Cerebrovascular Accident Case Study
Cerebrovascular Accident Case Study
COLLEGE OF NURSING
Villasis, Santiago City
CEREBROVASCULAR ACCIDENT
A CASE STUDY
SUBMITTED BY:
BSN 3B – GROUP 3
Agangaya, Bishy-Shelly
Asingal, Kristine Joy
Ballesteros, Whella Mhay
Blanza, Arlene
Buyucan, Gay Marie
De Guzman, Jingkie
Gomez, Lord Allen
Gupuchon, Disiryl
Faustino, Avegail
Mapili, Jay Ryan
Eden Anievas, RN
Clinical Instructor
April, 2024
OBJECTIVES:
GENERAL OBJECTIVE
This case study was able to acquaint us with realization, understanding the constant overall
disease, improved our skills and acquiring a way of thinking as needed in dealing with diverse
clients, performing basic nursing skills with confidence and competence as well as providing
appropriate nursing management to a patient incapable of dealing with Cerebrovascular Accident
(CVA)
SPECIFIