannitol
expands plasma volume, reduces blood viscosity, increases cerebral blood flow and oxygen
delivery and because of its osmotic effects may reduce brain water and secondary brain injury.
Mannitol (0.25g-1g/kg) can be used to lower ICP based upon clinical signs alone (herniation or
progressive neurologic decline) or ICP monitoring. Clinicians must maintain adequate
intravascular volume in the face of mannitol therapy.
57
Hyperventilation has theoretical benefits in lowering pCO2 and thus cerebral blood volume which
lowers ICP. Hypocapnea can, however, produce cerebral ischemia and recent data indicate that
hypocapnea may be more harmful than hypercapnea. Moreover, prolonged hyperventilation is
probably ineffective because adaptation occurs and cerebral blood flow returns to baseline.
Current guidelines for CO2 control are to achieve a pCO2=35 and avoid hyperventilation to pCO2
< 35 mmHg for the first 24 hours post-injury. Hyperventilation may be used as a temporizing
measure only in cases of refractory intracranial hypertension.
Additional treatments for severe traumatic brain injury include barbiturates which are
recommended for refractory intracranial hypertension in hemodynamically stable patients.
Hemodynamic instability and severe ileus are side effects of this therapy. Steroids are
contraindicated for the treatment of traumatic brain injury. Anticonvulsants (phenytoin) may be
used to prevent early post-traumatic seizures and therapy duration is ≈ 7 days. Prophylactic
antibiotics are not recommended for indwelling ICP monitors. DVT prophylaxis is recommended
in patients with TBI, with compression devices initially and progressing to anticoagulants as
neurosurgery indicates is appropriate.
Pre-Hospital:
Ventilate patient with 100% oxygen. Keep PaO2 > 60mmHg (Sat >90%).
Steroids have not been found to be of benefit to the head-injured patient and are not
recommended as therapy for severe head injury.
58
Insure patent airway (endotracheal intubation is indicated in patients with GCS < 8).
Maintain MAP > 90 throughout patient course in attempt to maintain cerebral perfusion
pressure (CPP) 50-70mmHg. Insure adequate volume repletion before adding
vasopressors (CVP >12).
59
Severe Head Injury Management Algorithm
(GCS < 8)
Neurologic assessment
(GCS < 8)
CT Head
(see protocol)
IVF NS (preferred)
MAP > 90
Decompress stomach
(Place NG/OG)
Insert Foley
Yes No Mannitol
¼ gm – 1
gm/kg
60
FACIAL BONE FRACTURE
EMERGENCY DEPARTMENT RADIOLOGIC ALGORITHM
Indications:
1. Obvious facial bone fracture or fracture suspected on physical exam.
2. Facial bone fracture detected on head CT.
3. Facial bone fracture detected on radiograph from referring facility.
Axial Spiral CT
3mm Slices
Coronal
Reconstructions*
*Axial spiral CT with 3mm slices provides sufficient detail for diagnosis and treatment planning
for facial fractures (forehead to mandible).
†These films are not often needed urgently and should be obtained at the request or discretion
of the maxillofacial trauma consultant.
Facial plain films are sometimes helpful for treatment planning.
While (formal) coronal CT is usually only needed when coronal reconstructions
do not provide adequate detail for surgical treatment planning, an urgent coronal
CT may sometimes be needed to resolve an equivocal CT with regards to optic
nerve integrity or compression.
§Mandibular Panorex is a useful study in patients who can sit upright and cooperate with the
exam. Thus Panorex is not often logistically obtainable in multi-trauma patients and axial CT
with coronal reconstructions, which provides satisfactory detail for treatment planning, is the
usual diagnostic of choice.
11/08
61
SPINE CLEARANCE
Basic Principles
General
1. Entire spine is immobilized during primary survey.
2. Radiographic clearance of the spine is not required before emergent surgical
procedures. Presence of a spinal column injury is simply assumed until excluded.
3. Secondary and tertiary exams include examination of the spine for tenderness as well as
testing all motor roots, sensation and reflexes.
4. Tertiary exams are performed only on alert and unimpaired patient without distracting
injuries.
5. If any spine fractures are found, entire spine must be radiographed.
6. For patients with radiographic injury spine consultation requested for focused pre-
operative evaluation regarding relative instability and severity of injury prior to intubation.
7. Patients remain on spine precautions until spine is cleared.
Cervical
1. C-spines are not cleared until after the tertiary exam is completed.
2. Cervical CT scan is the preferred radiographic modality when physical exam is not
adequate.
3. With impaired or unconscious patient, rigid collars are taken off within 2 hours and
replaced with semi-rigid pressure reducing collar.
4. Enter patients in cervical algorithm for C-Spine clearance.
Thoraco-Lumbar
1. CT scan of thoracic and lumbar spines if there are clinical findings on secondary or
tertiary exams or an unreliable exam. Multi-detector CT-scan with reformatted axial
collimation is superior to plain films.
2. Radiographic Thoraco-Lumbar clearance is not needed prior to OR for non spine
surgery. Thoracic & Lumbar clearance may however be required for some non supine
positioning in the OR, depending upon acuity and case type.
3. Tertiary exam is necessary to clear thoracic and lumbar spines.
8/08
62
63
Initial Management of Spinal Cord Injury
64
65
66
Penetrating Neck Injury Evaluation and Treatment Algorithm
University of Kentucky Hospital Trauma Center
Overview
The neck is divided into zones:
Zone 1-Clavicles to cricoid cartilage
Zone 2-Cricoid to angles of the mandible
Zone 3-Angles of the mandible to skull base
Management of patients with penetrating wounds to the neck has historically been determined
by zone of injury. Because zones 1 and 3 are challenging to expose surgically, patients with
injuries in zones 1 and/or 3 warrant thorough diagnostics because non-therapeutic surgery in
these areas is both difficult and morbid. Zones 1 and 3 should be approached surgically only if
an injury is felt to be present. However, zone 2 of the neck is easily exposed surgically.
Controversy has existed as to whether patients with zone 2 injuries should undergo exhaustive
diagnostics to exclude or characterize injuries in this area, or simply undergo neck exploration
with limited or no preoperative evaluation of the esophagus and cervical vasculature
(esophagography or esophagoscopy or both plus angiography).
Recently published studies have changed the management of penetrating neck trauma in 2
important ways. First, evidence suggests that for patients with no clinical evidence of vascular
injury (shown on the algorithm as ―Mandatory Criteria for Neck Exploration‖) then physical
examination has 100% sensitivity, thus definitively excluding vascular injury without any
angiography. This is most true if the injury is to Zone 2. The second major advance in the care
of these patients has been the advent of helical CT angiography as an alternative to
conventional catheter-based angiography, producing equivalent results.
Furthermore, the anatomic detail provided by the neck CT may permit the clinician to exclude
injury to the esophagus if the CT clearly shows a missile trajectory remote from the esophagus.
However, the precise role that CT will play in excluding injury to the esophagus in these patients
remains to be established. If the CT does not conclusively exclude injury to the esophagus,
contrast esophagography and/or esophagoscopy (or both) should be performed.
Summary
1. CT scan of the neck including CT cervical angiography is the initial diagnostic of choice.
2. Some asymptomatic patients may avoid angiography entirely.
3. Esophageal injury must be definitively excluded, which may require esophagography or
esophagoscopy or both.
References regarding the utility of CT/CTA for evaluating penetrating neck injury:
1. Gracias VH, Reilly PM, Philpott J, Klein WP, Lee SY, Singer M, Schwab CW. Computed
tomography in the evaluation of penetrating neck trauma: a preliminary study. Arch Surg
2001;136: 1231-5.
2. Mazolewski PJ, Curry JD, Browder T, Fildes J. Computed tomographic scan can be
used for surgical decision making in zone II penetrating neck injuries. J Trauma
2001;51:315-9.
3. Múnera F, Soto JA, Palacio DM, Castañeda J, Morales C, Sanabria A, Gutiérrez JE,
García G. Penetrating neck injuries: helical CT angiography for initial evaluation.
Radiology 2002; 224:366-72.
67
4. Fergusion E, Dennis JW, Vu JH, Frykberg ER. Redefining the role of arterial imaging in
the management of penetrating zone 3 neck injuries. Vascular. May-jun 2005;13(3):158-
63. [Medline].
References demonstrating the diagnostic accuracy of physical exam for vascular injuries in
Zone II requiring intervention:
1. Nason RW, Assuras GN, Gray PR, Lipschitz J, Burns CM. Penetrating neck injuries:
analysis of experience from a Canadian trauma centre. Can J Surg 2001;44:122-6.
2. Sriussadaporn S, Pak-Art R, Tharavej C, Sirichindakul B, Chiamananthapong S. Selective
management of penetrating neck injuries based on clinical presentations is safe and
practical. Int Surg 2001;86:90-3.
3. Azuaje RE, Jacobson LE, Glover J, Gomez GA, Rodman GH Jr, Broadie TA, Simons
CJ, Bjerke HS. Reliability of physical examination as a predictor of vascular injury after
penetrating neck trauma. Am Surg 2003;69:804-7.
4. Osborn TM, Bell RB, Qaisi W, Long WB. Computed tomographic angiography as an aid
to clinical decision making in the selective management of penetrating injuries to the
nec: a reduction in the need for operative exploration. J Trauma. Jun 2008;64(6):1466-
71. [Medline].
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Blunt Cardiac Injury (BCI)
Blunt cardiac injury (BCI), formerly called myocardial contusion, encompasses a spectrum of
disease ranging from histologic injury to the myocardium without clinical manifestation to blunt
cardiac rupture.1 BCI contributes to 20% of prehospital deaths from blunt trauma.2 An exact
incidence of BCI does not exist because there is no ―gold standard‖ for diagnosis, i.e., the
available data are conflicting with regard to how the diagnosis should be made (ECG,
echocardiography, enzyme analysis, etc). This lack of a diagnostic standard makes the
literature difficult to interpret and leads to confusion in clinical practice.
The major priority is identification of patients at risk for adverse events resulting from BCI and
providing appropriate workup, monitoring and treatment. Conversely, patients not at risk can
potentially be discharged home. Patients with appropriate mechanism of injury and clinical
evidence of cardiac dysfunction (electrophysiologic or mechanical) can be considered to have
BCI.
BCI results from 5 possible mechanisms: direct pre-cordial impact, crush between sternum and
spine, deceleration or torsion causing a tear in the heart at a point of fixation, hydraulic effect
resulting in rupture from elevated intra-abdominal and caval pressure, and blast injury.3 Few
clinical signs are diagnostic of BCI. Chest pain is the most common finding, but dyspnea, chest
wall ecchymosis and rib fractures may also be present. Associated injuries include hemothorax,
sternal fracture and great vessel injury. Clinical signs consistent with BCI include dysrhythmias,
cardiac ischemia, low cardiac output and hypotension.4
Diagnostic tests include ECG, echocardiography, and enzyme analysis. Controversy exists
regarding the application of these tests. Frequency of diagnosis of BCI will be proportional to the
aggressiveness with which it is sought. Appropriate workup commands achieving a balance
between cost-effectiveness and information acquisition with attention to the clinical value of
information gained in changing patient management. Guidelines for using diagnostic tests are
as follows:
A. Level 1 evidence supports obtaining an ECG in the emergency department for at-risk
patients (described above).5 Using any ECG abnormality, including sinus tachycardia,
bradycardia conduction delays and PAC’s/PVC’s, the diagnostic sensitivity of ECG is
100%.6
B. Echocardiography is not effective as a screening tool and does not identify patients at
risk for complications.7 Transthoracic (TTE) or transesophageal echocardiography
(TEE) should be obtained in patients with evidence of hemodynamic instability or in
whom coincident coronary ischemia is suspected.
C. CK and CKMB fraction analysis is NOT indicated in suspected BCI because associated
skeletal and visceral injury creates serum CK abnormalities that contribute to an
unacceptable false-positive and negative rate.8 However, a body of evidence exists
suggesting some value to cardiac troponin I (cTnI) or troponin T (cTnT). That evidence
is as follows:
i. Diagnosis of BCI should not rely solely on cTnI or cTnT. ECG should be
included.
ii. Normal ECG and normal cTnI is 100% sensitive for BCI.9
iii. Abnormal ECG and abnormal cTnI is 100% specific for BCI.9
iv. cTnI is of little added benefit in patients with a markedly abnormal ECG
(diagnosis is already made).10
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v. Though the utility of cTnI in patients with normal ECG’s is limited, cTnI obtained
4-6 hours after the injury in patients with sinus tachycardia or non-specific EKC
changes or in older patients may give reassurance that the likelihood of BCI-
related complications is low. 10
References:
1. Mattox KL, Flint LM, Carrico CJ, Grover F, Meredith J, Morris J, et al. Blunt cardiac injury. J
Trauma 1992;33(5):649-50.
2. Schultz JM, Trunkey DD. Blunt cardiac injury. Crit Care Clin 2004;20:57-70.
3. Fulda G, Brathwaite CE, Rodriguez A, Turney SZ, Dunham CM, Cowley RA. Blunt traumatic
rupture of the heart and pericardium: a ten-year experience (1979–1989). J Trauma
1991;31(2):167-73.
4. Miller FB, Shumate CR, Richardson JD. Myocardial contusion. When can the diagnosis be
eliminated? Arch Surg 1989;124(7):805-8.
5. Pasquale NK, Clarke J. Screening of blunt cardiac injury.1998. The Eastern Association for
the Surgery of Trauma. Available: [Link]
6. Fabian TC, Cicala RS, Croce MA, Westbrook LL, Coleman PA, Minard G, et al. A
prospective evaluation of myocardial contusion: correlation of significant arrhythmias and
cardiac output with CPK-MB measurements. J Trauma 1991;31(5):653-60.
7. Karalis DG, Victor MF, Davis GA, McAllister MP, Covalesky VA, Ross Jr JJ, et al. The role of
echocardiography in blunt chest trauma: a transthoracic and transesophageal
echocardiographic study. J Trauma 1994;36(1):53-8.
8. Fabian TC, Cicala RS, Croce MA, Westbrook LL, Coleman PA, Minard G, et al. A
prospective evaluation of myocardial contusion: correlation of significant arrhythmias and
cardiac output with CPK-MB measurements. J Trauma 1991;31(5):653-60.
9. Salim A, Velmahos GC, Jindal A, Chan L, Vassiliu P, Belzberg H, et al. Clinically significant
blunt cardiac trauma: role of serum Troponin levels combined with electrocardiographic
findings. J Trauma 2001;50(2):237-43.
10. Collins JN, Cole FJ, Weireter LJ, Riblet JL, Britt LD. The usefulness of serum Troponin
levels in evaluating cardiac injury [discussion]. Am Surg 2001;67(9):821-6.
Revised: 08/08
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Revised 8/08
THE DIAGNOSIS OF BLUNT INJURY TO THE THORACIC AORTA
What diagnostic test is best?
Angiography, Transesophageal Echocardiography,
or Computerized Axial Tomography
Background
Blunt chest trauma with resulting aortic injury is a significant cause of death following high-
speed motor vehicle collisions.1-3 The vast majority of these patients (80%-90%) expire at the
collision scene.1-3 For the remaining 10%-20%, the mortality rate is high. Thirty percent expire
within 6 hours and 40%-50% within 24 hours of injury.1 The recent multicenter trial by the
American Association for the Surgery of Trauma reported an overall mortality of 31%, with 63%
of the deaths attributable to aortic rupture.4 Expeditious evaluation and timely surgical
intervention are essential for patient survival.
Screening
The goal of screening patients is to attain a zero nontherapeutic surgery rate without
overlooking any significant aortic or arch vessel injury. Mechanism of injury, clinical exam, and
the initial chest radiograph should reliably select patients who require further diagnostic
evaluation. Chest radiographs demonstrating mediastinal hematoma have good sensitivity
(93%) for aortic and arch vessel injury.5-7 When combined with mechanism of injury, sensitivity
rises to 98%.5 More importantly the negative predictive value of a normal upright chest
radiograph is almost 100%. A normal chest radiograph virtually excludes aortic and/or arch
vessel injury.5-7 Unfortunately, the specificity of an abnormal chest radiograph is only 10%-45%.
Since most mediastinal hematoma originates from thoracic vascular structures other than the
aorta, a definitive diagnostic test is required to establish the diagnosis of aortic injury.
Computerized axial tomography (CT) of the chest can be used to screen patients for
subsequent aortography.8-12 CT is more sensitive than chest radiograph in detecting mediastinal
hematoma.8,9 Screening thoracic CT is cost-effective and the negative predictive value of a
normal study is 100%.8-10 Consequently, CT can be used to reduce the negative aortogram rate
in patients with an abnormal chest radiograph.8-10
Diagnosis-Aortography
Many authors advocate the liberal use of aortography based on mechanism of injury and chest
radiographs.1,3,9,13 Using this approach, aortography yields positive results in only 10% of
patients. Although angiography remains the ―gold standard‖, the procedure is invasive and time
consuming, requires the use of intravenous contrast material and ionizing radiation, and can
result in false negative or false positive results.3,9,13,14 Transporting an injured patient to the
angiography suite is not without risk and interrupts the patient’s ongoing evaluation,
resuscitation, and treatment. On the other hand, when performed properly, aortography has a
sensitivity and specificity of 99%.9 Complications occur in less than 1% of patients.9
73
Aortography remains the only study that provides detailed images of the entire thoracic aorta
and the arch vessels.
Diagnosis-Transesophageal Echocardiography
Transesophageal echocardiography (TEE) provides high-resolution real-time axial and
longitudinal images of the aorta.14-18 TEE can accurately demonstrate injury to the thoracic
aorta.14-18 In our hands, TEE was more sensitive (100%) and specific (98%) than
aortography.14,15 TEE offers a number of advantages over aortography and CT scanning. The
study can be performed at the bedside, eliminating transport risks. Concomitant diagnostic and
therapeutic procedures can continue unhindered. TEE provides real-time images so that areas
of interest can be examined repeatedly in different planes. Simultaneous evaluation of cardiac
pathology and function can also be obtained. If urgent surgical intervention is indicated for other
injuries, TEE can be performed in the operating room without delaying TEE or the surgical
intervention. The study can be performed more rapidly than aortography making TEE ideal for
the evaluation of the unstable trauma patient with a number of diagnostic and treatment
priorities. Unfortunately, TEE requires the expertise of a well-trained and interested
echocardiographer. Airway compromise, esophageal pathology, and unstable cervical spine
fractures are contraindications to TEE. The depth and extent of injury are frequently difficult to
determine particularly when atherosclerotic disease diminishes the sensitivity of the
examination. In our experience, this has resulted in nontherapeutic thoracotomy.14 There are
blind spots related to the tracheal air column and the arch vessels simply cannot be imaged.
Summary
Patients with suspected blunt injury to the thoracic aorta are a challenge for the trauma surgeon.
Multisystem trauma, critical illness, and hemodynamic instability in this patient group result in
diagnostic and treatment dilemmas. We employ a practical, evidence-based algorithm for the
screening and diagnosis of injury to the thoracic aorta. Both mechanism of injury and an
abnormal mediastinum on chest radiograph are required to trigger a diagnostic evaluation.
Every attempt is made to obtain an ―upright‖ AP chest radiograph to minimize distortion and
magnification. However, this is not possible in all patients. Widening of the mediastinum alone
is neither sensitive nor specific for mediastinal hematoma.5,6,7 Instead, we employ the criteria for
mediastinal hematoma (abnormal mediastinal silhouette) as defined by Mirvis and Ayella.6,7 No
further diagnostic evaluation is undertaken in patients with a normal chest radiograph, unless
there are compelling physical findings that suggest aortic or arch vessel injury (i.e. Pulse deficit,
74
unequal blood pressure/pulse measurement, unexplained hemodynamic instability, unexplained
neurologic deficit). Thoracic CT scan using the aortic protocol is performed. 9,19 A negative scan
yields observation. A scan positive for aortic injury prompts surgical intervention or appropriate
non-operative management. Patients with indeterminate scans undergo angiography or TEE to
establish or exclude the diagnosis.
REFERENCES:
1. Parmley LF, Mattingly TW, Manion WC, et al. Nonpenetrating traumatic injury of the
aorta. Circulation 1958;17:1086-1101.
2. Greendyke RM. Traumatic rupture of the aorta: special reference to automobile
accidents. JAMA 1966;195:527-530.
3. Culliford AT: Traumatic aortic rupture. In Hood RM, Boyd AD. Culliford AT (eds):
Thoracic Trauma. Philadelphia, WB Saunders, 1989, pp 224-244.
4. Fabian TC, Richardson JD, Croce MA, et al. Prospective study of blunt aortic injury:
multicenter trial of the American Association of the Surgery of Trauma. J Trauma 1997;
42:374-380.
5. Woodring JH. The normal mediastinum in blunt traumatic rupture of the thoracic aorta
and brachiocephalic arteries. J Emerg Med 1990;8:467-476.
6. Mirvis SE, Bidwell JK, Buddemeyer EU, et al. Imaging diagnosis of traumatic aortic
rupture: A review and experience at a major trauma center. Invest Radiol 1987;22:187-
190.
7. Ayella RJ, Hankins JR, Turney SZ, et al. Ruptured thoracic aorta due to blunt trauma. J
Trauma 1977;17:199-204.
8. Harris JH, Horowitz DR, Zelitt DL. Unenhanced dynamic mediastinal computed
tomography in the selection of patients requiring aortography for the detection of acute
traumatic aortic injury. Emerg Radiol 1995;2:67-76.
9. Patel NH, Stephens KE, Mirvis SE, et al. Imaging of acute thoracic aortic injury due to
blunt trauma: A review. Radiology 1998;209:335-348.
10. Mirvis SE, Shanmuganathan K, Miller BH, et al. Traumatic aortic injury: Diagnosis with
contrast-enhanced thoracic CT. Five year experience in a major trauma center.
Radiology 1996;200:413-422.
11. Demetriades D, Gomez H, Velmahos GC, et al. Routine helical computed tomographic
evaluation of the mediastinum in high-risk blunt trauma patients. Arch Surg
1998;133:1084-1088.
12. Dyer DS, Moore EE, Ilke DN, et al. Thoracic aortic injury: How predictive is mechanism
and is chest computed tomography a reliable screening tool? A prospective study of
1,561 patients. J of Trauma. 2000;48:673-683.
13. Kirsh MM, Behrendt DM, Orringer MB, et al. The treatment of acute traumatic rupture of
the aorta: A 10-year experience. Ann Surg 1976; 184:308-316.
14. Smith MD, Cassidy JM, Souther S, et al. Transesophageal echocardiography in the
diagnosis of traumatic rupture of the aorta. N Engl J Med 1995;332:356-362.
15. Buckmaster MJ, Kearney PA, Johnson SB, et al. Further experience with
transesophageal echocardiography in the evaluation of thoracic aortic injury. J of
Trauma 1994;37:989-995.
16. Vignon P, Gueret P, Vedrinne JM, et al. Role of transesophageal echocardiography in
the diagnosis and management of traumatic aortic disruption. Circulation 1995;92:2959-
2968.
17. Goarin JP, Catoire P, Jacquens Y, et al. Use of transesophageal echocardiography for
diagnosis of traumatic aortic injury. Chest 1997;112:71-80.
18. Mollod M, Felner JM. Transesophageal echocardiography in the evaluation of
cardiothoracic trauma. Am Heart J 1996;132:841-849.
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19. Mirvis SE, Shannuganathan K, Buell J, Rodriquez A. Use of spiral computed
tomography for the assessment of blunt trauma patients with potential aortic injury. J of
Trauma 1998;45:922-930.
20. Gavant M. Helical CT grading of traumatic aortic injuries. Impact on clinical guidelines for
medical and surgical management. Rad Clinics of North America 1999;37:552-574.
21. Wicky S, Capasso P, Meuli R, et al. Spiral CT aortography: an efficient technique for
the diagnosis of traumatic aortic injury. European Radiology 1998;8:828-833.
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BLUNT THORACIC INJURY WITH SUSPECTED INJURY TO THE THORACIC AORTA OR
ARCH VESSELS
Patients who sustain blunt thoracic trauma are at risk for injury to the heart and great vessels.
Patients should be selected for additional diagnostic studies based on mechanism of injury and
evidence of mediastinal hematoma on chest radiograph. Every attempt is made to obtain an
―upright‖ AP chest radiograph to minimize distortion and magnification. However, this is not
possible in all patients. Widening of the mediastinum alone is neither sensitive nor specific for
mediastinal hematoma (5,6,7). Instead, we employ the criteria for mediastinal hematoma
(abnormal mediastinal silhouette) as defined by Mirvis and Ayella (6,7). No further diagnostic
evaluation is undertaken in patients with a normal chest radiograph, unless there are compelling
physical findings that suggest aortic or arch vessel injury (i.e. Pulse deficit, unequal blood
pressure/pulse measurement, unexplained hemodynamic instability, unexplained neurologic
deficit).
INJURY
1. Sternal and/or scapular fracture
2. Multiple rib fractures and/or flail chest
* Isolated fractures of the first and second ribs without evidence of mediastinal hematoma do
not correlate with aortic or arch vessel injury and are not an indication for further imaging.
References:
1. Mirvis SE, Bidwell JK, Buddemeyer EU, et al. Imaging diagnosis of traumatic aortic
rupture: A review and experience at a major trauma center. Invest Radiol 1987;22:187-
190.
2. Ayella RJ, Hankins JR, Turney SZ, et al. Ruptured thoracic aorta due to blunt trauma. J
Trauma 1977;17:199-204.
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DIAGNOSTICS: ROLE OF DPL, CT, FAST (ultrasound)
05/08
The goal of the abdominal evaluation of multi-system trauma patients is the safe, accurate, and
timely determination of the presence or absence of intra-abdominal injury, particularly those
requiring surgical intervention. Physical examination can be unreliable in polytrauma patients,
particularly when the abdominal examination or the level of consciousness has been altered by
alcohol, drugs, central system trauma, or distracting pain. Diagnostic peritoneal lavage (DPL), CT
scan, and abdominal sonography (FAST) are the most frequently employed diagnostic studies
used for the abdominal examination of multi-system trauma patients. Each study has advantages
and disadvantages. The goal of this session is to integrate these complementary studies into a
rational diagnostic algorithm for the evaluation of the abdomen.
Introduced by Root et al. in 1965, DPL (Diagnostic Peritoneal Lavage) was the time-honored
standard for evaluation of the abdomen for many years. DPL is rapid (< 30 minutes) and safe (<
2% complication rate) with well-established standards for surgical intervention on the basis of cell
counts or the aspiration of free blood after lavage. Overall sensitivity is 98%, with a specificity of
98% and a diagnostic accuracy approaching 100%. The procedure is inexpensive and can be
performed at the bedside while other diagnostic and therapeutic interventions proceed. The
procedure is invasive. Previous abdominal surgery makes the procedure more difficult and may
enhance complications and diminish specificity. Pelvic fractures also diminish specificity.
DPL cannot evaluate extraperitoneal structures (thorax, retroperitoneum, and pelvis) and must be
supplemented with other diagnostic procedures. DPL is not organ specific. Therefore, a positive
study may lead to a non-therapeutic laparotomy for a trivial injury. In recent years, DPL has been
replaced by FAST in the unstable patient and has a limited role in the stable patient.
Abdominopelvic CT scanning for blunt abdominal trauma was introduced by Federle et al. in the
early 1980s and has gained wide popularity in the United States. CT scanning has a sensitivity of
96%, a specificity of 98%, and an accuracy of 97%. CT is organ specific, allowing the
identification and grading of injured organs and the quantification of intraperitoneal fluid or blood.
This allows for non-operative management of stable patients, thereby reducing the rate of non-
therapeutic laparotomy. Extraperitoneal injuries (thorax, retroperitoneum, and pelvis) can be
identified and graded. CT scanning is relatively expensive. Sensitivity and specificity depend on
quality of the scan and skill of the interpreter. Contrast aspiration and allergy may occur. Bowel
and pancreatic injuries may be missed. Even with the new, more rapid scanners, CT is time
consuming, and transport to the scanner interrupts other diagnostic and therapeutic interventions.
Consequently, CT is limited to hemodynamically stable patients. CT scan is the mainstay in the
abdominal evaluation of the stable blunt trauma victim. CT now has a role to play in the
evaluation of penetrating torso injury.
The use of ultrasound or FAST (Focused Assessment with Sonography in Trauma) in abdominal
trauma first evolved in Germany. FAST has been employed successfully at trauma centers in the
United States for over a decade. US is noninvasive, rapid (2 to 4 minutes), and relatively
inexpensive. The examination can be performed at the bedside, does not interfere with other
diagnostic and therapeutic interventions, and can be repeated as needed. The primary goal of
FAST is to detect free fluid in Morison’s Pouch, the pelvis, peri-splenic region and the
pericardium. The sensitivity of FAST ranges from 80% to 100%; the specificity, from 89% to
100%; and the accuracy, from 86% to 99%. Extraperitoneal structures can be imaged (thorax,
pericardium, retroperitoneum). The technique can be easily learned and performed by the
81
treating surgeon. Sensitivity, specificity, and accuracy of US clearly improve with experience.
Although extremely sensitive for peritoneal fluid, US is much less sensitive for specific organ
injury (liver, spleen, etc.). As is true of CT, US can miss bowel injuries. However, repeat
examinations mitigate the latter weakness, and organ-specific diagnostic sensitivity improves with
the experience of the examiner. Sensitivity, specificity, and accuracy also depend on image
quality. Obesity, bowel gas, and subcutaneous emphysema interfere with imaging.
References
1. Root HD, Hauser CW, McKinley CR, et al. Diagnostic peritoneal lavage. Surgery
1965;57:633-637.
2. Federle MP, Goldberg HI, Kaiser JA, et al. Evaluation of abdominal trauma by
computed tomography. Radiology 1981;138:637-644.
4. Kearney PA, Vahey T, Burney RE, et al. Computed tomography and diagnostic
peritoneal lavage in blunt abdominal trauma. Their combined role. Arch Surg
1989;124:344-347.
5. Hoffman R, Nerlich M, Muggia-Sullam M, et al. Blunt abdominal trauma in
cases of multiple trauma evaluated by ultrasonography: a prospective analysis
of 291 patients. J Trauma 1992;22:452-458.
6. Goletti O, Ghiselli G, Lippolis PV, et al. The role of ultrasonography in blunt
abdominal trauma: results in 250 consecutive cases. J Trauma 1994;36:178-181.
7. Forster R, Pillasch J, Zielke A, et al. Ultrasonography in blunt abdominal trauma:
influence of the investigator experience. J Trauma 1992;34:264-269.
8. Liu M, Lee CH, P’eng FK. Prospective comparison of diagnostic peritoneal
lavage, computed tomographic scanning, and ultrasonography for the diagnosis
of blunt abdominal trauma. J Trauma 1993;35:267-270.
9. Rozycki GS, Ochsner MG, Jaffin JH, et al. Prospective evaluation of surgeons’
use of ultrasound in the evaluation of trauma patients. J Trauma 1993;34:516-
527.
10. McKenney M, Lentz K, Nunez D, et al. Can ultrasound replace diagnostic
peritoneal lavage in the assessment of blunt trauma? J Trauma 1994;37:439-441.
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Genitourinary Trauma
5/08
Trauma to the GU tract is present in approximately 10% of all injuries. The following discussion
pertains to the evaluation and management of the stable patient.
Work-up of GU trauma begins with assessment of the urethra. Injuries of the female urethra are
rare. Urethral injuries are usually due to blunt trauma associated with pelvic fracture or
straddle-type injuries. The primary posterior site of urethral injury is the prostatomembranous
junction. The primary anterior urethral injuries are most commonly caused by straddle-type
injuries or perineal trauma. The location of injury is typically the bulbar urethra. Physical finding
of blood at the meatus, perineal hematoma or extensive laceration, a high riding prostate, or a
large hematoma found on rectal exam mandates a retrograde urethrogram prior to insertion of a
Foley catheter. Management requires suprapubic urinary diversion or endoscopic assisted
placement of Foley catheter.
Once urethral injury is ruled out, a Foley catheter should be inserted. It is essential to document
the color of the urine, as gross hematuria mandates further workup of the GU tract. Evidence
suggests the microscopic hematuria is not diagnostic for GU trauma and as such a urinalysis
should not be part of the workup.
If there is gross hematuria, evaluation of the remainder of the GU tract – kidneys, ureters (in
penetrating trauma), and bladder needs to be performed. This can be accomplished by various
imaging techniques. However, with the advancement of the CT technology, it has evolved to
become the standard of care. Therefore, for gross hematuria, a CT scan of the abdomen and
pelvis to evaluate the kidneys, as well as CT cystogram should be obtained.
The kidneys are the most commonly injured organs in the GU tract. Management depends on
hemodynamic stability and the grade of injury by CT scan. Most renal injuries do not require an
operation in a stable patient. However, incidental intra-operative finding of perinephric
hematoma should be explored if the mechanism is penetrating trauma.
A blunt ureteral injury is a case report! Most ureteral injuries are penetrating. They require
surgical intervention. Basic principles of ureteral reconstruction include debridement of
devitalized tissue, followed by tension-free anastomosis with absorbable suture in a spatulated
fashion over a double J stent.
Bladder ruptures can result from penetrating or blunt trauma. Extraperitoneal bladder rupture is
commonly associated with pelvic fracture; and intraperitoneal bladder rupture is a result of blunt
lower abdominal force on a full bladder. Classic physical findings of bladder rupture include
suprapubic pain, hematuria, and inability to void. A CT cystogram should be obtained.
Extraperitoneal bladder rupture is generally managed with Foley catheter drainage, and
intraperitoneal bladder rupture requires immediate surgical intervention.
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Traumatic Peripheral Vascular Injury
Any injured extremity should be thoroughly evaluated for a possible vascular injury. The
presence of obvious arterial injury from a blunt and/or penetrating mechanism rarely requires
imaging and should not delay emergent operative exploration. The presence of ―hard signs‖
strongly supports vascular injury and typically necessitates emergent repair. These ―hard signs‖
are:
1. Bruit/Thrill
2. Active/Pulsatile hemorrhage
3. Pulsatile/Expanding hematoma
4. Signs of limb ischemia and or compartment syndrome including the 5 "P's" - pallor,
paresthesias, pulse deficit, paralysis, and pain on passive extension of the compartment
(pain on passive extension is the earliest and most sensitive physical finding)
5. Diminished or absent pulses with + Doppler signals (this is not a sensitive prognostic
finding, as up to 30% of patients with major vascular injuries requiring repair have
normal pulses or Doppler signals distal to the injury due to collateral flow) [1]
The Arterial Perfusion Index, API, is a validated tool for screening for peripheral vascular
injury[2]. This is performed by placing a blood pressure cuff above the ankle or on the bicep of
the limb of concern. The systolic pressure is determined with a Doppler probe at the dorsalis
pedis or brachial artery. Repeat this procedure on the ipsilateral uninjured limb. The API is
calculated by dividing the systolic pressure in the injured limb by the systolic pressure in the
uninjured limb. An API < 0.9 has a sensitivity of 95% and specificity of 97% for a major arterial
extremity injury. In a study on blunt orthopedic extremity injuries the negative predictive value is
100% for an API > 0.9 to exclude an arterial injury.[3-5]
The purpose of these algorithms is to diagnose the occult injury early before irreversible tissue
ischemia is present. In patients where the ―hard‖ signs are NOT present it is imperative to
maintain a high suspicion of peripheral vascular injury in the injured extremity [2, 6, 7]. If ―hard
signs― are not present but peripheral vascular injury is suspected then expedient consultation
with Vascular Surgery is indicated and the use of imaging, per Vascular Surgery, should be
liberal to avoid missed injuries.
References:
1. Drapanas, T., et al., Civilian vascular injuries: a critical appraisal of three decades of
management. Ann Surg, 1970. 172(3): p. 351-60.
2. Bravman, J.T., et al., Vascular injuries after minor blunt upper extremity trauma - pitfalls
in the recognition and diagnosis of potential "near miss" injuries. Scand J Trauma
Resusc Emerg Med, 2008. 16(1): p. 16.
3. Mills, W.J., D.P. Barei, and P. McNair, The value of the ankle-brachial index for
diagnosing arterial injury after knee dislocation: a prospective study. J Trauma, 2004.
56(6): p. 1261-5.
4. Lynch, K. and K. Johansen, Can Doppler pressure measurement replace "exclusion"
arteriography in the diagnosis of occult extremity arterial trauma? Ann Surg, 1991.
214(6): p. 737-41.
5. Johansen, K., et al., Non-invasive vascular tests reliably exclude occult arterial trauma in
injured extremities. J Trauma, 1991. 31(4): p. 515-9; discussion 519-22.
6. Dennis, J.W., et al., Validation of nonoperative management of occult vascular injuries
and accuracy of physical examination alone in penetrating extremity trauma: 5- to 10-
year follow-up. J Trauma, 1998. 44(2): p. 243-52; discussion 242-3.
89
7. Gelberman, R.H., J. Menon, and A. Fronek, The peripheral pulse following arterial injury.
J Trauma, 1980. 20(11): p. 948-51.
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Open Long Bone Extremity Fracture Protocol
Open long bone extremity fractures are associated with significant trauma. The expedient
management of these injuries ensures the best possible fracture treatment outcome. The
determination of the grade of open fracture is the responsibility of the orthopedic trauma service.
The extent of the type of open fractures often requires intra-operative evaluation.
If patient exposed to barn or farm wound contamination then add high dose PCN x 24 hours
(Type I and II) or 48 hours in Type III. If patient is has pen-allergy consider flagyl.
Open fracture management and evaluation, including antibiotics should be initiated as soon as
possible from the timing of wounding, not based on arrival to ER.
If a patient gets an operation on this open fracture after completion of the above duration of
antibiotics is given they should get only a perioperative dose.
Wound care for open long bone extremity fracture requires coverage of the wound with a
Hibiclens or Betadine soaked gauze.
Mandatory documentation of neurovascular exam is required in all extremity injuries pre and
post extremity fracture care. Frequent neurovascular examinations are required before and
after fracture management to detect extremity compartment syndrome. The LAST clinical
finding lost in developing compartment syndrome is the pulse. The body has evolved to perfuse
cells until the very end so it makes sense that the pulse is the last clinical finding to be lost in
developing compartment syndrome.
* All open fractures must be evaluated by the Ortho Trauma Service for proper management
(stabilization, wound care, further fracture grading, and definitive fracture management)
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UNIVERSITY OF KENTUCKY HOSPITAL POLICY NUMBER: 08 CHANDLER
MEDICAL CENTER FIRST ISSUED: 05/07
PROCEDURE: To effectively manage trauma patients that are pregnant, the Trauma Team
must mobilize the resources essential to diagnosis and treat both mother and fetus. For
purposes of this policy, a potentially viable fetus is one at 24 weeks gestation, although some
exceptions may exist.
I. Mechanisms of Injury
1. Lab studies:
a. Urine pregnancy or serum beta-HCG, especially with a questionable history
b. Clotting factors and plasma fibrinogen
c. Kleihauer-Betke test, especially when blunt uterine trauma is suspected
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2. Imaging
a. Avoid duplicating films
b. Shield the fetus whenever possible
c. See Appendix 1 for estimated fetal exposure
Summary
______________________________ ________________________________
Medical Director, Emergency Medicine Trauma Program Director
94
UNIVERERSITY OF KENTUCKY HOSPITAL POLICY NUMBER: 08-55a
CHANDLER MEDICAL CENTER FIRST ISSUED: 05/07
Emergency Dept. Policy CURRENT AS OF: 08/08
Primary Survey
Roll backboard R-side up
10-15 degrees
PCXR
FAST
Fetal evaluation in OR
*FETUS IN DISTRESS*
Emergent C-section via
midline incision
(MAIN OR)
Trauma present for laparotomy
after delivery
*NORMAL FETUS*
Additional work-up as indicated
Fetal monitoring for at least 6
hours (or as directed by OB)
95
EMERGENCY DEPARTMENT EVALUATION OF BURN PATIENTS
Stabilization
A. Maintain airway - the supraglottic airway is extremely susceptible to obstruction from
edema as a result of exposure to superheated air. Assess for clinical signs of inhalation
injury:
- Facial burns/singeing of the eyebrows and nasal hairs
- Carbonaceous sputum and acute inflammatory changes in the oropharynx, raspy voice
- History of impaired mentation and/or confinement in a burning environment
- Assess for toxic inhalation, or carbon monoxide poisoning
B. Circulation
1. Replace volume based on Rule of Nines calculation. Follow the Parkland Formula (2 -
4cc x % TBSA burned x weight/kg = total to be administered in first 24 hours. Half should
be given in first 8 hours from time of burn and second half over next 16 hours) for
administration of fluid. The amount of fluid given should be adjusted according to
individual patient's response:
3. Initiate two large bore IVs; overlying burns shouldn't prevent IV placement (upper
extremities preferred)
Assessment
96
A. Assess for associated injuries
B. History - time and mechanism of the injury; enclosed fire, or if toxic chemicals involved
C. Assess burn
1. First degree: (Sunburns): characterized by erythema, pain, and the absence of
blisters – NOT COUNTED IN TBSA
2. Second degree: (Superficial Partial Thickness or Deep partial thickness):
characterized by a red or mottled appearance with swelling and blister formation. The
surface may have a weeping, wet appearance and is painfully hypersensitive
3. Third degree: (full thickness): skin appears dark and leathery; may also appear
translucent, mottled, or waxy white; the surface is painless and generally dry, but may
also be moist
D. Circumferential extremity burns:
1. Remove rings and bracelets and Assess distal circulation
2. Check pulses with a Doppler (absent pulse may indicate inadequate fluid
resuscitation)
d. Observe for cyanosis, impaired capillary refill, or progressive neurological signs (i.e.,
paresthesia and deep tissue pain)
E. Limb Escharotomy
Relieve compromised distal circulation in a circumferentially burned
limb by escharotomy, which can be done without anesthesia, due
to the insensitive full - thickness burn
1. The incision must extend across the entire length of the eschar
in the lateral and/or medial line of the limb including the fingers and
joints
2. The incision should be deep enough to allow the cut edges of
the eschar to separate
F. Thoracic Escharotomy:
Circumferential burns of the thorax occasionally impair respiratory
excursion. Bilateral, mid-axillary escharotomy incisions should be
considered
B. Electrical burns - frequently more serious than they appear on the surface
1. Initial care as above
2. Full spinal immobilization
3. EKG monitoring
4. Urinary catheter
a. Observe for myoglobinurea (due to rhabdomyolysis)
b. Increase IV rate fluid to ensure UO of at least 100 ml/hour
c. Consult Medical Control for Mannitol 25 GM IVP and IV infusion with 12.5 GMs of
mannitol/1000 cc NS to maintain the diuresis
C. Explosive Injuries –
97
Any patient involved in an explosion should be considered as having a mechanism for
traumatic injuries. Even if the patient states they were NOT thrown a distance. The force
of a flash flame explosion is enough energy to cause concussive type of injuries.
98
99
100
101
102
The Anticoagulated Trauma Patient
Warfarin use has been shown to increase the severity of head injury and a higher mortality rate.
Mortality of trauma patients with head injury while on warfarin ranges from 33% to 50%.i
Furthermore, it has been reported that the head injured patients on warfarin have an increased
risk of mortality from 2-fold to 4-fold, when compared with non-anti-coagulated patients with
similar degrees of head [Link]
As part of the trauma workup, one should always obtain an adequate history, which includes a
list of current home medication. Early identification of warfarin use has been shown to reduce
mortality on patients with intracranial hemorrhage from 48% to 9%.iii In the same study, mean
time to warfarin reversal (normal coagulation profile) was 1.7 hours in the early identification
group compared to 4.3 hours.
Another important aspect of the anti-coagulated patient is the decreased reliability of their
neurological exam. It has been shown that GCS of 15 and no loss of consciousness does not
reliably rule out intracranial pathology after trauma. Indeed, one study reported two anti-
coagulated patients with no loss of consciousness that eventually died from consequence of
intracranial hemorrhage.2 Therefore, all patients with known warfarin use should have a CT
scan of the head as part of their trauma workup regardless of their mental status.
1
Lavoie A, Ratte S, Clas D, Demers J, Moore L, Martin M, Bergeron E. Preinjury warfarin use
among elderly patients with closed head injuries in a trauma center. J Trauma. 2004
Apr;56(4):802-7.
1
Mina AA, Bair HA, Howells GA, Bendick PJ. Complications of preinjury warfarin use in the
trauma patient. Trauma. 2003 May;54(5):842-7.
1
Janczyk et al. Rapid warfarin reversal in anticoagulated trauma patients with intracranial
hemorrhage reduces hemorrhage progression and mortality. Abstract presented at AAST
annual meeting, September 30, 2004.
Close Neurosurgery
observation consult & check
fibrinogen level
103
SECTION 4: ICU SPECIFIC PROTOCOLS
Revised: 08/08
University of Kentucky Medical Center
Acute Care Surgery (Blue) Service Intensive Care Unit
Mechanical Weaning/Extubation Guidelines
*Nursing Guidelines:
Pre Extubation:
Extubation:
Post Extubation:
104
Post Extubation Therapy:
Aerosol Therapy: Stridor - Upper airway stridor is best treated with nebulized racemic
epinephrine
Percussion and Postural Drainage: The indication for therapy is very narrow and specific.
A. Increased secretions or infiltrate on Chest radiograph
B. Atelectasis not responding to breathing exercises
C. Collapse of lung, lobe or segment.
In order to achieve maximum benefit, the patient must be able to cooperate with the respiratory
therapist. A large number of contraindications to this form of therapy, some of which are listed,
limit its use in the critically ill patient.
References
Boulanger BR, Kearney PA, Hale G, Coughenour JC. Mechanical Ventilation: Division of
Surgery, Division of Trauma/Critical Care and the Department of Respiratory Therapy, 4th Ed ,
2008.
105
106
107
Nosocomial Pneumonia
The Ventilator Bundle was developed to prevent adverse events that prolong the amount of time
a patient requires mechanical ventilation. The Ventilator Bundle consists of stress ulcer
prophylaxis, DVT prophylaxis, Elevation of the head of the bed (>30 degrees), antiseptic oral
care, as well as Sedation Weaning. This has been incorporated into the Trauma ICU Admission
Order Set.
Technique:
1. Confirm physician order.
2. Gather necessary items/equipment.
3. Prepare the patient for the procedure by:
a. Adequate pre-oxygenation with 100 % FiO2.
b. Ensure patient is adequately sedated.
4. Using aseptic technique (mask, gloves, cap), advance the catheter out of the protective
sheath at the opened end and gently insert protected catheter through the endotracheal
tube or tracheostomy cannula into the pulmonary tract until resistance is felt.
5. Pull the set back 3-4 cm, remove the plastic spacer, and advance inner catheter to fully
expel the distal polyethylene glycol plug.
6. Instill 20 cc of non-bacteriostatic saline.
7. Aspirate lavage sample from the airway while maintaining catheter position
8. Remove catheter from the endotracheal tube with the syringe still attached.
9. Place sample in to specimen container without contamination.
10. Send specimen to lab without delay.
11. Return to previous ventilator settings.
108
References
Tablan, O.C., Anderson, L. J., Arden N.H., et al. (1994). Guidelines for prevention of nosocomial
pneumonia. Hospital Infection Control Practices Advisory Committee. American Journal of
Infection Control, 22, 247-292.
Kollef, M., (1993). Ventilator-associated pneumonia: a multivariate analysis. JAMA, 270, 1965-
1976.
Rello, J., Ollendorf, D.A., Oster, G., et al. (2002). Epidemiology and outcomes of ventilator-
associated pneumonia in a large US database. Chest, 122 (6), 2115-2121.
Attia, J., Roy, J.G., Cook, D.J., et al. (2001). Deep vein thrombosis and its prevention in
critically ill adults. Archives of Internal Medicine, 161, 1268-1279.
Mahul P., Auboyer, C., Jospe, R., et al. (1992). Prevention of nosocomial pneumonia in
intubated patients in respective role of mechanical subglottic secretion drainage and stress ulcer
prophylaxis. Internal Care Medience, 18, 20-25.
Drakulovic, M., Torres, A., Bauer, T.T., et al. (1999). Supine body position as a risk factor for
nosocomial pneumonia in mechanically ventilated patients: a randomized trial. Lancet, 354,
1851-1857.
Kress, J., Pohlman, A.S., O’Connor, M. F., et al. (2000). Daily interruption of sedative infusions
in critically ill patients undergoing mechanical ventilation. New England Journal of Medicine,
342, 1471-1477.
Schleder, B., et al. (2002). The effect of a comprehensive oral care protocol on patients at risk
for ventilator-associated pneumonia. Journal of Advocate Health Care, 4(1), 27-30.
Reviewed: 03/05
109
110
111
Reviewed 10/08
Introduction:
Injuries, surgical wounds, and the attendant critical illness are associated with moderate to
severe pain, as well as anxiety and agitation. Intense sympathetic and parasympathetic
responses can and do occur with pain and anxiety. When pain and anxiety are not promptly and
adequately relieved, excessive adrenergic activity and hormonal changes indicative of stress
occur. These physiologic stress reactions produce a marked increase in metabolism, cardiac
workload, and oxygen consumption that may compromise tissue oxygenation in the critically ill
patient. The goal is to provide effective, sustained pain relief and sedation with minimal side
effects.
Analgesia: Morphine is the analgesic drug of choice and may be used alone or in conjunction
with a sedative. Morphine may be given intermittently or as a continuous infusion. Continuous
intravenous infusion of morphine is preferable because it avoids peaks and troughs in blood
levels leading to over and under sedation. When pain control cannot be achieved with
morphine, fentanyl is an effective, inexpensive alternative.
Agitation: Haloperidol is preferred for agitation. Frequently, escalating doses are required to
reach therapeutic levels and clinical effect. Agitation can occur from inadequate analgesia.
Therefore, make sure analgesia is adequate before treating agitation. Although rare,
haloperidol can produce extrapyramidal symptoms, neuroleptic malignant syndrome, and
cardiac events, such as prolongation of QT interval and torsade de pointes.
Anxiety: Benzodiazepines are the principle class of drugs for treatment of anxiety. Short acting
agents may be used for patients undergoing ICU procedures (central venous access, intubation,
bronchoscopy, chest tube placement, tracheotomy, etc.). Benzodiazepines should also be used
to insure deep sedation for patients requiring neuromuscular blocking agents. Benzodiazepines
should not be used as first line drugs for treating agitation. However, agitation can result from
inadequate analgesia or severe anxiety. Benzodiazepines may be used for patients who do not
respond to haloperidol or for those patients in whom haloperidol is contraindicated.
This protocol is designed to provide effective and consistent pain management as well
as effective treatment for agitation and sedation.
112
113
Nursing Care:
1. Assess patients for subjective and objective signs of pain every 4 hours and PRN.
7. Obtain new pain/ sedation orders after patient is weaned, extubated and no longer
a. receiving mechanical ventilation.
The weaning guidelines are to serve as parameters for weaning narcotics when patients have
been receiving narcotics for more than 2 weeks. These are not iron clad rules. Weaning from
narcotic analgesics may be individualized for the patient. Listed below are signs and symptoms
of withdrawal from narcotics that the critical care clinician should monitor for when weaning the
patient from narcotic analgesics.
agitation
tachycardia
tachypnea
tremors
114
fever
increased lacrimation
diarrhea
nausea and vomiting
115
Riker Sedation-Agitation Scale SAS
7 Dangerous Pulling at ETT, trying to remove catheters,
Agitation climbing over bedrail, striking staff, thrashing
side to side
References
Jacobi, Judith, et al. Clinical practice guidelines for the sustained use of sedatives and
analgesics in the critically ill adult. Critical Care Medicine. 2002;30(1): 119
Riker, RR, et al. Prospective evaluation of the sedation-agitation scale for adult critically ill
patients. Critical Care Medicine. 1999 Jul;27(7):1325-1329.
Shapiro BA, Warren J, Egol AB, et al. Practice parameters for intravenous analgesia and
sedation for adult patients in the intensive care unit: An executive summary. Critical Care
Medicine. 1995; 23: 1596-1600.
Fox CA, Mansour A, Watson SJ. The effects of haloperidol on Dopamine Receptor Gene
Expression. Experimental Neurology. 1994; 130: 288-303.
Riker RR, Fraser GL, Cox PM. Continuous infusion of haloperidol controls agitation in critically
ill patients. Critical Care Medicine. 1994; 22: 433-440.
Guinard JP, Carpenter RL, Chassot PG. Epidural and intravenous fentanyl produce equivalent
effects during major surgery. Anesthesiology. 1995; 82: 377-382.
George KA, Wright PM, Chisakuta AM. Thoracic epidural analgesia compared with patient
controlled intravenous morphine after upper abdominal surgery. Acta Anesthesiologica
Scandinavia. 1994; 38: 808-812.
116
Ready LB, Oden R, Chadwick HS, et al. Development of an anesthesiology based acute
postoperative pain management service. Anesthesiology. 1988; 68: 100-106.
Uzbay T, Akarsu ES, Kayaalp, SO. Effects of bromocriptine and haloperidol on ethanol
withdrawal syndrome in rats. Pharmacology Biochemistry and Behavior. 1994; 49(4): 969-
974.
Sun K, Quinn T, Weissman C. Patterns of sedation and analgesia in the postoperative ICU
patient. Chest. 1992; 101: 1625-1632.
Singer M, Noonan KR. Continuous intravenous infusion of fentanyl: Case reports of use in
patients with advanced cancer and intractable pain. Journal of Pain and Symptom
Management. 1993; 8(4): 215-220.
117
Revised: 11/08
The use of neuromuscular blocking agents in the treatment of critically ill patients has increased
steadily over the past two decades. Increased use is largely due to advancements in medical
technology and a better understanding of the treatment of critical illness. Up to 8% of intensive
care patients who require mechanical ventilation may require neuromuscular blocking agents.
The decision to paralyze a patient should not be taken lightly. The benefits, however, outweigh
risks when consideration is given to the efficacy of short-term use of NMBA’s. Neuromuscular
blocking agents are indicated for the treatment of patients with severe acute respiratory failure
(ARDS), marginal tissue oxygenation ( VO2 or DO2), and severe agitation which compromises
patient safety.
To assess the depth of neuromuscular blockade, a peripheral nerve stimulator using Train-of-
Four stimulation will be utilized. Routine twitch monitoring is essential to ensure a consistent
and safe level of blockade while preventing prolonged neuromuscular blockade. Clinical
indicators should be utilized to assess effectiveness of blockade (e.g. PIP < 40, improved DO2,
ICP<15). Titrate to lowest dose of NMBA to achieve defined parameters. Since pain and
anxiety cannot be assessed easily, patients must receive scheduled sedation and analgesia.
The neuromuscular blockade will be discontinued every day to determine if the patient is
receiving adequate analgesic and sedation, and to evaluate if continued paralysis is needed.
The following protocol was developed to assist the physicians and nurses in critical care to
achieve optimal management of patients requiring NMBA’s.
118
NON DEPOLARAZING NEUROMUSCULAR
BLOCKING AGENTS
Intubated/Ventilator patients Only
II. Precautions and contraindications for the use of continuous infusion of NMBA:
C. Caution when used in patients with renal, hepatic or pulmonary impairment, and
in geriatric or debilitated patients.
D. Avoid use in patients who have either experienced or have a family history of
malignant hyperthermia.
A. Vecuronium: mix 50 mg in 50 ml NS
Concentration: 1 mg/ml
Load: 0.08 - 0.1 mg/kg [use dry weight]
Infusion: 0.04 - 0.15 mg/kg/hr to maintain one detectable twitch
119
by train of four monitor [1/4]
Start infusion @ 0.04 mg/kg/hr
1. Bronchospasm
2. Flushing
3. Erythema
4. Hypotension
5. Tachycardia
6. Pruritis
7. Urticaria
8. Wheal formation
1. Heart rate
2. Cardiac output
3. Cardiac filling pressure
120
4. Mean systolic blood pressure
5. Mean arterial pressure
6. Systemic vascular resistance
C. Malignant hyperthermia
Acidosis Alkalosis
Hypothermia Hyperthermia
Hypocalcemia Hypercalcemia
Hypokalemia Hyperkalemia
Neuromuscular Disease Burns
Aminoglycosides Azathioprine
Beta-blocking agents Carbamazepine
Clindamycin Methylxanthines
Midazolam Phenytoin
Polymyxin
Procainamide
Quinidine
Tetracyclines
Vancomycin
IX. The efficacy of NMBA’s for altering oxygen consumption can be measured by
monitoring the following parameters:
1. Cardiac output/index
2. Venous oxygen saturation
3. Arterial oxygen saturation
4. Oxygen consumption
121
2. Ulnar nerve is preferred site for assessing TOF.
Facial nerve and Superficial Peroneal nerve are alternative sites if both
upper extremities are not accessible.
4. Initially a TOF should be determined every hour until three consecutive TOF elicit
the same response. A TOF can be determined every 4 hours thereafter.
If the patient is demonstrating renal or hepatic insufficiency, the TOF should be
utilized every 2 hours.
Nursing Care:
122
Non Depolarizing Neuromuscular
Blocking Agents
Intubated/Ventilator Patients Only
Patient Responds
YES NO
Maintain Adequate
Pain and Sedation Initiate Neuromuscular Blockade
Restart drip at previous rate if patient exhibits signs and symptoms of inadequate oxygenation. Notify H.O. if
the patient is severely compromised & requires a bolus.
123
124
Trouble Shooting
Guidelines for
Neuromuscular Blockade
NO YES
Change electrodes
NO YES
NO YES
Battery Good
NO YES
125
References
Jacobi, Judith, et al. Clinical practice guidelines for the sustained use of sedatives and
analgesics in the critcally ill adult. Critical Care Medicine. 2002; 30(1): 119
Riker, RR, et al. Prospective evaluation of the sedation-agitation scale for adilt critically ill
patients. Critical Care Medicine. 1999 July; 27(7): 1325-1329
Shariro BA, Warren J, Egol AB, Greenbaum DM, et al. Practice parameters for sustained
neuromuscular blockade in the adult critically ill patient: An executive summary. Critical Care
Medicine. 1995; 23: 1601-1605
Coursin DB, Meyer DA, Prielipp RC. Doxacurium infusion in critically ill patients with atracurium
tachyphlaxix. American Journal of Health-System Pharmacy. 1995; 52: 635-638
McCoy EP, Mirakhur RK, Connolly M, et al. The influence of the duration of control stimulation
on the onset and recovery of neuromuscular block. Anesth Analg. 1995; 80: 364-367
Gooch JL, Suchyta MR, Balbierz JM, et al. Prolonged paralysis after treatment with
neuromuscular junction blocking agents. Critical Care Medicine. 1991; 19(9): 1125-1131.
Helbo-Hansen HS, Bang U, Nielsen HK, et al. The accuracy of Train-of-four monitoring at
varying stimulating currents. Anesthesiology. 1992; 76(2): 199-203.
Jarpe MB. Nursing care of patients receiving long-term infusion of neuromuscular blocking
agents. Critical Care Nurse. 1992; 10: 58-63.
Davidson JE. Neuromuscular blockade. Focus on Critical Care. 1991; 18(6): 512-519.
Clarens DM, Kelly KJ, Gilliland SS, et al. A retrospective analysis of long-term use of
nondepolarizing neuromuscular blocking agents in the intensive care unit, and guidelines for
drug selection. Pharmacotherapy. 1993; 13(6): 647-655.
126
Intra-Abdominal Hypertension and Abdominal Compartment Syndrome
Assessment and Monitoring Guidelines
The abdominal cavity can be considered a single cavity and change in the volume of contents
will elevate abdominal pressures. Abdominal Compartment Syndrome (ACS) is a condition in
which the increased pressure in the anatomic space results in organ dysfunction. Undetected
increases in intra-abdominal pressure (IAP) can be life threatening. Identification of patients at
risk is essential to prevent hemodynamic and respiratory compromise from undetected ACS.
ACS is preceded by intra-abdominal hypertension (IAH) and organ dysfunction may precede
development of ACS.
Definition of ACS: Intra-abdominal pressure (IAP) > 20 mmHg (with or without an APP < 60
mmHg) in a minimum of three standardized measurements taken four to six hours apart plus at
least one new end-organ failure.
Acute
A. Intra-abdominal Hemorrhage
Post resuscitation visceral hemorrhage
Hypothermic or consumptive coagulopathic bleeding
Rupture of abdominal aortic or visceral artery aneurysm
Post traumatic intra-abdominal hemorrhage
B. Retroperitoneal Hemorrhage
Blunt trauma (i.e., pelvic fracture, kidney laceration)
Hemorrhagic Pancreatitis
Ruptured abdominal aortic aneurysm
C. Accumulation of Fluid/Visceral Swelling
Septic shock
Peritonitis (i.e., perforated viscus, postoperative abscess)
Paralytic ileus
Bowel obstruction
Mesenteric venous thrombosis
Mesenteric ischemia/reperfusion
Pancreatitis
D. Other
Tension pneumoperitoneum
Intra-abdominal packing
Chronic
A. Ascites
B. Pregnancy
C. Large abdominal tumor/ovarian mass
127
II. Physiologic Consequences
Cardiopulmonary Effects
Increased IAP increases intra-thoracic pressure (ITP) which impedes venous return and
causes a number of physiologic derangements.
A. Pulmonary
Decreased compliance (see higher peak airway pressures)
Increased inspiratory pressure
Hypercarbia (decreased ventilation)
Hypovolemia (compresses SVC, decreasing preload = decreased CO)
Respiratory Acidosis (decreased FRC and TV = decreased ventilation)
Increased pulmonary vascular resistance (increases pulmonary shunt and
increases work on heart to generate same CO)
B. Cardiac
Decreased ventricular compliance (requires increased preload for same CO)
Increased CVP, PWP, PAP (Falsely elevated when euvolemic!)
Diminished venous return (compresses SVC/IVC)
Tachycardia (decreased preload, need increased HR to keep same CO)
Decreased cardiac output (seen when compensatory mechanisms fail)
Increased SVR
* Venous stasis may increase risk of DVT/PE
Renal Effects
Increased IAP compresses the inferior vena cava and renal veins. Direct extrinsic pressure on
the kidney creates a circumferential constriction. The combination of direct trauma,
hypoperfusion and venous backpressure can create an intra-renal compartment syndrome. As
a consequence, urine output diminishes.
Neurological Effects
By increasing ITP, increased IAP impedes venous outflow from the cerebral circulation.
ICP
CPP
128
Gastrointestinal/Hepatic/Wound Healing
Increased abdominal pressure reduces blood flow to the abdominal viscera.
celiac and portal blood flow
mucosal blood flow
fascial blood flow- (increases risk of wound infection and dehiscence)
bacterial translocation
Please notify physician if IAP is > 12, or per physician’s orders. Profound physiologic
derangements can occur with IAH reforcing the need to recognize and treat IAH early
before ACS develops.
A. Supplies Needed
129
B. Guidelines
References
Scalea TM, et al. Increased Intra-abdominal, Intra-thoracic, and intra-cranial pressure after
severe rain injury: multiple compartment syndrome. J. Trauma 62(3):647-656, March 2007.
Malbrain ML, et al. Results from the International Conference of Experts on Intra-abdominal
Hypertension and Abdominal Compartment Syndrome. Intensive Care Medicine
2006;32(11):1722-1732 & 2007;33(6):951-962
Sugrue M. Abdominal compartment syndrome. Curr Opinion in Critical Care
2005;11:333-8.
Watson, RA, Howdieshell, RT. Abdominal Compartment Syndrome. Southern
Medical Journal. 1998; 91(4):326-332
Harrahill M. Intra-abdominal pressure monitoring. Journal of Emergency Nursing.
1998;24(5):465 – 466
Iberti, TJ, Kelly KM, Gentili, DR, et al. A simple technique to accurately determine Intra-
abdominal pressure. Critical Care Medicine. 1987;5(12):1140 – 1142
Iberti, TJ, Lieber CE, Benjamin, E. Determination of Intra-abdominal Pressure
Using a Transurethral Bladder Catheter: Clinical Validation of the Technique. Anesthesiology.
1989;70(1):47 - 50
Gallagher, John J. Ask the Experts. Critical Care Nurse. 2000;20(1):87 – 91.
Schein, M, Wittmann, DH. The Abdominal Compartment Syndrome Following
Peritonitis, Abdominal Trauma, and Operations. Complications in Surgery 1996; 15(5)
130
Guidelines for Operative Procedures in the Intensive Care Unit
I. Operative Procedures
A. Non-Emergent Procedures
1. Percutaneous Tracheostomy
2. Percutaneous Endoscopic Gastrostomy
3. Planned relaparotomy for the management of intra-abdominal sepsis
4. Planned relaparotomy for pack removal
B. Emergent Procedures
Note: Indicated operative procedure to sustain life when patient condition or time precludes
transport to the operating room.
1. Compartment Fasciotomy
2. Bedside Decompressive Laparotomy
B. Emergent Procedures
1. Surgical Attending
2. Surgical Resident
3. OR Safari team
4. Anesthesia resident
5. ICU RN
1. Electrocardiogram
2. Pulse Oximetry
3. Arterial Blood Pressure
If indicated:
4. Train of Four twitch assessment
5. End-tidal CO2
Note: Adherence to strict nursing policies and procedures for monitoring will be maintained at
all times. Refer to Appendix A and Appendix B.
131
IV. Administration of Medications
1. Surgical Attending
2. Surgical resident
3. Anesthesiologist
4. RN
Note: If anesthesia is to be administered, the RN will not administer.
RN will follow guidelines set forth for conscious sedation – See Appendix A and Appendix B
V. Supplies Needed
A. Non-Emergent Procedures
B. Emergent Procedures
1. Overhead OR light
2. See Appendix G for Compartment Fasciotomy
3. See Appendix I for Bedside Decompressive Laparotomy
4. See Appendix H for Minor OR Tray Supplies
Note: Supplies provided by OR Sterile per Safari Team
A. Non-Emergent Procedures
B. Emergent Procedures
References
Mayberry, J.C. (200) Bedside Open Abdominal Surgery. Utility and Wound Management.
Critical Care Clinics, 16 (1), 200, 151-172.
Porter, J.M., Ivatury, R. R., Kavarana, M. & Verrier, R. (1999). The Surgical Intensive Care Unit
as a Cost-Efficient Substitute for an Operating Room at a Level I Trauma Center. The American
Surgeon, 64, 328-330.
Waydhas, C., Schneck, G., & Duswald, K. H. (1995). Deterioration of respiratory function after
intra-hospital transport of critically ill surgical patients. Intensive Care Medicine. 21, 784-789.
132
Smith, I. Flemming, S., & Cernaiano, A. (1990) Mishaps during transport from the intensive care
unit. Critical Care Medicine, 18, 278-281.
Indeck, M., Peterson, S., Smith, S., et al. (1988). Risk cost and benefit of transporting ICU
patients for special studies. Journal of Trauma, 28, 1020.
Stiegmann, G., Goff, J., Van Way, C., et al. (1988). Operative versus endoscopic gastrostomy in
the intensive care patient. Critical Care Medicine, 16, 62-63.
Toursark, B., Zweng, T. N., Kearney, P.A., et al. (1994). Percutaneous Dilatational
tracheostomy: Report of 141 cases. Annals of Thoracic Surgery, 57, 862-867.
Hill, B. B., Zweng, T.N., Maley, R.H., et al. (1996). Percutaneous dilatational tracheostomy:
Report of 356 cases. Journal of Trauma. 32, 133-140.
Hwang, T.L., Chiu, C.T., Chen, H.M., et al. (1995). Surgical results for severe acute pancreatitis:
Comparison of the different surgical procedures. Hepatogastroenterology, 42, 1026-1029.
Tsiotos, G. G., Smith, C. D., & Sarr, M. G., (1995). Incidence and management of pancreatic
and enteric fustulas after surgical management of severe necrotizing pancreatitis. Archives of
Surgery, 130, 48-52.
Brant, C. P. Prieve, P. P., Eckhauser, M. L. (1993). Diagnostic laparoscopy in the intensive care
patient. Avoiding the nontherapeutic laparotomy. Surgical Endoscopy, 7, 168-172.
133
HIGH DOSE VITAMIN C (Ascorbic Acid) Protocol
IV Meds
Calculation box
PHARMACY INSTRUCTIONS
1. 66mg/kg/hr x KG -> XX mg/hr/25mg/ml = Rate ml/hr
134
ICU Anemia Management Protocol (Adults)
Patients commonly suffer from anemia on admission to the intensive care unit (ICU).
Additionally, anemia frequently develops or worsens during the course of a patient’s ICU
stay. Anemia can cause prolonged ICU stays, increase the costs of healthcare, and lead
to other negative patient outcomes. Anemia is usually treated in these patients but not
always in a cost-effective and resource-sensitive manner. In recognition of the ongoing
shortage in the nation’s blood supply, the cost of agents used to treat anemia, the
potential harmful effects of transfused blood products and the need to improve patient
outcomes, the Pharmacy and Therapeutics Committee recently approved the Adult ICU
Anemia Management Protocol.
Prevention
Minimize phlebotomy (avoid daily lab draws, obtain all blood needed at once, use
specimen tubes that can be used for multiple labs, add tests to blood already in
the lab when appropriate)
Adequate nutritional support
Transfusion of PRBCs
The protocol defines guidelines for blood transfusions. To receive PRBCs, patients must
meet one of three criteria presented in the Table. After the transfusion of one unit, the
average 70 kg adult will experience an increase in the Hgb by 1 g/dL. The goal after
receiving a transfusion is a Hgb 7 g/dL (Hct 21). If clinically indicated to monitor
response and further bleeding, blood counts should be collected 30 minutes after the end
of the infusion. Routine post-transfusion hematocrits are not mandatory. The most blood-
economical manner of monitoring hematocrit is with blood gas panels, which require only
1cc of blood (venous or arterial), are cost neutral compared to specific hemograms or
hematocrits and for which results are obtained more quickly by virtue of processing on the
blood gas analyzer.
Since 2001, almost all red blood cell units prepared and stored in the United States are
leukocyte-reduced.
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If the decision to transfuse PRBCs is made then 1 U should be given first to monitor for a
response as measured by hemodynamic status and measurement of Hg/HCT for
appropriate transfusion response. If an appropriate clinical response is not seen then
increments of 1 U of transfusion with monitoring for clinical/laboratory response.
Adapted from, “Hébert PC et al. Controversies in RBC Transfusion in the Critically Ill.
Chest. 2007. May;131(5):1583-90.
Adjunctive Medications
Beta blockade to keep the heart rate less than 90 should be considered in patients with
known or suspected heart disease. The preferred agent is metoprolol starting at 12.5
mg orally/per tube twice daily, and titrated as tolerated to a maximum dose of 100 mg
twice daily. The patient must be adequately resuscitated and have appropriate pain
control before altering their physiology with beta blockade. Indicators of adequate
resuscitation are clearance of acidosis (normal lactate, base deficit and serum HCO3-),
normotension, adequate urinary output, and ScVO2, ScVO2 or StO2).
Revised 11/08
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SECTION 5: ICU and FLOOR PROTOCOLS
The goal of the Trauma/Emergency Surgery service is to provide cost-effective, high-quality patient
care. In order to achieve this goal a customer service model will be implemented. The physician/nurse
team will make excellent patient care the focus of their effort. This will be accomplished by the
development of ―best practice‖ models using evidence based clinical data. This will allow the
physician/nurse team to direct ancillary services in the most cost-effective manner for excellent patient
care.
Mobilization
There is clear, irrefutable evidence that extended bed rest is harmful to patients. Supine position and
lack of mobilization leads to reductions in pulmonary functional residual capacity, atelectasis,
diminished cough, and accumulation of dependent lung water. This makes the patient more prone to
pulmonary complications which are the most frequent cause of admission or readmission to the ICU in
the Medical Center. Bed rest also leads to rapid loss of muscle mass leading to de-conditioning which
may increase hospital length of stay, lengthen rehabilitation, or create the need for rehabilitation that
would have not been otherwise needed. Patients at bed rest are more prone to pressure ulcers, bowel
dysfunction, and venous thromboembolic disease.
A primary focus of patient care should be early mobilization of the patient. Weight-bearing status
should be determined within 24 hours of admission to the hospital. Ambulation of the patient should be
an immediate goal. If there are limitations on weight-bearing these should be identified and appropriate
resources (physical therapy, equipment, etc) should be applied immediately. At the very least, patients
should be out of bed and placed in a chair. This does not mean bringing the bed into a sitting position
but actually getting the patients out of the bed into a chair. If traction or hemorrhage risk mandates bed
rest, the patients should be nursed with the head of the bed elevated at least 30 degrees. When spine
precautions are in place the patient should be placed in reverse trendelenburg.
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Respiratory Function
Respiratory therapy is essential for acutely ill and injured patients. Many of our patients have co-
morbid lung disease and/or a history of heavy tobacco use. Lung function is further compromised by
bed rest, obesity, chest wall trauma, pain, surgical incisions, chest tubes, or via the use of cervical
collars, back braces, and/or traction. It is unrealistic to expect respiratory therapy service to assume
this task for most patients. Pulmonary toilet should be a major goal for excellent nursing care.
Tracheostomy
Tracheostomy is frequently required for the management of airway, pulmonary toilet, and/or respiratory
failure in patients with complex critical illness and/or injury. The vast majority of these are performed
percutaneously although some are still placed using the older, but still reliable, open surgical technique.
Regardless of placement technique, complications, daily tracheostomy tube/site care, and
downsizing/decannulation are the same.
Complications
Dislodgement: The most common early and lethal complication of tracheostomy is tube dislodgement.
In order to prevent this complication the tracheostomy tube is secured to the skin with sutures and fixed
in place with a secure neck strap. Sutures often give a false sense of security and will not in and of
themselves eliminate dislodgement. The most important defense against early dislodgement is a
secure neck strap and strict avoidance of undue tension and/or torque on the fresh tracheostomy. It
takes about four days after insertion to develop a mature tract. After four days, dislodgement rarely
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results airway compromise and the tube can simply be reinserted through the neck. Prior to four days,
airway loss is likely and the patient may require endotracheal intubation to reestablish the airway. This
is a true emergency that requires the immediate attention of the surgical house staff. The patient can be
temporized with 100% oxygen delivered via bag valve mask and gentle occlusion of the tracheostomy
site.
Plugging: The most common intermediate complication of tracheostomy is mucous plugging. This will
be the most frequent problem encountered with ward patients. Secretions build up in the tracheostomy
tube lumen leading to sudden occlusion and airway compromise. Virtually all of the tracheostomy
tubes in use at UKMC have a removable inner cannula. The routine practice of maintaining cuff
deflation and routine inspection/cleaning of the inner cannula will prevent this complication.
Stenosis: Tracheal stenosis is a late complication of tracheostomy. Virtually all tracheostomies (85%)
are associated with some degree of tracheal stenosis. Only 1-2% of patients develop critical stenosis
that compromises the airway and produces clinical symptoms. The duration of endotracheal intubation
(> 11 days) prior to tracheostomy, technique used (open > percutaneous), higher placement (between
rings 1-2 vs. 2 or lower), and smaller airway (children, females) all increase the rate of clinically
significant tracheal stenosis. Almost all clinically significant stenosis occur within twelve weeks of
tracheostomy. The hallmark of clinically significant tracheal stenosis is respiratory distress and/or
stridor and wheezing when the tracheostomy tube is plugged or removed. If plug removal or
tracheostomy tube reinsertion alleviates symptoms the tube should remain in place until a diagnostic
evaluation can be performed.
Tracheal-arterial fistula: Small amounts of bleeding may occur simply from the irritation of suctioning,
site care and/or the tube itself. A rare but lethal complication of tracheostomy is tracheal-arterial fistula
that occurs from erosion of the tip of the tracheostomy tube into the great vessels of the upper thorax.
The hallmark of this complication is a ―herald bleed‖ defined as a moderate to large amount of bleeding
that stops spontaneously. If bleeding persists or produces airway compromise, the endotracheal tube
cuff can be inflated to tamponade bleeding and maintain the airway. Herald bleeding requires
immediate investigation!
Infection: Tracheostomy site infection is exceedingly rare. The presence of purulent, foul–smelling
secretions accompanied by an expanding zone of erythema establishes the diagnosis. Site care and
appropriate antimicrobial therapy are effective in controlling this complication.
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removed after six days. Tracheostomy bypasses the normal humidification provided by the
oro/nasopharynx so patients are prone to evaporative water loss and desiccation of the airway mucosa.
Humidified air or oxygen (if required) should be used at all times to prevent the latter complications.
Intravenous Access
Three quarters (75%) of all hospital bactermia events are associated with intravenous catheters! There
is a general hospital wide practice to ―heparin lock‖ and keep both peripheral and central venous
catheters. Keeping multiple IV sites is simply not a good practice. Each and every IV site represents a
potential nosocomial infection site for patients. Both insertion technique and indwell time influence
subsequent thrombophlebitis. Many catheters are placed under less than ideal conditions and should
be removed as soon as possible. In all but the most unusual circumstances, a patient will require a
single functioning IV access site. Proper inspection and site care should be used to maintain function
and sterility. All other intravenous access sites should be removed.
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Wound Care
Wound care is much easier than most physicians and nurses think. The process has become
unnecessarily complicated, confusing, and expensive. The simple answer is soap, water, and gentle
handling of tissues. Astringents (peroxide, betadine, acetic acid, alcohol, Daken’s, etc.) should rarely if
ever be used in a wound. There is a widely held misconception that more frequent wound care
somehow makes wounds heal faster. This is simply not the case. Keeping wounds clean, moist, and
covered allows the body to heal the wounds considerably faster. Astringents, frequent dressing care,
and overzealous packing are more often responsible for delays in healing.
Wound Classification
Clean Wounds - These are surgical incisions that follow an elective surgical procedure that does not
involve the aerodigestive tract. Examples would be neurosurgical procedures, vascular procedures,
hernias, most elective orthopedic procedures.
Clean contaminated wounds – These are surgical incisions that follow an elective surgical procedure
that crosses the aerodigestive tract. Examples would be most ENT procedures, operations on the
gastrointestinal tract, or operation on the lung.
Contaminated wounds – These are incisions that follow an emergent surgical procedure where there is
obvious or potential infection. Examples would be perforations of the GI tract, strangulated hernias,
complicated soft tissue infections, open fractures.
Dirty wounds – This is really a matter of degree. The difference between contaminated and dirty
wounds is really the degree of contamination. Complex wounds with large devitalized areas, gross
fecal contamination, large amounts of purulent material, dirt, foreign bodies etc. are usually classified
as dirty.
Important Definition
Dehiscence refers to separation of the wound edges. Dehiscence can further defined as involving the
skin and subcutaneous tissue (superficial) or extending to the deeper layers (fascial dehiscence).
Evisceration refers to the protrusion of visceral contents through the wound. Not all dehiscence has
evisceration but by definition all eviscerations have dehiscence.
Wound management
Wounds are managed in one of three ways:
1. Primary closure of the skin and subcutaneous tissues (most wounds)
2. Delayed primary closure. Wounds are left open initially. The skin is then closed primarily
between day 3 and 4. Bacterial counts are lowest in the wound at this point and delayed primary
closure has the greatest success.
3. Healing by secondary intention. This technique applies for most open wounds.
Closed wounds
Wounds that have been closed primarily will seal within 36 hours. After that point it is very unlikely that
environmental contamination would compromise the wound. The general rule is to leave the surgical
dressing on for 24 hours. After that point the wounds can be covered with a light dry dressing to absorb
minor drainage, prevent irritation and for patient comfort. These wounds should be carefully inspected
at least once a day for signs of infection (redness, swelling, excessive tenderness, purulent drainage).
The subjective patient complaint of wound pain (fever may or may not be present) that increases or is
out proportion to wound size is often the earliest sign of surgical wound infection.
Open wounds
Contaminated or dirty wounds are often packed and left open. The main clinical reason for this practice
is the high incidence of wound infection if the wounds are closed. The wounds are generally packed
tightly to achieve hemostasis after the initial operative procedure. Unless there is a planned return to
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the operating room for exam under anesthesia, further debridement, and irrigation, these dressings
should be taken down and the wound examined at 24 hours. The first dressing change can be quite
painful and provisions should be made for adequate analgesia prior to proceeding. Most of the pain
emanates from the densely innervated wound edge and care should be taken in this area. If the
dressing is adherent gentle wetting with saline will facilitate removal and reduce discomfort. ―Clean
wound‖ care rather than ―sterile technique‖ should be the standard practice. At the first dressing
change the decision can be made to initiate saline wet to dry dressing or application of a vacuum
dressing. At this point a decision can also be made about washing/showering the wound with soap and
water. Dressing changes need only be once or twice a day and packing of the wound should be gentle.
Saline irrigation of the wound base is permissible. As mentioned previously, astringents should be
avoided and every effort should be made to avoid wound dessication by irrigating the wound in
between dressing changes if necessary. Regarding dressing fixation, the skin should be protected from
tape adhesives by using duoderm and Montgomery straps or by application of Bandnet dressing
(preferred). Absolutely every effort should be made to simplify wound care prior to discharge. Patients
should be given clear instructions on clean wound care, showering should be encouraged. When
possible, wounds can/should be dressed with tap water rather than sterile saline which is considerably
more expensive and unnecessary for most wounds.
Wound infection
The earliest and most frequent sign of wound infection is excessive wound pain and tenderness. Low
grade fever, wound redness, and drainage often appear later and can be easily seen with a good exam
and dressing change. Wounds should be opened in the affected area to allow drainage, irrigation, and
gentle packing just like in open wounds. Wound culture and antibiotics are totally unnecessary except
in rare circumstances such as when patients exhibit signs of systemic illness and/or there is prosthetic
material in the wound. WARNING! When dealing with abdominal wall wounds, drainage may indicate
deep wound problems such as fascial failure and/or evisceration.
Gastrointestinal Tract
There is a widely held misconception that the gastrointestinal tract in quiescent following illness, injury,
and/or surgery. The gut plays an active role in overall host defenses, gastrointestinal stress ulceration,
and systemic inflammation. The historical term attached to the clinical problem of post operative gut
dysfunction was ― paralytic ileus‖. This has been shortened in modern medical terminology to ―ileus.‖
The traditional clinical practice is to withhold oral intake and maintain nasogastric decompression until
there was clinical evidence indicating return of bowel function (passage of flatus, bowel movement, or
audible bowel sounds). This practice is outdated and not consistent with what is currently known about
bowel function in illness. The stomach and small bowel function very well following illness, injury, and
/or operative intervention unless there has been mesenteric ischemia or long standing obstruction. The
actual root cause of the clinical entity referred to as ―ileus‖ is delayed return of colonic function.
Although ingrained in our medical terminology, ―ileus‖ is a misnomer and the proper term to use is
colonic pseudo-obstruction.
There are a number of clinical practices that either exacerbate or contribute to colonic pseudo-
obstruction. Chief among these are bedrest, narcotic administration (particularly epidural catheters), as
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well as fluid & electrolyte abnormalities. Depending on the clinical circumstances, the clinician also
needs to consider other contributing factors such as fecal impaction, resolving peritonitis, intra-
abdominal abscess, pneumonia, wound infection, retroperitoneal hematoma, and pseudomembranous
colitis. Mesenteric ischemia and early mechanical bowel obstruction, although rare, must also be
considered in the differential diagnosis.
For most patients, early mobilization, judicious use of narcotics, as well as attention to fluid &
electrolytes can mitigate or prevent pseudo-obstruction. Routine use of an effective bowel regimen
and/or early enteral nutrition is also effective depending on the patient and clinical circumstances.
The main risk to the patient with pseudo-obstruction is colonic ischemia and/or perforation which are
dependent upon the degree of colonic distention. Perforation/ischemia is much more likely when
colonic/cecal diameter is > 11cm. Under these circumstances, more aggressive management is
warranted. For most patients, the treatment of pseudo-obstruction is relatively straightforward. Bowel
rest, hydration, and correction of electrolytes are essential. Narcotics should be reduced as much as
possible. Depending on the clinical situation, other treatable contributing factors need to be rectified or
excluded. Nasogastric tubes are completely unnecessary for the vast majority of patients because they
are not effective in reducing colonic distention. NG tubes should be withheld unless the patient is
vomiting and/or has evidence of gastric distention on X-ray. A combination of stool softeners and
cathartics accompanied by a prokinetic agent (metaclopramide) are usually effective. Cathartics and
prokinetic agents are more effective when given orally but other routes of administration may be
necessary depending on the clinical situation. Rectal stimulation with a suppository and/or enema may
also produce results. For refractory patients or those with significant colonic distension, a
parasympathomimetic agent (neostigmine) can be administered IV with excellent results. Routine use
of neostigmine is precluded by side effects such as bradyarrythmias, bronchorrea, and diaphoresis.
Ideally, patients should be monitored during drug administration particularly if they have known cardiac
disease. Decompressive colonoscopy which is both diagnostic and therapeutic may be required for
patients with significant colonic distention.
Enteral Nutrition
Not all patients require early enteral nutrition. Well nourished patients who sustain mild to moderate
injury or those undergoing elective operations tolerate up to seven days of fasting with little or no
adverse consequences. However, patients with documented pre-injury or pre-operative malnutrition as
well as those patients with complex critical illness/injury clearly benefit from early enteral nutrition. In
fact, the evidence is clear and irrefutable. Infectious complications are significantly reduced in patients
who receive early enteral nutrition. Early enteral nutrition also maintains gut integrity, reduces the risk of
gastrointestinal stress ulceration, and increases the rate of wound healing.
Access routes
The main difficulty with early enteral nutrition is achieving and maintaining a reliable feeding access.
Gastric feeding is well tolerated and most patients can be fed in the stomach. Unfortunately tolerance is
an issue for some patients, monitoring may be difficult and the aspiration risk is higher than that for
post-pyloric tubes. As the patient improves clinically, aspiration risk declines and the need for post-
pyloric access diminishes. Three approaches are used to establish feeding access; nasoenteral feeding
tube, surgical jejunostomy, and percutaneous endoscopic gastrostomy (PEG). For the vast majority of
patients, a nasoenteral feeding tube is a safe, temporary access. These can be placed blindly, via
endoscopy, fluoroscopy, or at the time of surgical intervention. Since these tubes frequently become
dislodged they are often secured in place with a bridle. Nasoenteral feeding tubes are not a reliable
long term access and should be replaced with a jejunostomy or gastrostomy tube. When a patient has
significant foregut pathology, a surgical jejunostomy can be placed. This allows enteral feeding to
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proceed in the absence of an intact/functioning foregut. The most frequently utilized long term feeding
access is the PEG. This is a safe, effective way of delivering enteral nutrition for most patients.
Vomiting/Aspiration
Vomiting and/or aspiration may be the first sign of feeding intolerance. In the awake patient, complaints
of nausea will precede the event, so don’t ignore this complaint. This manifestation is more likely in
patients being fed in the stomach via PEG or nasoenteral feeding tube. Remember that post-pyloric
feeding reduces but does not eliminate vomiting/aspiration risk. The most prudent course of action is to
hold feedings. Depending on the clinical suspicion for aspiration, an evaluation by a physician is
warranted. Vomiting will usually dislodge a nasoenteral feeding tube, so replacement and/or
verification of position is warranted.
Abdominal distention
Frequently overlooked, abdominal distention and bloating are the earliest and most reliable signs of
intolerance. All patients receiving enteral nutrition should be evaluated daily. Not all patients require
and intervention but this should be noted and brought to the attention of the physicians caring for the
patient. Acute and/or significant distention may indicate mesenteric ischemia or colonic pseudo-
obstruction.
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Diarrhea
Diarrhea is probably the most frequent complication of enteral nutrition. Oddly enough tube feeding is
usually not the cause. Sorbitol containing medications are frequently to blame so a review of the
medication record is warranted. If indicated, clostridium difficile colitis should be excluded. Higher tube
feeding rates may produce diarrhea so a change in rate may be warranted. Anti-diarrhea agents can
be utilized if the problem persists. Changes in formula can be made. If the volume of stool exceeds
500-1000cc per day then holding the feeds may be necessary.
Tube maintenance
Enteral access tubes are expensive and vital to patient care. Every effort should be made to maintain
patency and protect against dislodgement.
PEG
These tubes can and do become clogged and or dislodged. One of the most important aspects of daily
PEG care is to assess the tube site and determine tube depth. Nurses should pay close attention to
the insertion site for redness, swelling, and/or tube feeding reflux. Following placement, PEG tubes are
secured in place using a silastic bumper. These bumpers are applied loosely to maintain the PEG at
the original depth of insertion which is charted in the endoscopic procedure note. Each PEG tube has a
centimeter marker on the side. The general depth for most patients is between 3-6 centimeters. The
depth at insertion should be recorded on the nursing assessment. If the depth marker is <3cm or >
6cm or there is tube feedings refluxing through the insertion site, feedings should be held and the
physician notified immediately. Remember, a sudden change in PEG feeding tolerance accompanied
by abdominal distention can indicate PEG tube migration or dislodgement.
Diarrhea
Not all liquid stools constitute diarrhea. Diarrhea is defined as frequent loose stools exceeding 1000cc
per day and/or producing fluid/electrolyte abnormalities. The clinical objective is to identify and remove
treatable causes of diarrhea. Medications are probably the most frequent cause of diarrhea. Drugs that
produce diarrhea such as prokinetic agents, oral macrolides, cathartics, and sorbitol containing elixirs
should be eliminated when possible. Adjustments in tube feeding rate and/or formula change may be
required. Clostridium difficile colitis should be excluded or diagnosed and treated. Diarrhea may
accompany a high-grade fecal impaction. Diarrhea may also be the only manifestation of intra-
abdominal infection. Surgical resection of the small or large bowel (ileocecal valve in particular) may
produce post-operative diarrhea. Diarrhea may follow resolution of pseudo-obstruction or surgical relief
of a mechanical small bowel obstruction. In general, treatment is defined by the cause. Medications
should be changed/eliminated. Fecal impaction should be cleared. With the exception of C. difficile
colitis, symptomatic relief can be provided with anti-diarrhea agents such as combinations of lomotil,
Imodium, paregoric, and narcotics.
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C. difficile bacterium. The disease can be produced by as little as one dose of antibiotics and most
often follows single dose antimicrobial administration for perioperative prophylaxis. There is some data
to suggest that mechanical bowel preparation/cleansing may produce the disease as well. The most
frequent offending antimicrobials are cephalosporin (Rocephin), Clindamycin, ampicillin, and
fluroquinolones such as levaquin. The primary manifestation of the disease is diarrhea which may
occur up to 14 days after the last antibiotic administration. Occasionally the patients will have
abdominal pain and distention. Rarely they will present with or have constitutional symptoms such as
fever and systemic toxicity that accompany the diarrhea and abdominal pain. The diarrhea associated
with the disease is quite distinct. The frequent passage of small amounts of foul smelling liquid stools
should raise clinical suspicion. The diagnosis is easily established by sending a stool specimen for
toxin assay. Cultures are of no value because the bacteria are normal resident flora in many patients.
First line therapy is metronidazole (flagyl) administered 7-10 days via the enteral route. Intravenous
flagyl is effective for patients who will not tolerate the oral route. Vancomycin given enterally is
reserved for patients who present with severe disease and/or fail on flagyl. Empiric therapy is
appropriate after stool cultures have been obtained and can be stopped if the toxin assay is negative.
Endoscopy to identify pseudomembranes is occasionally required to establish the diagnosis. Rarely, a
patient will develop toxic megacolon and require emergent surgical intervention.
Constipation
There is overlap between constipation and colonic pseudo-obstruction. Abdominal pain, distension,
nausea, and vomiting accompanied by absence of a bowel movement for more that several days are
the most common symptoms. Unfortunately these symptoms are identical to colonic pseudo-obstruction
so the diagnosis is difficult in the hospitalized patient. Narcotics, bedrest, dietary changes, fluid &
electrolyte abnormalities, particularly dehydration all predispose the patient to constipation. The key is
prevention. Early mobilization of the patient and adequate hydration are essential. Patients who require
pain medications should receive stool softeners as a routine. Once constipation develops attention
should be turned to correcting the problem. Fecal impaction should be excluded by rectal exam.
Remember that diarrhea may be a manifestation of fecal impaction. Stool softeners alone are usually
not enough. Unless the patient is vomiting, oral cathartics should be tried initially. Oral dulcolax tablets
and/or milk of magnesia (MOM) can be administered along with oral metaclopramide (reglan) 10-20mg.
Osmotic agents such as sorbitol or phosphate of soda are also effective particularly if administered with
dulcolax. These regimens can be repeated. If the patient is vomiting or does not respond to oral
therapy, digital rectal stimulation with a dulcolax suppository with or without enemas can be employed.
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5th Floor Practice Standards
Mobilization Standards:
Patient will be out of bed to chair three times a day. Patient will also ambulate in hall
three times a day if patient is ambulatory.
1. For non-ambulatory patients: OOB three times a day. Bending the bed is not
equivalent to getting them out of bed. Getting them out of bed requires patient effort and
therefore respiratory and other muscle work. For bedridden patients, active/passive ROM will be
performed every 12 hours along with turning every 2 hours to decrease soft tissue injury.
Every patient should have incentive Spirometer when admitted unless specifically
contraindicated. Incentive Spirometer will be included in admission kit by nursing tech.
Incentive Spirometer will be used by every patient 10 times every hour while awake.
Turn/cough/deep breathe will be done for all non-ambulatory patients unless
contraindicated every 2 hours. The surgical literature clearly shows that pulmonary
complications are reduced by any method of pulmonary care that leads to maximum voluntary
ventilation on the part of the patient. Coughing, deep breathing, exertion, and incentive
Spirometer use should be encouraged and stressed.
IV Standards:
Nurse will have MD order prior to IV initiation.
1. Unless an emergency situation arises.
IV’s will not be heparin-locked for procedures.
1. Unless specifically ordered by MD
Each patient will have only one IV access.
1. Unless specifically ordered by MD.
2. Unless additional IV sites are needed for blood transfusions or non-compatible
drugs.
RN will attempt two IV sticks, if unsuccessful; will ask for assistance from one other RN.
If IV access is not acquired, RN will notify MD and suggest PICC or deep line placement.
IV’s and IV tubing will be changed every three days and more often as needed by night
shift RN’s.
RN will assess IV for patency, infiltration, infection every 8 hours and with medication
administration.
Patient may be heparin-locked with adequate PO intake, and IV will be d/c’d unless
patient has medication requiring IV, if so the IV will be heparin locked.
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Flush with 10cc of NS and 2cc of heparin in a 10 cc syringe every 12 hours.
After blood draw, lipid administration, blood transfusion, PICC line will be flushed with
20cc of NS.
RN is able to discontinue PICC line: pull line, measure length, assess condition (tip of
catheter) and document findings.
Diet Standards:
Diet will be as tolerated. (Progressing diet will be discerned by RN).
1. Unless specific diet is ordered by MD.
When a procedure is ordered, MD will specify post-procedure diet or NPO status.
Bath/Shower Standards:
Baths will be divided between days/evenings/nights.
1. Baths that are appropriate for night shift will be determined by the RN.
Appropriate patients will have a bath/shower every day.
1. If patient refuses bath, RN will be notified by NCT.
Oral care must be offered by NCT 3x’s/day (with am care, after lunch and after supper).
Oral care on NPO patients must be done at least 3x/day and more as needed (every 8
hours).
1. If patient refuses, RN will be notified by NCT.
Hair care will be done when blood or other debris is in hair.
Hair care will be offered by NCT when appropriate.
Male patients will be shaved daily unless patient has facial hair that they desire to leave
unshaved.
Denture care will be offered in morning and at night by NCT.
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Foley Catheter Standards:
RN must have MD order for foley catheter to be initiated.
Foley catheter will be assessed for kinks and emptied every 4 hours.
Foley catheter will be discontinued the night before discharge.
1. Unless patient is to be discharged with foley catheter in place.
For total joint patients, foley catheter will be discontinued 2 days post-operative.
In and Out catheterization may be carried out by RN if patient has not voided 6-8 hours
after foley catheter has been discontinued. RN may repeat in and out catheterization a
second time. If patient has 400cc’s or more of urine out with catheterization, RN will
place catheter and notify MD.
Foley catheter care will be completed with bath every day and more often as needed.
Admission Standards:
New patients will be admitted during the shift on which they arrive.
1. Unless patient arrives to floor 30 minutes prior to end of shift (7am/3pm/7pm/11pm).
2. If patient arrives to floor one hour prior to end of shift (6am/2pm/6pm/10pm), patient
will be settled into room, vital signs assessed and admission paperwork initiated;
admission paperwork will be completed by next shift.
Patients should be reassigned when there are multiple discharges on one wing so that
admissions are evenly divided between RN’s.
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Discharge Standards:
Discharge will be completed in a timely manner upon completion of all discharge orders
and upon pending discharge being entered into the computer by MD or Patient care
facilitator. Foley catheter/IV/PCA will be discontinued the night before discharge.
1. If discharge orders and pending discharge are not completed 30 minutes prior to end
of shift, RN will be responsible for ensuring that IV is discontinued, patient has
transportation and belongings are collected. RN will initiate paperwork when
possible. Discharge paperwork and process is to be completed by next shift.
Transfer Standards:
Transfer paperwork of patients that are being transferred to another facility by 11am will
be completed by night shift RN. Report will be called by transferring RN.
Night shift RN will do as much preparation for transfer as possible (bath, dressing
change, IV discontinued). RN should also verify pending transfer order in computer/MD
chart and that discharge summary has been requested.
Report Standards:
Report MUST start at 7am/3pm/7pm/11pm. This is the beginning of the shift!
RN must pull up E-MAR and address any overdue tasks and both RN’s will check
orders in SCM to assure that they are up to date. This is the beginning of the shift!
Patient assignment will be done by the off going shift. (Night nurses will divide patient
for the day shift nurses and day shift nurses will divide patient assignment for night
nurses.) Patient assignment will be made with consideration of who had patient during
the previous shift, acuity level, # of patients being discharged).
Assessment Standards:
1. Nursing assessments will be done at the beginning of every shift
Except for patients needing more frequent assessment (per RN’s discretion)
2. Patients will be assessed upon arrival to floor from procedure, surgery or transfer
from ICU.
150
Logroll Guidelines
General rules:
1. Patients are transported to UK Hospital immobilized so must consider pre-hospital board
times (don’t forget referring hospital pre-transfer time as well), thus you may receive patient that
has had extended length of time on a board.
2. Evaluate your patient for risk factors associated with skin breakdown such as poor nutritional
status, circulatory impairment from cardiac or vascular disease, diabetes, lack of adipose tissue,
etc.
3. Hardboards should be utilized in the ICU/Floor for patient transfer and obtaining films only
and must be discontinued as soon as possible to prevent breakdown. Do not keep patient on a
board for any longer than necessary (2 hours is maximum time on board).
5. Don’t use slider board to transfer the patient. Sliders are flexible devices that do not offer
appropriate spine immobilization.
6. Must reassess sensory/motor function with every turn, transfer and prn.
D. HOB flat
1. maintained at all times
2. Reverse Trendelenburg may be used to elevate patient head after
logroll procedure completed
151
E. Inspect Cervical Collar
1. Correct size?
2. Appropriately applied/positioned?
C. Leader directs assistants to turn patient (in unison on count of ―3‖) toward them
onto patient’s side
1. Leader monitors alignment (nose & umbilicus) continuously
152
G. Leader re-assesses sensory/motor function after all logroll procedures
C. Slider boards must not be used to LIFT or TRANSFER patient. Slider is not a
rigid surface, thus not a suitable lifting or transfer device.
Exception: slider may be used under hardboard (not next to patient) to
reduce friction associated with movement from surface to surface.
D. Portable Diagnostic X-rays
1. Place patient on rigid backboard per log roll procedure
2. Leader and assistants lift patient on rigid hardboard in unison count ―1-
2-3‖
3. Pancake x-ray board is placed between bed and patient on rigid
backboard
4. Count ―1-2-3‖ in unison to lower patient/hardboard onto pancake board
5. Notify radiology that patient is ready for films
6. Patient remains on hardboard and pancake board until radiology
approves quality of films obtained
References
Hadley, MN. Cervical spine immobilization before admission to the hospital. Neurosurgery.
2002, Mar;(50):S7-S17.
Monhadevan, SV, et al. The evaluation and clearance of the cervical spine in adult trauma
patients: clinical concepts, controversies, and advancement, part 1. Trauma Reports. 2004,
July.
153
University of Kentucky Medical Center
Fever has both beneficial and harmful effects. Fever is a beneficial survival mechanism that
enables the body to combat infection. Endogenous pyrogens stimulate production of T & B
lymphocytes which increases the body’s antibody production twenty-fold. The harmful effect of
fever is a result of increased metabolic activity which in turn increases oxygen consumption.
The goal of therapy should be aimed at allowing the body’s defense mechanism to help combat
the infectious organism without compromising tissue oxygenation. If the fever is not
compromising tissue oxygenation it is not necessary to treat fever.
Diagnosis and source of infection are often difficult to identify in the critically ill patient. Also,
there are many clinical conditions which produce hyperthermia that should be considered when
attempting to identify the source of fever.
The following algorithm was designed to assist the resident and the intensive care unit nurse in
clinical decision making for appropriate intervention and treatment of the critically ill febrile
patient Protocol for Fever
154
Fever Evaluation
Temperature > 38.9 C [102 F.] or > 38.3˚C [101˚F] if patient is neutropenic
YES NO
YES NO
YES NO
Receiving NMBA
YES NO
155
Cannot use external cooling if not receiving NMBA or not properly sedated. Cooling may cause shivering, peripheral
vasoconstriction driving deep thermal temperature higher and a marked increase in oxygen consumption if not paralyzed or
adequately sedated.
*Blood cultures should be obtained from 3-4 different sites ~20-30ml. (No data to support waiting between draws)
**Pan cultures defined as sputum, blood and urine cultures.
***Special Considerations: Also, consider sinusitis or central nervous system infections [Refer to common source of
infections in ICU patient].
****Acetaminophen 650mg po/pr q4-6 hours PRN should be used for patients with renal insufficiency (Cr>1.5),
thrombocytopenia (<50,0000), a known bleeding diathesis, gastrointestinal stress ulceration, or a hypersensitivity to
NSAIDS.
Alcohol withdrawal
Atelectasis
Deep venous thrombosis
Drug withdrawal
Drugs *
Hematoma
Neuroleptic hyperthermia
Pancreatitis
Subarachnoid hemorrhage
SIRS (secondary to shock, trauma)
Tissue necrosis
Transfusions
*Drugs:
Allopurinol
Antibiotics (penicillin, sulfonamide, cephalosporins)
Antihistamines
Barbiturates
Dobutamine
Hydralazine
Methyldopa
Procainamide
Phenytoin
Quinidine
156
Possible Sites of Infection Seen in the ICU Patient
Pneumonia: (The most common nosocomial infection in the ICU). Risk factors include
prolonged intubation, chest trauma and ARDS. Pathogens commonly found include: gram
negative enteric organisms (Hemophilus, Pseudomonas, and Enterobacter) and/or gram
positive organisms (Enterococcus, other strep species and Staphylococcus aureus).
Wound Infection: The wound may be erythematous with or without purulent drainage, or
subcutaneous crepitus. A surgical wound infection may not be clinically apparent until 5 to 7
days post-operatively.
Vascular Catheter Related Infection: The risk of line infection increases with the length of
time the vascular cannula has been in place.
Sinusitis: The risk factors include: nasogastric tube, nasotracheal tube, nasal packing, facial
fractures, recumbent positions, and high dose steroids.
Acalulous Cholecystitis: Any critically ill patient is at risk. Contributing factors include:
opiates, fasting, TPN and shock.
Empyema: The risk factors include: pneumothorax, hemothorax, penetrating chest trauma,
unrecognized diaphragmatic perforation and pneumonia.
Tracheitis: Usually associated with tracheal intubation. Manifestations may include foul
smelling purulent tracheal secretions.
Vascular Grafts: Manifestations of vascular graft related infections include: wound drainage,
wound infection, graft thrombosis, septic emboli and pseudoaneurysm.
Mediastinitis: Can be seen after surgical procedures performed through a median sternotomy
and with injuries to the aerodigestive tract.
Central Nervous System Infection: The risk factors include CSF leak (following craniotomy
or basilar skull fracture), craniotomy, intraventricular catheter or penetrating spinal cord injury.
157
Nursing Guidelines
The goal of therapy is to allow temperature elevation considering the possible benefits of
immune functioning, but be aware of the harmful effects that require
immediate interventions.
1. Tissue oxygenation: Keep SvO2 > 60, SaO2 > 90, in the absence of shivering
2. Hydration: Keep PCWP > 10, CVP > 8, UOP > 30cc/hr
3. Nutrition: Consult R.D. to ensure metabolic needs are being met with current feeding
regimen.
4. Pain/Sedation/Monitor for signs and symptoms of pain and assess need for sedation.
Adequate sedation will help control shivering and if unsuccessful will need NMBA.
5. External cooling should only be used if temperature is > 40 C. AND the patient is receiving a
NMBA or is properly sedated.
6. Antipyretics may be given if patient temperature is < 39.0 C. and adequate tissue
oxygenation cannot be achieved.
7. Rectal temperatures correlate most closely with core temperatures and should be used if
the patient does not have a pulmonary artery catheter, unless contraindicated or temperature
sensing Foley catheter.
External Cooling:
Hyperthermia is a natural adaptive mechanism in critical illness. Hypothalamic temperature
regulation is adjusted upward to accommodate the hyperthermia associated with
hypermetabolism and infection. Under these circumstances, attempts to lower temperature to
normal can be harmful because CNS autoregulation has been reset at a higher core
temperature. External cooling will produce increases in sympathetic tone that markedly
increase oxygen consumption. The body will attempt to restore temperature, during external
cooling by stimulating skeletal muscle, producing shivering, will increase tissue oxygen
consumption. External cooling will cause peripheral vasoconstriction. This will shunt heat
deeper and make it more difficult to cool. Recognizing the role of hyperthermia in critical illness,
a more permissive attitude is taken towards temperature elevation. Modest rise in core
temperature is monitored without treatment, and moderate temperature elevation (>102.2 ) is
treated with antipyretics. External cooling is reserved for extreme temperature elevation (>104 )
when compromise of tissue oxygenation and/or direct tissue damage may occur. More
aggressive temperature control can and should be employed when marginal tissue oxygenation
occurs with lower temperatures.
158
References
Rosenthal KE, Joshi, M. Evaluating the septic-appearing multiple trauma patient. Critical Care
Report. 1990; 1: 323-335.
Bruce JL, Grove SK. Fever: pathology and treatment. Critical Care Nurse. 1992; 12(1):40-49.
Wasserman MR, Keller EL. Fever, white blood cell count, and culture and sensitivity: their
value in the evaluation of the emergency patient. Topics in Emergency Medicine.
1989; 10(4): 81-88.
S, Livingston DH, Elcavage J, et al. The utility of routine daily chest radiography in the surgical
intensive care unit. The Journal of Trauma. 1993;35(4): 643-646.
Larach MG, Localio AR, Allen GC. et al. A clinical grading scale to predict malignant
hyperthermia susceptibility. Anesthesiology. 1994; 80(4): 771-778.
Bessey PQ. Metabolic response to critical illness. Scientific American Medicine. 1994.
Revised: 12/08
159
Guideline for Rib Fracture Management
Rib fracture repair has been selectively performed for more than 50 years; however,
clear operative indications have not been established yet. The long-term outcome of a
strictly nonoperative approach to flail chest, flail sternum, and rib series fracture may not
be optimal with the development of chronic pain, chest wall deformity, reduced chest
wall compliance and rib fracture nonunion.
In order to improve patient’s morbidity and mortality, indications for rib fracture repair
should be considered in patients with flail chest, flail sternum and painful movable rib
fractures refractory to conventional pain management1-7.
When considering rib fracture repair, anterior plating with bicortical locking screws or
locked intramedullary nails should be used. The anterior approach, which can be
performed in a minimal invasive technique, does not violate the pleural space. Further
locking screws, where the screw is threaded in to the plate improves fixation stability
especially in thin and osteoporotic bone2,4,7.
Conservative rib fracture management should be adequate with the use of PO/IV pain
medication as well as intercostal pain pumps6,7.
ORIF of rib fractures does not normally require an OnQ pump for post operative pain
management. PO/IV medication or IV PCA should be considered first for pain
management.
Rib series: > 6 fractured ribs. Consider ORIF repair for pain management only if
fractures occur at rib levels 3 – 7 with a failure of conservative pain management.
Sternal flail: sternum is dissociated from the hemi-thoraces of bilateral, multiple, and
anterior cartilage or rib fractures.
160
References:
161
162
Chest Tube Management Protocol
A pneumothorax occurs when the resting negative pressure in the pleural space is lost,
leading to collapse of the ipsilateral lung. Pneumothoraces can occur spontaneously (primary
pneumothorax) after for example, a bleb rupture; or result from trauma (secondary
pneumothorax). In penetrating trauma, air can enter the pleural space from the atmosphere. In
blunt trauma, air escapes the pleura after barotrauma or laceration by a fractured rib. Signs and
symptoms of a pneumothorax can include: dyspnea, pleuritic chest pain, anxiety, cough, and
tachypnea. Large or symptomatic pneumothoraces are treated with chest tubes (tube
thoracostomies).
Chest tubes are removed when the pneumo/hemothorax has resolved and the amount
of pleural effusion is decreased. Traditionally, chest tubes are removed from suction when the
air leak has resolved. Chest tubes on water-seal with no air leak, less than 200cc drainage in 24
hours and minimal or no residual pneumothorax on CXR are considered for removal. A chest x-
ray is usually obtained 3-8 hours after placement on water-seal to identify any expansion of the
pneumothorax. Sometimes, a second follow-up radiograph would be taken the next morning
and if still negative, the chest tube is removed. If the initial chest x-ray on water seal shows
development of or significant expansion of the pneumothorax, the chest tube is placed back on
suction and/or reevaluated. An interval chest x-ray would follow.
There is emerging evidence to support a more rapid and systematic approach to chest
tube removal. Recent studies have determined which technique, water-seal or suction, allows
for shorter chest tube duration, when radiographs should be obtained, and finally, the optimal
time interval for identifying a recurrent pneumothorax on chest x-ray after placing a chest tube
on water-seal (Schulman 2005).
Some authors have suggested that the placement of chest tubes to water seal is
unnecessary, prolongs dwell time and that tubes on suction with no air leak may simply be
removed without a period of water seal. Martino et al. determined that there was no difference in
total chest tube dwell time between chest tubes placed on water-seal vs. those in which water-
seal was not used, although it appears that a short trial of water-seal allows for occult air leaks
to become clinically apparent and significantly reduces the need for another chest tube (Martino
1999). This water seal trial followed by a CXR is the practice of the Blue Surgery Service.
Recently, Schulman et al. studied the time interval for identifying a pneumothorax on
chest x-ray after placing a chest tube on water-seal (Schulman 2005) and from this work
formulated a new chest tube removal algorithm. This study concluded that a chest x-ray
obtained 3 hours after placing a chest tube on water-seal effectively excludes development of a
clinically significant pneumothorax. Their algorithm (modified below) will allow patients to
progress from water-seal to chest tube removal and discharge on the same day, enhancing
patient satisfaction with potential associated cost-savings (Schulman 2005).
Pizano et al. studied the time interval between the removal of a chest tube and a chest
x-ray (Pizano 2002) and concluded that a chest x-ray obtained within 1-3 hours after chest tube
removal effectively identifies a pneumothorax. These data add further support to Schulman’s
algorithm which uses a 3 hour time interval between chest tube removal and ―post-pull‖ CXR. In
addition, this study was conducted in mechanically ventilated patients suggesting that it is safe
to remove a chest tube from patients undergoing positive-pressure ventilation (Pizano 2002).
163
Chest tube removal algorithm (From Schulman CI et al. J Trauma 2005;59:92-95).
References
Pizano LR, Houghton DE, Cohn SM et al. When should a chest radiograph be obtained after
chest tube removal in mechanically ventilated patients? A prospective study. J Trauma
2002;53:1073–1077.
Schulman CI, Cohn SM, Blackbourne L et al. How Long Should You Wait for a Chest
Radiograph after Placing a Chest Tube on Water Seal? A Prospective Study. J Trauma
2005;56:92-95
08/08
164
© University of Kentucky Medical Center
Trauma Surgery Service
DVT Risk Factors
Iatrogenic factors
Anticipated need for central line > 24 hours 1
More than 4 units transfused first 24 hours 2
Surgical procedure > 2 hours 2
Immobility > 72 hours 2
Immobility > 5 days 3
Repair of ligation of major venous injury 3
Age
40 to 59 1
60 to 74 2
> 75 3
165
DVT PROPHYLAXIS ALGORITHM
Contraindication to Heparin
Active hemorrhage
Solid Organ injury
Intracranial Hemorrhage
Etc.
(Should be cleared by NS or appropriate service prior to admission)
NO YES
YES NO
Apply SCD’s
Discontinue SCD’s
*Patients who are non ambulatory at discharge should be maintained on Lovenox or switched to
low dose Coumadin to maintain INR at 1.5-2.0. (Follow Ortho recommendations on discharge for
Lovenox. If no Ortho recommendations then order X 2 weeks or until ortho clinic follow-up.)
** Routine Duplex Surveillance is no longer performed. Venous Duplex should be requested
when clinical suspicion for DVT is present.
166
THERAPY FOR DOCUMENTED DVT/PE
1. Unfractionated heparin
IV heparin bolus of 80units/kg followed by a continuous heparin infusion @18units/kg/hour.
The goal is prolongation of the PTT to twice control (+ 10%). Obtain PTT 6 hours
after initiation of therapy. Dose adjustments can be made per protocol.
167
168
Revised 10/08
University of Kentucky Medical Center
Trauma Service
Pathophysiology
The splanchnic hypoperfusion that causes stress-related mucosal damage in critically ill
patients is multifactorial and results from sympathetic nervous system activation,
increased catecholamine release and vasoconstriction, hypovolemia, decreased cardiac
output, and the release of proinflammatory cytokines.
Other Considerations
Early enteral nutrition may be an important factor for preventing gastrointestinal mucosal
ischemia. Enteral feedings produce splanchnic vasodilation and increased mucosal blood flow
thereby preventing mucosal ischemia and its untoward consequences.
169
UK Trauma Service
Stress Ulcer Prevention Guidelines
Hospital Admission
≥ 1 Risk Factor?
Yes No
170
Diagnosis and Treatment of Stress Related Mucosal Bleeding
Diagnosis
Endoscopy is the diagnostic modality of choice.
Overt Bleeding
Overt bleeding is defined as hematemesis, melena, hematochezia, gross blood >
(100cc) in the NG tube, or coffee ground emesis or nasogastric drainage.
Treatment
Initial bolus of pantoprazole 80mg, followed by pantoprozole 8mg/hr continuous infusion
for 72 hours. Follow with once daily PPI for ___ months.
References
Cook DJ, Fuller HD, Guyatt GH, et al. Risk factors for gastrointestinal bleeding in
critically ill patients. The New England Journal of Medicine. 1994; 330(6): 378-381.
Tryba M. Sucralfate versus antacids or H2-antagonists for stress ulcer prophylaxis: a
meta-analysis on efficacy and pneumonia rate. Critical Care Medicine. 1991; 19(7):
942-949.
Schepp W. Stress ulcer prophylaxis: Still a valid option in the 1990s? Digestion. 1991;
54: 189-199.
Pickworth KK, Falcone RE, Hoogeboom JE, Santanello SA. Occurrence of
nosocomial pneumonia in mechanically ventilated trauma patients: a comparison of
sucralfate and ranitidine. Critical Care Medicine. 1992; 21(12): 1856-1861.
Pemberton LB, Schafer N, Goehring, et al. Oral ranitidine as prophylaxis for gastric
stress ulcers in intensive care unit patients: Serum concentrations and cost
comparisons. Critical care Medicine. 1993; 21(12): 339-342.
Loperfido S, Monica F, Maifreni L, et al. Bleeding peptic ulcer occurring in
hospitalized patients: Analysis of predictive and risk factors and comparison with out-of-
hospital onset of hemorrhage. Digestive Diseases and Sciences. 1994; 39(4): 698-
704.
Cook D, Gordon G, Marshall J, et al. A Comparison Of Sucralfate And Ranitidine For
The Prevention Of Upper Gastrointestinal Bleeding In Patients Requiring Mechanical
Ventilation. The New England Journal of Medicine. 1998; 338(12): 791-797.
171
172
Revised: 09/08
University of Kentucky Medical Center
Trauma Service
Guideline for Maintenance of
Endoscopic Feeding Tubes
Early enteral nutrition is very important in the management of critically ill patients. Obtaining
and maintaining access to the GI tract is an essential component of patient care. The gastric
dysfunction that accompanies critical illness necessitates post pyloric placement of small-bore
feeding tubes to insure tolerance of enteral formulas and the administration of medication.
Reliable post-pyloric placement of feeding tubes using a blind technique is difficult. Surgical
and endoscopic methods are both difficult and expensive. Maintaining patency of these
important enteral access devices to insure continuous nutrient flow and to prevent costly tube
placement is essential for proper patient care.
These guidelines were developed to assist the clinician with medication administration,
maintenance of the feeding tube, and trouble shooting guideline when the feeding tube
becomes clogged.
Maintenance
Routine flushing with 20 - 30 ml of water before and after medications is essential for
maintaining patency, especially with medications containing sorbitol. If enteral nutrition is held
for any length of time the feeding tube should be flushed with 30 ml of water. Adequate flushing
of the feeding tube is the key to maintain patency and cannot be over emphasized.
173
Assessment of Small Caliber Feeding Tubes and Feeding Tube Regimen
1. Auscultation is not adequate to verify feeding tube location initially or during ongoing
assessment. An x-ray must be done to confirm position.
2. Monitor for potential regurgitation of tube feeding into the stomach by aspiration from the
nasogastric tube every 4 hours.
3. Change tube feeding canister every 24 hours, formula every 12 hours for canned
preparations, and every 6 hours if pre mixed by dietary.
4. Document cessation of tube feeding, amount given, versus the amount prescribed.
Administration of Medications
Nursing Guidelines
174
Troubleshooting Obstructions for Small Caliber Feeding Tubes
1. If a small caliber feeding tube becomes clogged, attempt unclogging with warm (not hot)
water or carbonated soda.
2. Pancreatic enzymes (Viokase) can be used to unclog tubes. Prior to administration of
above agent, aspirate tube-feeding formula using a 30 - 50 ml syringe. This will clear
tube up to the obstruction site. Clamp tube post administration for 15 minutes, and then
flush with water.
3. Due to rare instances of tube perforation, reinsertion of the stylet is not recommended
without the physician’s approval or supervision
175
Clogged Feeding Tube
Clogged Unclogged
Clogged Unclogged
Clogged
Clogged Unclogged
176
References
Byerly WG. Drug information questions and answers. Hospital Pharmacy Hotline. 1992; 5: 2.
Drug Information Center, University of Kentucky Medical Center. Pharmacy News for Nurses.
1992; 13(4):
Kohn CL, Keithley JK. Enteral nutrition-Potential complications and patient monitoring. Nursing
Clinics of North America. 1989; 24: 339-353.
Lehmann S, Barber J. Giving medications by feeding tube. How to avoid problems. Nursing
91. 1991; 21: 58-61.
Paauw JD, Fagerman KE, McCamish, et al. Enteral nutrition solutions. The American Surgeon.
1984; 50(6): 312-316.
Potts RG, Zaroukian MH, Guerrera PA, et al. Comparison of blue dye visualization and glucose
oxidase test strip methods for detecting pulmonary aspiration of enteral feedings in intubated
patients. Chest. 1993;103: 117-121.
Powell KS, Marward SP, Farrior ES, et al. Aspirating gastric residuals causes occlusion of
small-bore feeding tubes. Journal of Parenteral and Enteral Nutrition. 1993; 17: 243-246.
Thurlow PM. Bedside enteral feeding tube placement into duodenum and jejunum. Journal of
Parenteral Enteral Nutrition. 1986; 10: 104-105.
White WT, Acuff TE, Skyes TR, et al. Bacterial contamination of enteral nutrient solution: A
preliminary report. Journal of Parenteral and Enteral Nutrition. 1979; 3(6): 459-461.
Zaloga GP. Bedside method for placing small bowel feeding tubes in critically ill patients. A
prospective study. Chest. 1991; 100(6): 1643-1646.
177
UNIVERSITY OF KENTUCKY HOSPITAL POLICY NUMBER: HP08-39
CHANDLER MEDICAL CENTER FIRST ISSUED: 7/04
HOSPITAL POLICY CURRENT AS OF: 9/06
INFORMATION
To facilitate delivery of enteral nutrition in adults, the Hospital has established guidelines for the
verification and maintenance of small-bore feed tubes.
Clinical Nutrition’s consultation service should be involved with all patients receiving enteral
nutrition, and will be responsible for ordering the feeds.
Prevention/Assessment of Aspiration
For All Patients on Tube Feeding
1. Unless contraindicated (i.e., patient with cervical spine injury or subdural drain), elevate the
head of the bed to 30 to 45 degrees to reduce the risk of aspiration. Patients with spinal
injuries may be able to be placed in reverse Trendelenburg position at 30 to 45 degrees to
reduce the risk of aspiration.
Note: The nurse should verify head of bed elevation with the physician.
2 -- HP08-39, Verification and Maintenance of Small-Bore Feed Tubes in Adults
178
For Critically Ill Patients with One Major Risk Factor (refer to following list of risk factors),
it is recommended: • to provide all care listed above, • maintain tight glycemic control (blood
glucose between 80-110 mg/dl), and • use continuous infusions rather than intermittent boluses.
For Critically Ill Patients with Two or More Major Risk Factors (refer to following list of
risk factors), it is recommended: • to provide all care listed above, • that all feeding tubes be
in the small bowel, and • to use prokinetic agents (Metoclopramide).
Major Risk Factors • previous episode of aspiration • decreased LOC (sedation or elevation in
intracranial pressure) • neuromuscular disease • neuromuscular blocking agents • endotracheal
tube or intubation • vomiting • persistent high residuals For
179
Revised 9/08
University of Kentucky Medical Center
Trauma Intensive Care Unit
Bowel Regimen Program
The injured/critically ill patient undergoes physiologic alterations in gut function. Gastric motility
slows; use of narcotics slows intestinal motility, warranting bowel function surveillance to ensure
timely identification and intervention of alterations. All trauma service ICU patients will be
started on a bowel protocol upon admission consisting of Docusate Sodium 250 mg twice daily
and Biscodyl suppository twice daily prn. Assessment of the patient should include bowel
patterns pre-injury, other medical conditions that predispose patient to alterations in bowel
function, medications,
High risk indicators: use of narcotic analgesia, lack of BM x 24 hours. Diarrhea and constipation
in critically ill patients may either be serious or benign conditions. There are many causes for
constipation such as improper diet, intestinal obstruction, tumors, excessive use of laxatives and
weakness of the intestinal musculature. What constitutes constipation is very subjective and
individualized. However, absence of stool > 3 days in patients receiving enteral nutrition should
be investigated and appropriate therapy initiated when indicated.
Diarrhea is common in critically ill patients, occurring in 24% of patients admitted to intensive
care units. Many factors contribute to diarrhea, such as infection, antibiotics, and drugs
containing sorbitol, bowel edema, inflammation, enteral feeding rate, and hyperosmolar enteral
feedings. Incomplete digestion may occur in patients who have had feedings withheld for
several days, therefore low rates with slow advance are recommended when beginning enteral
feedings. However, diarrhea associated with enteral feedings is usually seen in patients who
have gut atrophy from prolonged fast or patients on high hourly feeding rates (> 75 ml/hour).
Hypoalbuminemia (<2.5g/dl) and attendent bowel edema have been implicated as a cause of
diarrhea, but this association has never been firmly established.
Diarrhea associated with antibiotic therapy is usually benign, but can be serious. Antibiotic
therapy alters colonic flora which may allow overgrowth of pathogenic and or diarrheagenic
bacteria. Clostridium difficile is present in the normal colonic flora of some patients.
Overgrowth by toxigenic C. difficile can occur in patients receiving antibiotic therapy. C. difficile
infection can present as diarrhea that is mild to moderate or can cause severe colitis with
pseudomembrane formation. Pseudomembraneous colitis can lead to severe diarrhea,
hypovolemic shock, toxic megacolon, perforation and death. Specific findings of
pseudomembraneous colitis include watery, green, foul-smelling, non-bloody or bloody diarrhea,
cramping abdominal pain, fever (> 39.5 C) and leukocytosis. Stool specimen should be sent for
C. difficile toxin. C. difficile culture is a less efficient method and does not differentiate between
nontoxiocogenic colonic flora and toxic strains of C. difficile. Turnover time for the C. difficile
toxin assay is usually 24 to 48 hours. Severe diarrhea, abdominal distention, tenderness,
unexplained SIRS, megacolon on plain film and a high index of suspicion should prompt an
endoscopic evaluation to exclude pseudomembranes.
Treatment varies according to the severity of the illness. Enteral metronidazole is the first line
drug of choice. If a patient cannot tolerate oral medication intravenous metronidazole should be
given. Intravenous vancomycin is not effective in the treatment of C. difficile enterocolitis.
Enteral vancomycin is the drug of choice for severe colitis and treatment failures on
metronidazole.
180
181
182
Nursing Guidelines:
Diarrhea:
Constipation:
1. Notify physician when patient who is receiving PO/enteral feeding has not had a stool for > 3
days
2. Ensure patients are receiving adequate hydration
3. Assess for medications or dietary intake (low residue, low fiber) that may contribute to
constipation
4. Lubricant should be used when manually removing impaction
5. Abdominal pain and/or distention may be a clinical manifestation of a serious problem
References
Thomspon, et al. (1992) cited in Powell, M and Rigby D. Management of bowel dysfunction:
evacuation difficulties. Nursing Standard. 2000; 14(47): 47-54.
Fekety R, Shah AB. Diagnosis and treatment of clostridium difficile colitis. JAMA. 1993; 269:
71-75.
Kelly CP, Pothoulakis C, LaMont JT. Clostridium difficile colitis. The New England Journal of
Medicine. 1994; 33(94): 257-262.
Hwang TL, Lue MC, Nee YJ, et al. The incidence of diarrhea in patients with hypoalbuminemia
due to acute or chronic malnutrition during enteral feeding. American Journal of
Gastroenterology. 1994; 89(3): 376-378.
Johnson, DA. Diarrhea in critically ill Patients. Problems in Critical Care. 1989; 3(3): 392-408.
183
Blue Surgery Bladder Management Protocol
for the Urologically Non-complicated Patient
UTIs account for 31% of nosocomial infections in US medical ICUs, thus it is essential to
understand the logistics of Foley monitoring and maintenance. The risk of bacteriuria from an
indwelling catheter is 3-10% per day. Daily assessment for the continued necessity of an
indwelling catheter and removal when they are no longer indicated are simple but important
steps to reduce infection, encourage a return to normal physiologic function, and improve
patient care.
Intermittent catheterization deals with draining the bladder at specific time intervals to simulate
normal physiologic function. An adult bladder holds approximately 400 ml of urine and voids 4-5
times/day. Intermittent catheterization should likewise be performed a 4-5 times/day with
adjustments based on volume drained. The total volume drained should not exceed 400 ml. If
volume drained is high, either the frequency of intermittent catheterizations should be increased
or fluid intake should be decreased.
Spinal Cord Injuries: According to several studies, intermittent catheterization is a better option
than indwelling catheterization for both male and female patients. Some studies show that for
patients who are undergoing catheterizations approximately 4 times/day, bacteriuria occurs at
an incidence of 1-3% per catheterization. Antibiotics are usually not required during intermittent
catheterizations unless the patient is in a high-risk population (immunosuppressed, internal
prosthesis, etc.).
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University of Kentucky Trauma and Critical Care Recommendations for
Central Venous Catheter Insertion
Adapted from the American College of Surgeons Guidelines for Central Venous Access*
Indwelling vascular catheters are essential for patient care. All indwelling vascular catheters
have associated mechanical and infection risks. Catheters should be place when needed and
the insertion should be performed properly using strict aseptic technique. Catheter
maintenance should follow accepted guidelines including sterile technique for infusions, lines,
and hubs. Site inspection and care are essential for preventing infection. Catheters should
never remain in place for caregiver convenience and should be removed when no longer
needed.
Placement
1. Appropriately trained and experienced personnel identified to place CVC
2. Time out performed prior to procedure (patient ID, consent, site selection, diagnosis)
3. Choose the most appropriate site for CVC insertion based on patient’s needs
4. Full sterile precautions must performed on ALL non-emergent CVC insertions. This
includes sterile hat, mask w/shield, gown and gloves. A fully body sterile sheet is
applied after appropriate antisepsis.
5. Appropriate antisepsis is achieved with 2% chlorhexidine gluconate for 30 seconds and
then allowed to air dry. If this is not available, it is appropriate to use iodine, iodophore
or 70% ethanol, no organic solvents.
6. Choose a central venous catheter with the minimum number of lumens for your patient’s
needs.
7. All internal jugular and femoral lines should be placed under ultrasound guidance unless
emergent line placement is required.
8. If a CVC is malfunctioning then a new one may be placed by exchange over a guidewire
ONLY if there are no signs of bacteremia and/or infection
9. >3 needle sticks for access increases risk of insertion complications and consideration
should be given to more experienced personnel insertion and/or new stick site
10. After successful insertion a chlorhexidine impregnated sponge should be placed around
the catheter at the insertion site.
11. Position of CVC must be evaluated with a STAT chest x-ray. Correct CVC should have
the tip near the SVC and right atrial junction. All CXR should be evaluated for evidence
of pneumothorax.
12. If line is in satisfactory position the line needs to be cleared for use in SCM. Alert the
nursing staff that the line is ok to use and that the order for clearance is in SCM.
13. A procedure note is to be performed on all CVC insertion attempts, successful or not.
SCM has a procedure note dedicated to CVC insertion. This is the default and expected
method of procedure note completion. A brief note in the chart should indicate the
patient name, date and time with reference to the complete note in SCM.
Maintenance
1. The routine replacement of central lines does not prevent CRBSI and is not
recommended in the absence of CRBSI
2. The CVC site should be inspected daily and PRN
3. Before manipulation of any CVC proper hand aspesis should be performed by washing
with soap and water or alcohol scrub (even when gloves are worn)
4. Clean gloves should always be donned before CVC manipulation
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5. If CVC lumen access is attempted via the injection ports or caps then asepsis should be
applied to the ports/caps with 2% chlorhexidine gluconate or another appropriate
antisepsis
6. 2% chlorhexidine gluconate, iodine, iodophore or 70% ethanol (no organic solvents)
should be used for all CVC dressing care and dressing changes. All antisepsis should be
allowed to air dry before manipulation/dressing changes.
7. Use a sterile, transparent dressing over catheter site with clean gloves and a no-touch
technique. If the site is not dry, then apply a sterile dry gauze and change dressing when
it becomes saturated. Change to a transparent sterile dressing as soon as possible.
8. Sterile dressing should be replaced q7 days unless it becomes loose.
9. There is no indication for antimicrobial prophylaxis (systemic or local) with an indwelling
CVC.
10. Avoid anticoagulants for clot or CRBSI infection unless certain patient conditions
mandate their use
11. Use sterile NaCl (heparin if indicated) to flush and lock to maintain patency
12. The use of needle free connectors is encouraged to prevent needle stick injuries, and
aseptic technique should always be followed.
Central venous, pulmonary artery, and peripheral venous lines placed outside the
Intensive Care Unit
All deep lines place outside of the intensive care areas must be changed to a new sight within
24 hours of admission to the unit. Exceptions to this policy are as follows:
1. Lines placed under aseptic conditions in the operating room, on the floor, or in another ICU
where sterility of the procedure can be documented.
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2. All emergent lines, placed under non-sterile conditions, placed in the ED for emergency
resuscitation must be removed within 24 hours of admission to the ICU without exception.
Route of Insertion
1. The percutaneous route of arterial line placement is preferred to surgical cutdown.
a. Surgical cutdown for arterial cannulation should only be performed after approval by
the critical care attending.
Site of Insertion
1. The preferred site of arterial cannulation is the radial artery. Alternate sites of arterial
cannulation are for most patients listed in descending order preference.
a. Femoral artery*
b. Dorsalis Pedis artery*
*In patients with peripheral arterial disease, percutaneous femoral arterial cannulation has a
much higher complication rate and should be avoided. Dorsalis Pedis cannulation may lead to
inaccurate blood pressure measurements. Caution must be exercised in choosing the site of
arterial cannulation in this group of patients.
2. The complications associated with brachial artery and axillary artery cannulation are
higher than other routes.
a. Brachial artery: Used uncommonly because of the high complication rates. The major
complication is thromboembolic occlusion usually without ischemia.
b. Axillary artery: Limb threatening ischemia negligible. However,
catheter infection rate is much higher than other sites and line care
is difficult.
Insertion Procedure
1. The site chosen for arterial cannulation should be prepped and draped to create a sterile
field.
2. Sterile gloves and mask should be worn during insertion of the line.
3. The line should be sutured in place using aseptic technique.
4. At the completion of the procedure, a chlorhexidine impregnated sponge is applied to the
insertion site and covered with a sterile MVP (Tegaderm © or OP-Site ©) dressing.
Line Changes
1. The infection rate in percutaneously placed arterial cannulas are very low. Routine
catheter removal and change to a new site is unnecessary. Arterial cannulas can remain
at the original site of insertion until no longer needed.
Exception: Catheters should be changed to a new site when:
a. There is evidence of infection at the insertion site manifested as pain, redness,
swelling, or purulence. Catheters should always be cultured in this situation.
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b. There is evidence of ischemia distal to the site of insertion.
c. The catheter is implicated as a potential source of unexplained systemic
sepsis.
2. The decision to change the arterial line to a new site or to culture the catheter tip is left to
the discretion of the treating physician. It is recommended that catheter tips be
submitted for culture when there is evidence of infection at the site of insertion or when
the patient exhibits unexplained sepsis.
Reference
Freel AC, et al. American College of Surgeons Guidelines Program: A Process for Using
Existing Guidelines to Generate Best Practice Recommendations for Central Venous Access.
2008 June 19.
189
Revised: 11/08
Introduction:
Alcohol abuse and/or addiction are frequent problems in the trauma patient population.
Acute alcohol withdrawal increases mortality and morbidity for the critically ill and/or
injured patient. It is essential that physicians and nurses obtain a thorough history to
ascertain if the patient has a history of alcohol abuse or dependency.
Symptoms of alcohol withdrawal begin 6 - 12 hours after cessation of alcohol and can be
difficult to differentiate from injury/illness related symptoms. Tremor, nausea, vomiting,
tachycardia, hypertension, diaphoresis, irritability and profound anxiety are frequently
seen. Seizures are the most life threatening symptom of alcohol withdrawal. Seizures
cause additional stress on body reserves which will further compromise the critically ill
patient. Delirium tremens is classified as the ―late‖ phase or major withdrawal.
Patients will often have a combined alcohol and hypnosedative abuse which requires a
different treatment approach to prevent withdrawal symptoms.
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[Differentiation from behaviors associated with closed head injuries, and
agitation/anxiety related to uncontrolled pain is essential]
1. Monitor vital signs q 1 hour x 24 hours
2. Monitor agitation/sedation level q 1 hour x 72 hours then q 4 hours and PRN
Assess and record the following vital signs parameters prior to each dose:
Heart Rate
MAP
Respiratory Rate
SpO2
Level of sedation
B. Carbamazepine:
Carbamazepine level prior to starting maintenance dose if patient is having signs
and symptoms of withdrawal Goal is to have level > 12.
Assess and record the following vital sign parameters prior to each dose:
Heart Rate
MAP
Respiratory Rate
SpO2
Level of sedation
Hold next dose and/or notify physician if: Heart rate < 60 [if a change from
baseline]
191
C. Haloperidol:
References:
Ries, RK, Roy-Byrne, PP, Ward, NG, et al. Carbamazepine treatment for benzodiazepine
withdrawal. American Journal of Psychiatry. 1989; 146: 536-537.
Klein, EK, Uhde TW, Post RM. Preliminary Evidence for the utility of carbamazepine an
alprazolam withdrawal. American Journal of Psychiatry. 1986; 143: 235-236.
Butler D, Messsiha FS. Alcohol withdrawal and carbamazepine. Alcohol; 1986; 3: 113-129.
Denicoff KD, Meglathery SB, Post RM, et al. Efficacy of carbamazepine compared with
other agents; a clinical practice survey. Journal of Clinical Psychiatry. 1994; 55(2): 70-75.
Strasen, L. Acute alcohol withdrawal syndrome in the critical care unit. Critical Care
Nurse. 1982: 24-30.
Menza MA, Murray GB, Holmes VF, et al. Decreased extrapyramidal symptoms with
intravenous haloperidol. Journal of Clinical Psychiatry. 1987; 48(7): 278-280.
Fraser GL, Riker RR. Controlling severe agitation in the critically ill with intravenous
haloperidol. Hospital Pharmacy. 1994; 29(7): 689-691.
192
Hyatt M. Anxiolytic Drug Abuse, Addiction and Withdrawal
193
Minimum time from Minimum time from last dose
last dose to catheter to catheter removal
placement/spinal
injection
Standard Anticoagulants
Warfarin INR < 1.5 Avoid use while
Heparin, full dose IV/SQ INR < 1.5 catheter in place
Heparin prophylaxis (BID, TID) No restrictions
Newer Anticoagulants
Xigris (Drotrecogin Alfa) 24 hours
Arixtra (Fondaparinux) 24-48 hours Avoid use while
Lovenox (1 mg/kg BID) 24 hours catheter in place
Lovenox (30 mg SQ BID) 10-12 hours
Lovenox (40 mg SQ QD) 10-12 hours 10-12 hours
Direct Thrombin Inhibitors
Argatroban aPTT < 40 Avoid use while
Lepirudin aPTT < 40 catheter in place
Antiplatelet Agents
ASA/NSAIDs No restrictions
Plavix 7 days Avoid use while
Ticlid 14 days catheter in place
Thrombolytic Agents
TPA (full dose for stroke, MI, etc.) 10 days Avoid use while catheter in
place
TPA (2 mg for catheter clearance) No restrictions
References:
University of Washington Medical Center, Nursing Epidural Policy and Procedure
[Link] Accessed May 14, 2008
Second ARSA Consensus Conference on Neuraxial Anesthesia and Anticoagulation, Reg
Anesth Pain Med 2003;28:172-97
05/08
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SECTION 6: DISCHARGE INSTRUCTIONS
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Discharge Instructions
D/C Home:
Please be thinking ahead of time on patient’s D/C home plans. Please inform the bedside RN of
D/C plans so home care instructions can be completed. If the pt. needs home health or home
equipment, please contact patient care facilitator assigned to Blue Surgery for this before the
day of D/C. The Care Facilitator will contact you if they need written prescriptions for any of the
Home Health/DME orders. On the weekends, the weekend social worker can set up Home
Health; the patients do not need to be kept over the weekend for home health needs only.
*For patient’s who live out-of-state, you will need to do a STAT D/C summary to send
with the patient in case they can find a local MD to provide their follow-up care.
Inter-Facility Transfers:
The Social Worker for our service can assist you with inter-facility transfers. If you suspect the
pt. will need some type of placement, contact the S.W. on pager number found on Blue Surgery
Census. Inform the pt./family that they may have some rehabilitation needs and that our S.W.
will come by to talk with them. Please never tell a pt./family we will send them to a specific place
(such as Cardinal Hill). Many factors need to be looked at before a pt. can go to a facility and
not all pts. are candidates for CHH.
Once a facility has accepted the pt. please complete the following:
1. Complete a STAT D/C summary. This will have to go with the pt. It takes several hours to
get this done so please plan accordingly.
2. Complete the UK Discharge Orders Form (H341). This form still needs to be completed
except for the prescriptions, since the list of meds are in the D/C summary. Please make
sure you write for all needed F/U appointments on the form so the clerk can schedule these
before the pt. is transferred.
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3. There is an Inter-Facility Form (J076) that has to be completed prior to transfer. This form
has 2 pages. You are responsible for completing the M.D. section on page 1 and the Risk &
Benefit section on page 2
4. Write an order ―Transfer to _______‖ in routine M.D. Orders
Subject: Physician responsibility in completion of Inter-Facility Transfer Form (J076) that
must be completed by the physician prior to transfer of patient to another facility
that will assume care of the patient.
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Discharge Pain Medication Algorithm
Outpatient Surgery, Including Laparoscopy-8 Day Taper
Long-Bone & Pelvic Fractures and Ribs-20 Day Taper (until ortho follow-up)
Percocet 5/325mg
1-2 PO q4hr PRN x 5 days then
1-2 PO QID PRN x 5 days then
1-2 PO TID PRN x 5 days then
1-2 PO BID PRN x 5 days and stop
Disp #150
Tylenol or Motrin thereafter unless contraindicated
Lortab is available as 5/500mg. Therefore, tapers with Lortab must not exceed 2 tabs
q6hr so that total Tylenol dosage does not exceed 4g/day.
Instruct the patient that they may take ibuprofen 600mg QID with Percocet or Lortab but
that Tylenol/acetaminophen should not be taken with Percocet/Lortab. Discharge Pain
Medication Protocol
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SECTION 7: WITHDRAWAL OF CARE/POTENTIAL ORGAN DONATION
INFORMATION
In order to ensure appropriate patient care and abide by KRS 446.400 as it defines and
applies to a patient's death, staff physicians of the University of Kentucky Hospital
should follow these established guidelines in determining death.
Ordinary Circumstances
In ordinary circumstances, the signs of death are:
1. Unresponsiveness,
2. Absence of pulse and heartbeat,
3. Absence of spontaneous respiratory movement and all other movement, and
4. Absence of reflexes.
Cerebral Death
Cerebral death is defined as the absence of cortical and brain stem function.
Certification of signs of cerebral death shall be attested to and documented by a
member of the active medical staff.
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7. No respiratory movements when the patient is disconnected from the
mechanical ventilator. Adequate testing for apnea is very important. An accepted
method is ventilation with pure oxygen for a 10-minute period before withdrawal
of the ventilator, followed by passive flow oxygen. A 10-minute period of apnea is
sufficient to attain hypercarbia (60 Torr or greater) which adequately stimulates a
respiratory effort. Testing or arterial blood gases can be used to confirm this
level. Any spontaneous breathing efforts indicates that part of the brainstem is
functioning and that the patient is not brain dead.
In the absence of confirmatory tests, the seven conditions described above must
persist unchanged for at least six (6) hours. A confirmatory test may shorten the
observation period.
Special Circumstances
• In cases of anoxic brain death, with demonstrated electrocerebral (EEG) silence
but without radiologic, nuclear scan, or doppler demonstration of absence of
cerebral circulation, a six-hour period of observation and repeat examination,
excluding apnea testing, is required.
• In cases of children under the age of one year, where absence of cerebral
circulation has not been demonstrated, a 72-hour period of observation and
demonstrated isoelectric activity
on EEG at the end of the observation period is required.
• In cases of children age one through five, where absence of cerebral circulation
has not be demonstrated, a 24-hour period of observation and demonstrated
isoelectric activity on
EEG at the end of the observation period is required.
• In cases of gross anatomical brain injury, the period of observation for the
persistence
of clinical criteria for cerebral death may be reduced to one hour.
Gross anatomical brain damage may be appropriately assessed by physical
examination or craniotomy or by cranial MRI or CT studies, interpreted by a staff
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radiologist, that indicate that the brain is irreparably damaged, extruded, divided,
or destroyed.
Pronouncing Death
In all cases, the patient’s physician will arrive within one hour to pronounce a
patient dead.
Notification of death must be provided to Admitting within one hour of
pronouncement of death.
Organ Donation
In the case of potential organ donors:
• All criteria of HP06-27, Organ and Tissue Procurement for Transplantation,
must be met.
• Legally responsible family member or designated health care surrogate must
provide
witnessed informed consent for specific donation.
The two physicians determining death must not be involved in determining the
suitability of the donor and must not be members of the surgical team performing
the transplant.
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UNIVERSITY OF KENTUCKY POLICY NUMBER: HP05-25
CHANDLER HOSPITAL FIRST ISSUED: 5/85
HOSPITAL POLICY CURRENT AS OF: 3/08
"Coroner's case" means a case in which the coroner has reasonable cause for believing
that the death of a human being within their county was not natural (homicide, suicide,
accident, under suspicious circumstances) or poses a threat to the public health.
According to KRS 72.020 and KRS 72.025, the attending physician, designee, or any
person finding or having possession of the body of any deceased person must notify the
coroner whenever the death:
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In recognition of an Attorney General's Opinion (OAG 62-964), University of Kentucky
Hospital also requires that:
• Deaths that occur in the operating room or PACU in which the operation was
necessitated by violence -- accidental or otherwise -- be reported to the coroner.
The law places responsibility for reporting to the coroner the types of death listed above
with the attending physician. As a result, University of Kentucky Hospital requires that:
1. The attending physician will
A. determine whether each death is a coroner's case,
B. notify the coroner and document the notification in the appropriate section of
the
Notification of Death form, if death is determined to be a coroner's case,
C. complete and sign the Notification of Death form and submit it to Admitting
within one hour of time of death.
2. The admitting clerk will verify that the coroner has been contacted when the Admitting
Department receives the Notification of Death certificate on a coroner's case. (The
Capacity Command Center fulfills the role of Admitting when Admitting is closed.)
Note: If "coroner's case" is checked in the Notification of Death section and the
Coroner/Medical Examiner Section has not been completed and signed, the
admitting clerk will contact the coroner immediately. The coroner should be called
in any case in which doubt exists as to whether the death represents a “coroner’s
case.”
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Approved by Richard Lofgren, M.D., Chief Medical Officer
Approved by Kathleen Kopser, Interim Chief Nursing Officer
Authorized by Murray B. Clark, Jr., Associate VP for MC Operations
UNIVERSITY OF KENTUCKY HOSPITAL POLICY NUMBER: HP06-26
CHANDLER MEDICAL CENTER FIRST ISSUED: 10/07
HOSPITAL POLICY CURRENT AS OF: 10/07
INFORMATION The purpose of this policy is to outline the steps needed to enable
patients or their surrogates who have elected to withdraw life support to be able to
donate organs after death is declared on the basis of cardio-pulmonary criteria. This
policy pertains to patients where the attending physician, patient, or their surrogates
have already decided to withdraw life-sustaining support. Organ donation after cardiac
death (DCD) refers to organ donation from a deceased donor who has been declared
dead on the basis of traditional cardio-pulmonary criteria (permanent cessation of
circulatory and respiratory function), rather than on neurological "brain death" criteria
(permanent cessation of whole brain function).
Other conditions that may lead to consideration of DCD eligibility include end
stage musculoskeletal disease, pulmonary disease, and high spinal cord injury.
The decision to withdraw life sustaining measure must be made by the Hospital’s
patient care team and legal next of kin, and documented in the patient chart.
DEFINITIONS
Brain Death: The irreversible cessation of all brain functions, including brain stem
function. Donation from this group of patients is detailed in the Hospital organ donation
policy (HP06-27)
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Cardiac Death: The complete and irreversible cessation of cardiac and respiratory
function. The diagnosis of death by traditional cardiopulmonary criteria requires
confirmation of correct EKG lead placement and absent pulse via an arterial catheter.
The patient must be pulseless, apneic, and unresponsive with five minutes of ventricular
fibrillation or five minutes of electrical asystole (no complexes, agonal baseline drift only)
or five minutes of electromechanical dissociation.
Irreversibility: It is the persistent cessation of function during a 5 minute period of
observation meeting the definition of cardiac death. DCD donor death occurs when
respiration and circulation have ceased and cardiopulmonary function will not resume
spontaneously.
INSTRUCTIONS
Eligibility for Donation after Cardiac Death
As a patient is being evaluated for withdrawal of life sustaining therapy, the Kentucky
Organ Donor Affiliates (KODA) must be notified at 278-3492 or 1-800-525-3456.
After withdrawal of life sustaining therapy has been approved and documented in the
chart by nursing, an assessment will be made by KODA to determine suitability for organ
donation before addressing donation with the patient’s next of kin. If the patient’s family
raises the subject of donation, they should be informed that KODA will be contacted
immediately and the family’s interest expressed to the KODA representative, or if
already on site, guided to the KODA representative. Only a designated requestor can
approach the family regarding consent for organ donation in DCD cases.
If the patient is not already a DNR, the patient should be made so by their attending
physician, as per UK Hospital policy HP07-03, Patient Payment and Financial
Allowance.
Consultation of another critical care attending physician to assist with ICU support
through the DCD process is appropriate. Transfer of the patient to another
attending/service specifically for the DCD process is acceptable but not essential.
If the patient is initially found suitable for organ donation after cardiac death, and likely to
die soon after removal of life sustaining therapy, a KODA coordinator, in collaboration
with medical staff, will approach the family to discuss the possibility of organ donation.
If the next of kin or authorized party wishes to pursue DCD, the KODA coordinator will, if
clinically indicated, request permission from the patient's physician for the physician to
evaluate the patient for likelihood of cardiac arrest within an hour after extubation.
If the physician consents to this evaluation, the patient is disconnected from the
ventilator by a physician for a period of up to ten minutes to record vital signs and
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respiratory parameters. If, during the evaluation, the patient becomes unstable, the
patient will be replaced on the ventilator. This suggests a likelihood of expiration shortly
after extubation and DCD should be pursued.
If the patient does not meet criteria for probable cardio-pulmonary death within one hour,
no further evaluation will take place, and the coordinator will explain to the family the
reason why organ donation cannot take place. As these criteria are not absolute,
however, it may be reasonable to proceed at the family's request and with the
physician's consent.
Consent
Permission for organ donation is granted in the State of Kentucky according to the
following order:
• Authorized Donor Designation. This is listed in Section I, KRS 311.175, which
allows a signed donor card or the first person consent. Section II includes those
that follow:
• Legal spouse
• Mentally competent adult child age 18 or over.
• Parent(s) • Mentally competent adult sibling age 18 or over
• Legal Guardian of the decedent
• Person responsible for disposing of the body
Elements of consent must include an overview of the DCD process with ample
opportunity for the family to ask questions and to demonstrate understanding. The
discussion will include the need for additional testing to determine suitability; the
possibility that donation will not take place if cardiopulmonary death does not occur
within one hour following removal of life support; and that in such a case the patient will
return from the OR to their ICU bed and team or an appropriate floor bed, where comfort
care will continue. The patient’s ICU bed will be held until declaration of death has been
made or an appropriate floor bed has been designated. The family will also be informed
that if they accompany the patient to the OR, they will be asked to leave promptly after
the patient dies.
The legal next to kin may elect to consent to procedures or drug administration for the
purposes of organ donation (e.g., heparin, regitine, femoral line placement, ECMO, and
bronchoscopy). No donor-related medications will be administered or donation-related
procedures performed without specific written consent.
Once all questions have been answered, the person authorized to give consent will sign
the consent form (KODA Authorization for Removal of Anatomical Gifts consent form) for
organ and tissue donation after cardiac death. Documentation of two witnesses of
family/next of kin’s consent is required. A copy of the signed consent form will be added
to the patient’s medical record. At this time, consent will also be obtained for
administration of heparin, et al to maintain organ viability.
The family will be provided appropriate emotional and spiritual support. The KODA
coordinator, social worker and nurse will play important roles at this critical time. Pastoral
Care services will be offered and available for additional services.
Withdrawal of life support shall take place in the operating room only, and organs shall
not be retrieved in the ICU for controlled DCD donation. The family will be given the
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option of saying goodbye to their loved one in the ICU. If they wish, however, members
of the immediate family may accompany the patient to the OR and be present until the
patient expires or until an hour has passed without cardio-pulmonary arrest. (A space
must be defined in the OR for families accompanying these patients.) Families must be
informed that they must leave the OR immediately after death is declared. The KODA
family support liaison or pastoral care will accompany the family to the Hospital chapel.
The Kentucky Organ Donor Affiliates will be responsible and billed for all costs related to
the evaluation of medical suitability and recovery of organs and tissue for
transplantation. No donation-related charges will be passed to the donor family. Should
the patient not reach a determination of death in one hour, the patient will be returned to
the patient care area for further comfort measures and family support. Upon return to
ICU or floor responsibility for care reverts back to the patient’s physician of record (if
care had been transferred as part of DCD) and financial charges revert back to the
Hospital and responsible pre-DCD payors.
The KODA coordinator will arrange the time of the organ recovery based on the family's
wishes, and the availability of the OR and donor surgery team. The OR nursing
director/coordinator will be notified as soon as the decision to carry out DCD is made.
The anesthesia attending on call will be notified by the patient's critical care attending
physician or designee prior to transfer of the patient to the OR and, will be fully briefed
concerning the details of the case and the plans for DCD.
The patient will be transferred to the OR and may be accompanied by the ICU nurse
who has been managing the patient. A floor bed should be pre-arranged and held or the
patient’s ICU bed should be held during this time period in case the need arises to return
the patient to a bed because death does not occur within the hour window. If the patient
has a cardiac arrest prior to transfer to the OR, no resuscitation will be carried out in
deference to the patient's DNR status. Organ recovery may still be possible if
circumstances allow.
The patient will be prepped and draped prior to extubation in order to keep warm organ
ischemic time to a minimum, however, the donor surgery team will not be present in the
OR during the terminal wean and until the patient has been declared dead. This will
have been explained to the family at the time consent was obtained. If the patient's
family chooses to be in the OR, they will be accompanied by the KODA coordinator. In
pediatric cases, exceptions can be made, with agreement between the surgeon and
family, as some families may wish to hold their child during this time. The patient should
be accompanied to the OR by a staff member from the primary care team (ICU nurse) or
OR support personnel but without involvement of any anesthesia provider.
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Appropriate orders must be documented prior to transfer to the OR, as follows:
● Heparin to be administered in the OR as per organ donation protocol
● Authorization for transport on monitor and, if necessary, opiate titration for comfort.
● A DNR order must be in place.
Whether and how much to sedate the patient pre-extubation is a decision that will be
made by the patient’s treating physicians following accepted hospital practice. It is a
decision that should not be influenced by the possibility of organ donation. The usual
measures to ensure patient comfort will be followed. If opioids or sedatives are to be
used, they may be administered in the ICU prior to transfer to the OR, or they may be
begun in the OR. Further dose titration will be left to the discretion of the physician who
will be managing the patient in the OR.
The comfort and dignity of the patient will be maximized during the withdrawal process
as it is in every patient death. All members of care team must ensure that the highest
level of palliative care is provided to the patient at all times during this process.
No member of the transplant team shall be present for the withdrawal of life sustaining
measures.
No member of the organ recovery team or OPO staff may participate in the guidance or
administration of palliative care, or declaration of death.
Pronouncement of Death
If, within one hour following life support withdrawal, the patient develops apnea,
asystole, or pulseless electrical activity that is sustained for a period of five minutes (as
defined in the Definitions section of this policy), the patient then will be pronounced dead
by the attending critical care physician or designee (licensed fellow/resident).
The physician will record the date and time of death in the medical record and will fill out
the death certificate.
The physician declaring death may not participate in the procedure to retrieve or
transplant the organ(s) or be associated with the transplant team or with the care of any
potential recipient. After pronouncement of death, the patient's family will be escorted
from the OR. If not present in the OR, the family will be notified promptly by the declaring
physician or designee that death has occurred and that organ recovery will proceed.
The donor surgery team will proceed with organ retrieval immediately after the patient is
pronounced dead.
211
external reviewers are invited to observe the case reviews as well. Quality
documentation throughout the process is critical for these reviews to be able to ensure
compliance with the procedures, identify potential or actual problems as well as to
protect the interests of the organ donors, recipients, and health care workers.
212
UNIVERSITY OF KENTUCKY HOSPITAL POLICY NUMBER: HP06-27
CHANDLER MEDICAL CENTER FIRST ISSUED: 1/87
HOSPITAL POLICY CURRENT AS OF: 7/07
INFORMATION
Federal and state legislation requires hospitals to consider every death as a potential
organ or tissue donation. These laws also require that families be given the opportunity
to donate. The families should be approached with discretion and sensitivity as
appropriate to the circumstances, beliefs, and desires of the family and potential donor.
All Hospital personnel shall make an effort to convey to the attending physician any
known intent of any hospitalized patient to make a donation of all or part of the body.
Note: The attending physician or another physician designated by the attending
physician will make the declaration of death in accordance with recognized
criteria
(see Hospital policy HP06-19, Diagnosis of Death).
INSTRUCTIONS
1. Whenever a patient has died or death is imminent, the attending physician, another
physician designated by the attending physician, or the nurse shall notify Kentucky
Organ Donor Affiliates (KODA) immediately. Imminent deaths should be referred to
KODA after determining the Glasgow coma score is equal to or less than 4 or if for a
severely brain injured patient’s further care is determined to be futile; and prior to
discontinuing ventilation or life-sustaining treatments when the decision is made to
downgrade, withhold, or withdraw care. KODA will evaluate the patient for suitability of
donation. Only a KODA representative or a designated requester trained by KODA, in
collaboration with the health care staff, may speak with the family about the option to
donate or deny organ donation. KODA will participate in approaching the family to
discuss the option to donate or deny organ donation, and will document the family’s
decision. KODA will also assist with family counseling. If the patient is determined to be
suitable for donation the Hospital will work with KODA to maintain the potential donor
while the necessary testing and placement of organs, tissues, and eyes takes place.
The local KODA telephone number is 278-3492; the 24-hour number is (800) 525-3456
or (866) 206-9250.
213
2. The attending physician, physician designee, or the nurse shall document on the
Provisional Report of Death form that KODA has been contacted, including the name of
the KODA coordinator, if the patient is suitable for donation and the name of the family
member 2 -- HP06-27, Organ and Tissue Procurement for Transplantation approached
with their decision. The Provisional Report of Death form shall be filed in the patient's
medical record.
3. All cardiac standstill deaths (i.e., when artificial respiration and circulation are not
maintained and there is an irreversible cessation of spontaneous respiration and
circulation) will be referred to KODA by the attending physician, the physician designee,
or the nurse. KODA will obtain information about the deceased from the caller and
determine if the patient is suitable for tissue donation. If the patient is suitable, as
determined by KODA, KODA may direct the caller to contact a Hospital staff member
who is trained as a designated requester to offer the family tissue donation (corneas,
eyes, skin, bone, veins, and heart valves). If the family desires donation or wants more
information the caller will contact KODA to speak with the family. The trained designated
requester’s name will be documented in the medical record or the provisional report of
death.
4. When a staff member informs KODA of a potential organ/tissue/eye donor they should
report the patient’s name, age, reason for hospitalization, and medical/social history;
they should also have the patient’s medical record available in order to provide any
additional information as appropriate.
Two witnesses should confirm the next-of-kin consent by signing the witness
attestation on the Kentucky Organ Donor Affiliates/Lion’s Eye Bank Authorization
for Removal of Anatomical Gifts form.
214
The original copy of the authorization form should be filed in the medical record.
6. The KODA coordinator can write all medication and patient care orders in accordance
with established guidelines and protocols for the care of the donor patient.
a. The name and title ―KODA coordinator‖ shall be documented in the body of the
order, not in the physician’s signature space.
b. Pharmacy shall dispense medications upon the written order of the KODA
coordinator. c. All orders will be implemented by the nursing staff upon the
written order of the KODA coordinator.
d. The medication orders written by the KODA coordinator shall be countersigned
by the transplant physician at the time of the organ recovery, or within 24 hours
of the time the orders were written.
215
Brain Dead Organ Donor Management
Guidelines and Protocols
University of Kentucky
Lexington KY
E Hessel
Version 1.1 (July 12, 2007; 1400)
A new paradigm in management of brain dead organ donors has appeared in the last
few years based upon the observation that the pathophysiologic changes associated
with brain death may not result in irreversible organ damage, but in fact that if the donor
is optimally resuscitated, function of potentially transplantable organs may improve
resulting in more and better organs being available for successful transplantation. This
improved outcome (number of organ donors, and number of organs donated and
transplanted per donor) has been found to be associated with initiation of protocols and
involvement of Intensivists in the management of these donors. The recognition that by
improving the care of one brain dead donor, an intensivist may have the ability to
improve the lives of six other patients is a powerful incentive for this effort.
Instead of rushing the donor to the operating room to retrieve the organs as soon as
possible, the new paradigm proposes that it is important to take the necessary time in
the ICU to optimize multi-organ function to improve transplant outcomes. Resuscitation
and re-evaluation can improve reversible organ dysfunction (myocardial and
cardiovascular dysfunction, oxygenation impairment related to potentially reversible lung
injury, invasive bacterial infections, and hypernatremia) and allow time to evaluate
temporal trends in hepatic aspartate aminotransferase (AST), alanine aminotransferase
(ALT) and creatinine or any other potentially treatable situation. This treatment period
can range from 12 to 24 h and should be accompanied by frequent re-evaluation to
demonstrate improvement in organ function toward defined targets. Once organ function
is optimized, surgical procurement procedures should be arranged emergently. (Shemie,
2006)
216
c. Latest laboratory studies
d. Determine if meeting KODA goals (see Item IV below)
e. Look for common complication
i. Hypothermia or hyperthermia
ii. Hypertension and tachycardia
iii. Hypotension and/ or low cardiac output
iv. Pulmonary dysfunction and/ or edema
v. Lactic acidosis
vi. Hypernatremia
vii. Other electrolyte abnormalities
viii. Diabetes Insipidus
ix. Hypovolemia
x. Coagulopathy
4. Confirm orders regarding routine management (see item V. below) and
laboratory studies (see item VII. below)
5. Initiate any required invasive monitoring (see item VI below)
6. Assure appropriate consultation i.e., with pulmonary medicine and cardiology,
and potential receiving transplant surgeons.
7. Initiate interventions to achieve KODA donor goals.
217
V. General Management of all potential donors
1. Baseline laboratory studies and evaluation (See item VII below)
2. Insert monitors
a. Arterial line
b. Central venous catheter
If not already in place, insert introducer suitable for placement of a
PAC and volume therapy (e.g., MAC or AVA) via right internal
jugular vein
c. Pulmonary artery catheter (Consider- see item VI.11 below)
d. Urinary catheter (If not already in, place temperature sensing catheter)
e. Core temperature monitor
f. TEE (Consider- see item VI. 12 below)
3. NG tube to low suction
4. Type and cross match 4 units
5. Ventilator
a. PRVC
b. Tidal volume 8-10 ml/kg (IBW) (Canadian)
c. PEEP 5 (Canadian)
d. Peak pressure limit 30 cm H2O (Canadian)
If not possible, consider lowering tidal volume or accepting higher
pressure limit
e. FiO2 as low as possible to maintain SpO2 ≥ 95%
f. Rate initially about 10
Adjust to keep PaCO2 and pH within desired limits
g. Obtain ABG within 30 minutes of initial settings
Recruitment maneuvers periodically (Canadian) or at least if PaO2
<80 on FiO2 >0.4, or P/F < 300:
-PC ventilation with PAP 25 and PEEP 15 for 2 hours
(SALT protocol; Angel 2006), or
-Sustained positive pressure of 30 cm H2O for 30-60 sec
218
VI. Monitoring (At least hourly unless stated otherwise)
1. Arterial pressure (S, D, M)
2. Pulse oximetry
3. ECG: Heart rate and rhythm
4. Respiratory rate,
5. Ventilator settings (Mode, tidal volume, FiO2, PEEP, PAP)
6. Temperature (central: bladder, esophageal, PA, nasopharyngeal)
7. Urine output
8. Fluid balance
9. CVP
10. Central venous oxygen [continuous or intermittent (Q4h)]
11. PAC: Consider in all. Definite if hemodynamically unstable (hypotensive, evidence of
impaired
cardiac output requiring vasopressors or inotropic drugs or fluid boluses) potential lung and
or
heart donors
12. TEE: consider in hemodynamically unstable patients, or if abnormal TTE
13. Repeat laboratory studies (See item VII below)
7. Calcium Daily
8. Magnesium Daily
9. Phosphorous Daily
10. Liver panel Q 6 hrs
a. AST
b. ALT
c. Bilirubin
11. Viral studies
a. Hepatitis B surface antigen HBsAg) and HCVAb
b. HIV
12. Type and screen
13. Coagulation studies Daily
a. PT, INR
b. PTT
14. CBC (WBC, hematocrit/hemoglobin, platelet count)
15. Urine analysis and microscopic examination
16. Blood culture x 2 Daily
17. Urine culture Daily
18. Sputum gram stain Daily
19. Sputum culture Daily
20. ABG Q 6 hr
21. ECG
22. CXR Q 12 hrs
219
23. TTE or TEE
24. Troponin; repeat in 6 hours Q 12 hours
25. Lactate Q 2-4 hours
26. Central venous or mixed venous hemoglobin oxygen saturation;
Q 2-4 hours
27. See special evaluation and laboratory studies for potential Lung and Heart
donors (item IX. A. and B below).
B. Tachycardia
-No established guidelines
-Need to differentiate the probable cause of the tachycardia, i.e., whether it is
compensatory for a low stroke volume (depressed LV function) or is secondary
to the hyperdynamic state due to CNS injury.
- If due to latter, and not needed to maintain cardiac output, consider
administering esmolol bolus followed by an infusion (see item VIII.A. above.)
220
-If hematocrit >30
-Give fluid bolus over 20-30 minutes
-500 ml hetastarch, or
-500 ml 5% albumen, or
- 15 ml/kg crystalloid (e.g., NS, LR)
-If cardiac output is still low
-Start dobutamine (5-20mcg/kg/min) or milrinone (load 50
mcg/kg over 20 minutes; infuse 0.5 mcg/kg/min)
E. Hyperglycemia
If > 500 mg/dl administer 20 units insulin and begin infusion (below)
If > 400 mg/dl administer 15 units insulin and begin infusion (below)
If > 300 mg/dl administer 10 units insulin and begin infusion (below)
If > 200 mg/dl administer 5 units insulin and begin infusion (below)
If > 100 mg/dl administer 3 units insulin and begin infusion (below)
Plus infusion
Start at 0.1 unit/kg/hour and adjust to keep glucose 80-120
F. Hypocalcemia
If Ionized <4.6mg/dl or total <8.5 mg/dl give calcium gluconate 1 gm over 10 mins
If ionized <3.6mg/ml or total<7.0mg/dl give calcium gluconate 2 gms over 30
mins
G. Hypernatremia
If > 155 administer D5/water at 100 ml per hour (expected to decrease serum
sodium about 0.4 mEq/ hour in 70 kg patient)
If > 150 administer D5/water at 50 ml per hour (expected to decrease serum
sodium about 0.2 mEq/ hour in 70 kg patient)
H. Hypomagnesemia
If <1.8 gm/dl give 1 gm Magnesium sulfate over 30 minutes
If <1.2 gm/dl give 1 gm magnesium sulfate over 60 minutes
I. Hypohosphatemia
If < 2.7 mg/ml give potassium phosphate 9 mMol/150 ml over 4 hours
If < 1.0 give potassium phosphate 15 mMol/150 ml over 4 hours
221
L. Prevention of Contrast Induced Nephropathy (CIN) (REMEDIAL protocol, Briguori et
al, Circ 2007; 115; 1211-7)
If plan to perform contrast arteriogram (coronaries, hepatic or renal)
-Assure normovolemia
-Administer1200 mg of N-acetylcysteine (NAC) enterally immediately and
repeat in 24 hours
-Infuse 154mEq/L sodium bicarbonate in D5/W at 3ml/kg/hr for one hour
immediately before contrast injection and at a rate of one ml/kg/hour for 6
hours afterwards.
Management
T4 protocol ((KODA)
1. 50 ml of 50% dextrose plus 20 units of insulin
2. Solumedrol 2 gm
3. Levothyroxine (T4) 20 mcg bolus followed by 10mcg/hour
4. Vasopressin 1 unit/hour
5. Replace potassium as needed.
222
B. Lung
Special evaluation
1. PAC
2. Measure P/F ratio after being on 100% oxygen and 5 cm PEEP for 10
minutes. Repeat Q 4 hours
3. CXR. Repeat Q 4 hours
4. Bronchoscopy and BAL
5. If Louisville KY Jewish Hospital recipient
Obtain sputum culture for AFB and toxoplasmosis
6. Pulmonary medicine consult for evaluation as possible lung donor
Management
1. Narcan 10 mg
2. LINK vest treatment 10 min per hour
3. If Louisville KY Jewish Hospital recipient
a. Administer gentamicin 80 mg as aerosol
b. Vancomycin 1 gm, ceftazidime, 1 gm, cleocin 900 mg.
4. If P/F < 300 repeat bronchoscopy and performs recruitment
maneuvers:
a. PC ventilation with PAP 25 and PEEP 15 for 2 hours (SALT
protocol per Angel 2006)), or
b. Sustained positive pressure of 30 cm H2O for 30-60 sec
5. Rotate full lateral Q 2 hours
6. Diurese to normovolemia (ARDS-Net Conservative fluid management
protocol, NEJM 2006; 354; 2562-75):
C. Liver
Nothing special other than treating hypernatremia (see item [Link])
D. Kidney
Nothing special other than CIN prophylaxis (see item VIII. L above)
223
X. A. References
1. KODA Routine Adult Organ Donor Maintenance Orders, Revised June 2007
3. Angel LF, Levine DJ, Restrepo MI, et al. Impact of a lung transplantation donor-
management protocol on lung donation and recipient outcomes. Am J Respir Crit Care
Med. 2006;174(6):710-6.
4. National heart. Lung and blood institute acute respiratory distress syndrome (ARDS)
clinical trials network. Comparison to two fluid-management strategies in acute lung
injury. NEJM 2006; 354(24): 2564-75
7. Dellinger PR, Carlet JM, Masur H, etal. Surviving sepsis campaign guidelines for
management of severe sepsis and septic shock. Crit Care Med 2004; 32(3): 858-73
8. Osman D, Ridel C, Ray P, Monnet X, Anguel N, Richard C, Teboul JL. Cardiac filling
pressures are not appropriate to predict hemodynamic response to volume challenge.
Crit Care Med. 2007 Jan;35(1):64-8.
2. Demetriou JL, Foale RD, Ladlow J, et al. Canine and feline pyothorax: a retrospective
study of 50 cases in the UK and Ireland. J Small Anim Pract. 2002;43(9):388-94.
3. Hevesi ZG, Lopukhin SY, Angelini G, Coursin DB. Supportive care after brain death
for the donor candidate. Int Anesthesiol Clin. 2006;44(3):21-34.
4. Wood KE, Becker BN, McCartney JG, et al. Care of the potential organ donor. N Engl
J Med. 2004;351(26):2730-9.
224
UNIVERSITY OF KENTUCKY HOSPITAL POLICY NUMBER:
HP06-17
CHANDLER MEDICAL CENTER FIRST ISSUED: 11/85
HOSPITAL POLICY CURRENT AS OF: 12/07
INFORMATION
The University of Kentucky Hospital recognizes that the dignity of human life and the
right of all persons to a dignified and peaceful death requires that a medical procedure or
treatment should be used only if it furthers one or both of the following goals:
• the relief of suffering, or
• the prolongation of a life satisfactory to the patient.
Definition of Terms
Do-Not-Resuscitate (DNR) is an order to withhold cardiopulmonary resuscitation
measures (as designated by the American Heart Association standards) that could
restore blood circulation and breathing in a person who has experienced cardiac and/or
respiratory arrest.
Patient without Decision-Making Capacity is a person who has been declared legally
incompetent by a court of law; or a minor (under 18 years of age, unless the minor is
emancipated); or an adult, or an emancipated minor, who is unable to appreciate the
reasonably foreseeable consequences of a decision or lack of decision, and unable to
communicate a decision. A person may be incapable with respect to some treatments
and capable with respect to others. A person may be incapable with respect to a
treatment at one time and capable at another.
225
Family or Proxy
• the adult child of the patient, or if the patient has more than one child the
majority of the adult children who are reasonably available for consultation; or
• the parents of the patient; or
• the nearest living relative of the patient, or if more than one relative of the same
relation is reasonably available for consultation; or
• the majority of the nearest living relatives. (KRS 311.631); or
• any other person designated by the patient orally or in writing when the patient
had capacity.
Kentucky Emergency Medical Services (EMS) DNR is the form designed and supplied
by the state for use by EMS personnel that allows DNR status for a transported patient.
Advance Directives
• Living Will is a written declaration instructing the maker’s (patient’s) physician to
withhold or withdraw life prolonging treatment if the patient has a terminal condition or is
permanently unconscious. (KRS 311.625)
• Substitute or Proxy Decision Maker is a person(s), including a health care surrogate,
who has the responsibility (subject to certain limitations) to make decisions on behalf of
a patient without decisional capacity. (KRS 311.625)
INSTRUCTIONS
Autonomy
The decision to withhold or withdraw any potentially life-sustaining treatment should be
based on the principle that a patient with decision-making capacity has the right to reject
potentially life-sustaining treatment even when such treatment is medically
recommended.
226
rarely be the case if the patient is provided access to appropriate palliative care.
Double Effect
Relief of pain and suffering is central to exemplary patient care. Commitment to the relief
of pain and suffering of the dying patient is essential, even if death may be hastened as
a result of effectively relieving the patient’s pain.
Category 1
Full support, including all CPR modalities. The patient receives full resuscitative care.
Unless an attending physician writes an order to the contrary, the patient will be provided
full support, including CPR.
Category 2
DNR. This order only addresses cardiopulmonary resuscitation and does not specifically
limit other appropriate treatments. Active DNR orders will not automatically be rescinded
when patients leave their hospital units for invasive or non-invasive testing.
Note: It is inappropriate to divide resuscitation into components that could be initiated
(e.g. defibrillation, pharmacologic agents) without providing others (e.g. airway
management, mechanical ventilation.
Category 3
Withholding/withdrawing other measures. The patient (or proxy) may, in consultation
with the attending provider decline these treatments. When an order to discontinue
treatment is written but the physical means for providing the treatment remains in place
(e.g., mechanical ventilator or feeding tube) the physician or other willing member of the
health care team should remove or disconnect the treatment device with the assistance
of auxiliary staff, as appropriate. If upon removal of said device, death is expected
imminently, then a physician member of the health care team should be present.
227
• Determining the appropriate surrogate decision-maker: If an adult patient does not
have decision-making capacity, then healthcare decisions on behalf of the patient may
be made by any of the following responsible parties in the following order of priority:
1. The judicially appointed guardian to the patient, if medical decisions are within
the scope of the guardianship;
2. Person named in a properly executed written advance directive;
3. The spouse of the patient;
4. An adult child of the patient, or if the patient has more than one child, the
majority of the adult children who are reasonably available for consultation;
5. The parents of the patient;
6. The nearest living relative of the patient, or if more than one relative of the
same relation is reasonably available for consultation, the majority of the nearest
living relatives. (KRS 311.631);
7. Any other person designated by the patient orally or in writing when the patient
had capacity.
Minors
If a patient is a minor child under the age of eighteen years and not emancipated under
the laws of the Commonwealth of Kentucky, treatment may be withheld or withdrawn
when:
1. the parent(s) or legal guardian of the minor patient expresses to the physician
an understanding of the purpose of treatment and the risks of forgoing the
treatment and declines life-sustaining treatment or indicates that such treatment
be withdrawn.
2. and advance directive has been issued on behalf of the patient declining
treatment and any conditions contained in the directive are met. (Kentucky law)
3. Treatment is medically inappropriate.
An individual authorized to consent for another shall act in good faith, in accordance with
any advance directive executed by the individual who lacks decisional capacity, and in
the best interest of the patient who does not have decisional capacity. (KRS 311.631)
The physician is responsible for providing the patient or surrogate decision maker with
all pertinent information concerning the possible benefits and risks of treatment (see
Hospital policy HP06-09, Consent to Treatment). If support services (e.g., palliative care,
pastoral care, social services) have not been involved with the patient’s case, the
attending physician is encouraged to engage these services when a decision to forgo
life-sustaining treatment is made.
228
Wards of the State
1. When the attending physician believes that potentially life-sustaining treatment should
be withdrawn/withheld from a patient who is a ward of the state, the family social worker
should be contacted to obtain the necessary state documents to request
withdrawing/withholding treatment.
Note: State regulations specify that withdrawing or withholding potentially life-sustaining
treatment can only be considered for patients who are terminally ill and permanently
unconscious.
2. The attending physician will complete the state-provided form requesting permission
to withhold/withdraw treatment. The attending physician will also request a second
physician to complete a consultative opinion form, also provided by the state, concurring
in the request.
3. The attending physician may or may not seek a consultation from the Clinical Ethics
Consult Service prior to making such a request.
4. The completed documents will be submitted to the patient’s state guardian, who, in
turn, will submit them to the state health decision committee for review. This review is
usually completed in 24 to 48 hours.
5. After receiving written notice from the guardian of the state health decision
committee’s approval, the physician may write orders for the withdrawing/withholding of
potentially life-sustaining treatment (see Documentation section below). The written
notice and physician orders will be added to the patient’s chart.
Documentation
1. In any case in which a health care decision is made, the decision shall be noted
in writing in the patient’s medical record. (KRS 311.631) The note should include the
parties involved, key elements of the discussion and the agreed upon conclusions or
plans. If the level of support designated for a patient excludes CPR, the attending
physician should initiate a conversation with the patient (or proxy). Documentation in the
chart of this discussion should include:
• patient decisional capacity when the order was given, and
• patient consent to the level of support, and
• if the patient did not have decisional capacity:
• whether the patient had a living will that authorized the level of support,
and
• the name and relationship of the proxy who, on the behalf of the patient
consented to the level of support.
• a written order by the attending physician to reflect the decision made.
2. An order must be entered to institute DNR status or withdraw life-sustaining
measures.
3. Telephone Orders: Verbal orders for DNR may be given by an attending physician to
a senior resident in exceptional situations. In such situations, the resident may write the
order, but the attending physician must personally cosign the order and document the
related information within 24 hours.
229
4. Review and Revocation of DNR Orders: The patient with decision-making capacity (or
proxy) who initially requested a DNR order has the right to change his/her mind and
retract the agreement, requesting a different level of support by informing the physician.
DNR orders should be reviewed at least every 30 days.
Conflicts
The attending physician is responsible for taking the lead in resolving conflicts between
or among interested parties including the patient, surrogate, family members and health
care team members.
If the disagreement or confusion is not resolved, the patient (or proxy), the patient’s
family, or any member of the health care team can request a clinical ethics consult
through the Clinical Ethics Consult Service.
If the attending physician, in consultation with a patient’s care team, decides that certain
therapeutic interventions are medically inappropriate for the patient, the physician should
discuss carefully with the patient (or proxy) the nature of the ailment; the care options
available and useful for the patient, including palliative and hospice care; the prognosis;
and the reasons why certain interventions are considered inappropriate. If the patient (or
proxy) and the attending physician agree, treatment may be discontinued.
If the patient (or proxy) and the attending physician do not agree with reference to the
futility of a proposed course, the attending physician will discuss with the patient (or
proxy) obtaining an additional medical opinion on the patient’s case from another
physician. The physician who will provide the second opinion will be selected by the
chief medical officer.
If, after considering the opinion of the attending physician and a concurring opinion of
the consulting physician, the patient (or proxy) desires interventions that have been
judged to be inappropriate, and if the attending physician does not want to proceed with
such interventions, the attending physician should:
• request a clinical ethics consult through the Clinical Ethics Consult Service;
• inform the patient (or proxy) that the request has been made; and
• inform the patient (or proxy) that the patient is at full liberty to effect the patient’s
transfer to another hospital or to another physician.
If the consulting physician disagrees with the attending physician about the treatment of
the patient, and if the patient (or proxy) desires, the chief medical officer will facilitate the
patient’s (or proxy’s) request for transfer to another physician.
230
If the patient (or proxy) initiates a clinical ethics consult, a clinical ethics consultant
(CEC) will be contacted and will review the case and make recommendations in a timely
fashion. The CEC may call in additional ethics consultants, or other expert consultants
as needed, or bring the case before the entire Hospital Ethics Committee for review. A
CEC letter will document the issues in the case and be inserted into the patient’s chart.
If the CEC agrees with the attending physician that such treatment is inappropriate for
this patient, such treatment will not be initiated or will be discontinued and document the
decision in the patient’s record.
If the CEC disagrees with the attending physician and if the attending physician wishes
to withdraw from the case, the CEC will contact the chief medical officer, to either secure
an attending physician to carry out the patient’s wishes or, failing this, will attempt to
effect the patient’s transfer to another hospital.
Special Situations
EMS Transport of Patients with or Without DNR Designations
Kentucky law requires that patients with DNR designations have a complete, signed
original EMS DNR form, which is given to EMS personnel before transport.
DNR orders without EMS DNR forms cannot be honored by EMS transport personnel.
The EMS DNR form must be notarized, or it may be witnessed by two persons who are
not:
• blood relatives of the patient
• beneficiaries under the descent and distribution laws, or
• directly financially responsible for the patient’s health care.
Note: An EMS DNR can be witnessed by Hospital staff as long as those staff members
are not related to the patient or responsible for the patient’s health care.
231
attached to the patient.
6. The transport staff will give the original Kentucky EMS DNR form to the patient
or responsible family member upon arrival at their destination.
7. If the patient has a medical emergency during transport, EMS personnel will
retain the original EMS DNR form.
Surgical, anesthesia, and invasive diagnostic procedures for patient with DNR
order or other orders limiting life-saving measures.
A patient with a DNR order may require anesthesia and surgery for palliative care or
other medical reasons. In such circumstances, discussion and clarification of the support
status between the patient (or proxy) and attending surgeon and anesthesiologist will
occur prior to admission to the operating room or procedure area. Consent to anesthesia
and surgery requires delineating the usual treatments necessary to prevent foreseeable
and unanticipated life-threatening events during surgery (e.g., endotracheal intubation,
intravenous fluids, CPR, blood products and inotropic drugs, etc.). Written consent for
surgery and anesthesia will be required by both surgeon and anesthesiologist. Following
that discussion, a note will be written in the chart delineating which forms of life support
will be utilized in the event of need during the perioperative or periprocedural period.
After completion of the post-anesthesia or post-procedure recovery period, a new DNR
order should be written. It seems reasonable to apply these principles to all procedures
requiring informed consent. A review of the risks and benefits of the procedure and its
alternatives should include a review of the DNR decision. DNR orders should not be
automatically rescinded without discussion with patient or proxy.
When intraoperative or periprocedural arrest results in the death of a patient who has a
DNR order in force, the death will be classified ―expected‖ rather than ―unexpected.‖
232
SECTION 8: DISASTER MANAGEMENT
Alert 1
UK Hospital utilizes the Hospital Emergency Incident Command System (HEICS) as its
command and control structure to facilitate response to emergency situations.
In addition, the Hospital has developed a contingency plan to guide Hospital personnel
in the early stages of response to a mass casualty disaster, until a command center can
be established.
This plan assumes that the Hospital and its operations have not been affected by the
disaster. If the Hospital has been damaged or operations have been interrupted, the
incident commander or liaison will notify DEEM to channel patients to alternate health
care facilities until necessary operations can be restored.
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The Hospital uses the code Alert 1 to notify staff that the community has experienced a
mass casualty disaster and that the Hospital is preparing to receive an influx of patients.
The Alert 1 announcement is made over the paging system and preceded by chimes.
For the purposes of this plan, triage is defined as the sorting and classifying of incoming
patients so that they can be channeled to appropriate treatment areas. Although medical
care may be available, treatment at the triage point will be kept to a minimum to facilitate
prompt attention to all disaster victims.
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Treatment Areas
• Triage—ED parking lot
• Critical and Acute Treatment—ED trauma bays
• Acute Overflow Treatment—Pre-operative holding area
• Minor Treatment, General Adult—Cardiac Cath recovery area
• Minor Treatment, Adult Psychiatric—3 West
• Minor Treatment, Obstetric—Labor and Delivery
• Minor Treatment, Pediatric—Children’s Hospital
• Morgue—Morgue
Medical Center PPD will hold both CCC patient transport elevators w on the ground
floor. One will be used to transport victims from the Emergency Department to other
treatment areas. The second patient transport elevator will be used to transport patients
from OR and ICU to inpatient units.
The Kentucky Clinic will close all clinics when an Alert 1 has been confirmed and issued.
Kentucky Clinic personnel who do not have specific disaster response assignments will
report to the personnel pool. Clinic areas may be used as back-up treatment areas as
needed during disaster response.
To facilitate immediate response while the command center is being established, the
Alert 1 responsibilities of certain staff are outlined in this plan and in departmental plans.
Staff who has specific assignments will report to designated locations to assume their
roles.
Nurses who do not have specific assignments will report to the Nursing Resources
Allocation Center. Other employees who do not have specific assignments will report to
the personnel pool.
Off duty employees who are reporting for emergency response duties will enter the
Hospital grounds from University Drive and park in the Hospital or KY Clinic parking
structure. Staff will be required to present their Medical Center ID badges to enter
hospital grounds.
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Work Shifts
During emergency response all Hospital employees are considered essential personnel.
• If an employee is on duty at the time the Alert 1 is announced, they are
expected to remain on duty until notified that disaster response efforts are
complete or until they are officially relieved of duty.
• If an employee is notified to report to the Hospital for disaster response duty,
they are expected to report as soon as possible and to remain on duty until the
disaster response effort is complete or until they are officially relieved of duty.
Before finalizing the plan, the service director will submit the plan to the Hospital safety
officer and Emergency Management Subcommittee for approval. Once the plan has
been finalized, the director will provide comprehensive training for their employees. New
employees will be trained as a part of departmental orientation.
The director will review the plan at least annually and will revise the plan whenever
personnel, location, or structural changes occur.
Each employee learns about their role in emergency management and specific
contingency plans during departmental orientation and completes the emergency
management CBL. Continuing education is provided annually and whenever the plan
changes.
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Appendix 1
Initial Roles and Responsibilities under Plan
Incident Commander
• When notified of community disaster (Alert 1)
• Gather information about disaster from ED Attending
• Type and nature of incident
• Number of casualties
• Number of patients coming to UK
• Nature of injuries
• ETA
In consultation with ED Attending, determine whether to initiate Alert 1. If decision is
made to initiate Alert 1:
• Notify paging operator, 3-5200.
• Report to N102, establish Command Center
• Assign HEICS roles as appropriate to disaster
Under normal circumstances, the HOA will serve as the medical care director, reporting
directly to the operations chief.
• Establish auxiliary command center (H-178).
• Activate HEICS section positions.
• Assess bed and staffing availability.
• Organize information and send to operations administrator.
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• Report to ED parking lot or area where triage is established.
• After casualties are triaged, transport to designated treatment area.
• After transport, if stretcher/wheelchair is soiled, take to HA063 for cleaning.
• Return to ED entrance to continue transport.
• If assigned to clean patient rooms: (All remaining staff)
• Stand by in H16 for assignment.
Appendix 2
Patient Tracking
Initial patient information will be transmitted from triage to the Command Center by fax or
runner. The planning chief will assure that the patient tracking officer receives the
information.
• The patient tracking officer will share information with the patient information officer so
that family notification can begin.
• The patient tracking officer will maintain a log of patient locations. Patient location and
condition updates will be transmitted to the Command Center using a logging system
kept on each treatment unit.
• The patient information officer, working with the Red Cross and other agencies, will
attempt to notify the victim's family, using information on the tag. When the family is
reached, the patient information officer will document notification and send information to
public information officer in Command Center.
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UNIVERSITY OF KENTUCKY HOSPITAL POLICY NUMBER: HP12-15
CHANDLER MEDICAL CENTER FIRST ISSUED: 2/00
INFORMATION
University of Kentucky Hospital recognizes the potential for a chemical, biological agent,
or radiation exposure posed by Hospital operations, University and community
research, local industry, terrorism, or interstate transport. In an effort to respond more
effectively to such an event, the Hospital has adopted an all-hazard approach to all
disasters, using the Emergency Incident Command System (HEICS). HEICS provides for
a simplified chain of command or management structure for communications decision-
making during disasters.
University of Kentucky Hospital has a mutual aid agreement with other hospitals in the
Lexington-Fayette county area and with several community hospitals throughout the
state.
The Hospital may allow the evacuating facility to transfer staff to care for patients.
The Capacity Command Center may provide preliminary bed availability information,
but plan activation is the decision of the Hospital administrator-on-call.
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• gather appropriate information, including a contact name and telephone number
• notify the Admitting supervisor-on-call
2 -- HP12-15, Mutual Aid Plan
The administrator-on-call will confer with appropriate parties and determine the
Hospital’s ability to accept evacuated patients. (Plan activation assumes that patients
will remain at UK Hospital for at least 24 hours.)
If a decision is made to activate the mutual aid plan, the administrator-on-call will
notify:
• Evacuating hospital or LFUCG EOC (258-3847 or 258-3870)
• Admitting supervisor-on-call
• Emergency Department charge nurse
• Chief of staff
• Hospital safety officer
• Security director
• Emergency Transport communications officer
• Pharmacy director
Command Center
The Hospital recognizes that success of emergency response activities is due to an
integrated effort by all functional areas of the Hospital. In order to ensure coordination
of all Hospital resources allocated to the disaster response effort, the Hospital will
establish a command center in N102 or other suitable location.
Like most disaster response efforts, mutual aid response requires significant real-time
planning and implementation, based on the specific situation. The primary purpose of
the command center is to identify and allocate resources appropriately and to provide
administrative coordination and support for all Hospital resources allocated to the
response effort.
In most cases, Hospital administrators, the chief of staff, and other key staff will
assume disaster response responsibilities consistent with their primary responsibilities.
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Logistics: The logistics officer will work with key personnel to ensure that patient
care and support services have the supplies, equipment, and utilities necessary
to perform essential functions.
Finance Officer: The finance officer will work with key personnel to track
expenditures for cost recovery and to ensure that funds can be allocated for
special purchases essential to disaster response.
Liaison: The liaison will establish contact and work with external agencies
responding to the disaster.
HP12-15, Mutual Aid Plan -- 3
Public Relations Officer: The public relations officer will establish a media center
and provide official information to the media. The public relations officer will
coordinate release of patient information with the command center and the
family center.
Security Officer: The security officer will work with Security and other assigned
staff to limit building and grounds access.
The liaison will notify the Red Cross that the mutual aid plan has been activated and
that the Hospital is accepting patients evacuated from another facility. The Red Cross
will assist in notifying families.
If necessary, Patient & Family Services staff may establish a family center to assist in
communicating with patients’ families.
Transportation Resources
The administrator-on-call, in consultation with Emergency Transport, will determine
whether UK Hospital-based transportation resources can be allocated to assist in
moving patients from the evacuating facility to the UK Hospital.
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If the incident has resulted in casualties or the evacuating hospital is transferring
seriously ill patients, arrangements will be made for these patients to be transported
directly to and triaged in the Emergency Department.
Triage personnel will assess arriving patients and work with the Capacity Command
Center to provide expeditious bed assignment. In some cases, a boarding ward may be
established in the holding area only until beds become available. Once a room is
assigned, transport aids will transport patients and all available patient records from
the holding area to the unit.
If the Hospital segregates patients evacuated from the other hospital by opening a
vacant patient care unit, the Hospital may request clinicians from the evacuating
hospital to care for transferred patients. This decision will be based on the identified
staffing needs of UK Hospital, as the receiving facility.
4 -- HP12-15, Mutual Aid Plan
Plan Termination
The Hospital command center will oversee a phased-approach to plan termination.
Primary response will cease when all evacuated patients, designated to be transferred to
University of Kentucky Hospital, have been received, triaged, and appropriately placed.
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UNIVERSITY OF KENTUCKY HOSPITAL POLICY NUMBER: HP12-13
CHANDLER MEDICAL CENTER FIRST ISSUED: 3/92
INFORMATION
University of Kentucky Hospital recognizes the potential for a chemical, biological agent,
or radiation exposure posed by Hospital operations, University and community
research, local industry, terrorism, or interstate transport. In an effort to respond more
effectively to such an event, the Hospital has adopted an all-hazard approach to all
disasters, using the Emergency Incident Command System (HEICS). HEICS provides for
a simplified chain of command or management structure for communications decision-
making during disasters.
This plan has been developed to establish guidelines for treating patients who present
to the Emergency Department and are thought to be contaminated with chemicals,
biological agents, or radioactive materials. The plan has been developed in consultation
with the Lexington-Fayette County Division of Environmental and Emergency
Management (DEEM), which assumes responsibility for community emergency response
planning.
Each Hospital area and department that uses hazardous or radioactive materials or
handles biological agents will, in collaboration with the Hospital Environment of Care
Committee, have written procedures specific to their areas for dealing with exposures.
Note: If exposures seem crime-related, all personnel will treat personal belongings and
clothing as medico-legal specimens.
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other than normal emergency channels.
• number of victims.
• signs/symptoms of exposure.
• nature of injuries.
• name of chemicals, radioactive materials, or biological agents involved.
• extent of decontamination (MSDS, if available).
• if radiation victim, whether patient is contaminated or irradiated, and the
physical identity of the contaminant.
• estimated time of arrival of victims.
3. Request assistance from Lexington Fire Department by calling the Lexington Fire
Department dispatch if assistance is needed for unknown chemicals or biological
agents, primary contamination, or for specific information. The Lexington Fire
Department Hazardous Materials team may fax information on identified chemicals or
biological agents.
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Additional information may be obtained or requested as necessary from:
ATSDR Hotline at 1-404-639-6360,
CHEMTREC at 1-800-424-9300 and/or Poison Control,
CDC 24-hour emergency response at 1-770-488-7100
Micromedex
Departmental Response
Emergency Department staff will:
• Establish and prepare decontamination area, at the direction of the charge nurse or
decontamination leader.
• Before patient decontamination begins, the charge nurse or decontamination leader
will assign decontamination responsibilities, inspect the set-up, and brief the
decontamination team to ensure that all protocols for decontamination appropriate to
the situation and all other safety precautions are being followed.
PPD personnel will:
• Turn off air handlers in affected areas (internal exposures).
• Turn on air line valves.
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Medical Center Security will:
• Station guard at ambulatory and ambulance entrances of Emergency Department to
ensure that no contaminated patients enter the Emergency Department.
• Block off ambulance entrance area with hazardous material tape.
• Direct all other ambulances to alternative entrance.
• Block the entrance to the Children’s Hospital.
• Block off entrance to decontamination zone.
• Direct pedestrian traffic on the Hospital walkway.
• Secure the contaminated area until the waste is removed.
• Station guard at all other public entrances to the Hospital.
Note: Attendants will stay with ambulance until they and the ambulance have been
monitored for contamination.
• If attendants are not contaminated, they will be released for duty.
• If attendants are contaminated, they will proceed to decontamination area.
When the Hazardous Materials Management is notified that the Hospital has
implemented the decontamination plan, designated personnel will stand by for
notification to begin clean up and disposal activities. In cases in which mass
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casualties are transported to the Hospital for decontamination, Hazardous
Materials Management may be asked to assist in set-up of decontamination
equipment and to pump rinsate into a reservoir during decontamination
activities.
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The Trauma Alert Team plays critical roles in managing major trauma patients by quickly assembling and mobilizing essential resources for diagnosis and treatment. Physicians such as the Trauma Chief Resident or Senior Emergency Medicine Physician coordinate the response and assume roles as outlined in the Trauma Resuscitation Roles and Responsibilities . The Emergency Department (ED) nurses are responsible for preparing the trauma resuscitation room, designating roles such as Trauma Nurse #1 and #2, and documenting personnel arrival times . The Radiology Technologist ensures rapid radiograph processing, and the Ultrasound and CT Scan Technologists ascertain equipment availability . The Blood Bank personnel prepare blood within minutes of notification, and Operating Room personnel evaluate room availability and readiness for emergent intervention . In case of a Trauma Alert Red, the Pediatric Surgery Chief Resident assumes care upon arrival, providing direction for resuscitation . Only the Senior Trauma/Emergency Physician can deactivate a Trauma Alert and dismiss personnel . Responsibilities are divided among team members to ensure rapid and efficient resuscitation, evaluation, diagnosis, and treatment of patients .
Key ethical considerations in the DCD process include obtaining informed consent from the family, ensuring the patient's dignity and comfort, and maintaining a clear separation of roles for the medical team involved in withdrawal of life support and organ recovery. Family consent involves a comprehensive understanding of the DCD process, including possible outcomes and additional testing required to determine donor suitability. Written consent is mandatory before any donation-related procedures or medications are administered . Families must be informed of the process, allowed to ask questions, and support services such as KODA coordinators, pastoral care, and social workers should be offered to provide emotional and spiritual support . The family is allowed to be present during the withdrawal of life support and must be informed they need to leave immediately following the declaration of death . The withdrawal process must proceed independently of donation considerations, and any decision must respect the previously expressed wishes of the patient or the legal next of kin .
The process begins with a request for withdrawal of life-sustaining therapy, independent of organ donation considerations, following hospital policy HP06-17 . KODA coordinators discuss donation possibilities with the family if the patient is suitable for organ donation after cardiac death and likely to expire soon after life support withdrawal . Coordination involves confirming organ viability through evaluation consent and implementing specific protocols, such as administering heparin, with all consents documented . Post-pronouncement, organ retrieval is scheduled, ensuring family involvement and support throughout the process . Withdrawal of life support is managed to maintain patient dignity and is strictly separated from organ retrieval activities to prevent conflicts of interest .
When signs of feeding intolerance like high gastric residuals or abdominal distention occur, feedings should be stopped temporarily to reassess and address underlying causes . The position of the feeding tube must be confirmed via radiography, and a prokinetic agent may be added to alleviate symptoms . If symptoms persist, further clinical evaluations, such as ruling out mesenteric ischemia, are warranted . Adjustments to feeding rates or an evaluation of medication contributions, particularly those causing diarrhea, should be considered to address intolerance effectively .
Neuromuscular blockers are used in critical care for patients with severe acute respiratory failure or limited tissue oxygenation . Their use requires careful assessment to prevent over-paralysis and ensure adequate sedation and analgesia before administration . Regular monitoring using the Train-of-Four stimulation assesses the depth of the blockade to prevent prolonged paralysis . Troubleshooting includes checking electrodes and leads if expected twitch response is absent . Routine clinical indicators and assessments ensure patient safety and efficacy of the blockade .
In cases of suspected aortic injury due to blunt thoracic trauma, clinical guidelines stress the importance of considering both the mechanism of injury and findings on chest radiographs to determine the need for further diagnostic evaluation. Characteristics that necessitate further investigation include significant deceleration injuries such as high-speed motor vehicle collisions and evidence of mediastinal injury, such as a widen mediastinum seen on radiographs . If an abnormal chest radiograph is present, further imaging is recommended depending on patient stability. For stable patients, a thoracic CT scan using the aortic protocol is advised, as modern helical CT technology can provide direct evidence of aortic injury . In the event of an indeterminate CT scan, confirmatory tests such as angiography or transesophageal echocardiography (TEE) should be performed . TEE is particularly advantageous in unstable patients as it can be done at the bedside without delaying necessary interventions .
The decision-making process for using neuromuscular blocking agents (NMBAs) in critically ill patients is complicated by several factors. These include the need to appropriately manage sedation and analgesia, as NMBAs do not possess sedative or analgesic properties, and pain and anxiety assessments are challenging in paralyzed patients . Additionally, patients may have underlying conditions, such as neuromuscular diseases, renal or hepatic impairment, and potential drug interactions, that necessitate caution when using NMBAs . There are also risks of adverse effects like prolonged paralysis after NMBA discontinuation and the possibility of histamine release causing bronchospasm and cardiovascular effects . Monitoring and reversing neuromuscular blockade require careful consideration of physiological conditions such as acidosis and hypothermia, which affect NMBA dosing . Furthermore, daily discontinuation of NMBAs is necessary to assess adequacy of sedation and analgesia and to decide on the need for continued paralysis .
The primary indicators of feeding intolerance in enteral nutrition include tube feeding reflux, high gastric residuals, vomiting, aspiration, abdominal distention, and diarrhea . Tube feeding reflux is an initial sign of intolerance, especially in patients with a nasoenteral or nasogastric tube . High gastric residuals, particularly in patients with PEG feeding, should be monitored every 4-6 hours and are considered elevated if they exceed the total volume of feeding during that period; elevated residuals often require holding the feeding and reassessment . Vomiting and aspiration can also signal intolerance and typically follow patient complaints of nausea . Abdominal distention and bloating are early and reliable signs that warrant daily evaluation , and diarrhea, though often not caused by feedings themselves, can be indicative of intolerance especially if it occurs alongside high feeding rates or prolonged feeding cessation .
Pediatric Trauma Alert activation is evaluated for quality assurance by the Pediatric Trauma Coordinator who reviews all records for completeness and details of the response efforts . Criteria for assessment include the response of trauma alert members, appropriateness, conduct of resuscitation, and times to the operating room (OR) and OR preparedness .
Maintaining patency of small-bore feeding tubes involves routine flushing with 20-30 ml of water before and after medications . Other strategies include avoiding aspirations from small-bore tubes, preventing mixing of aspirated formulas with gastric acid, and ensuring feeding flow rates exceed 50 cc/hr to avoid residue buildup .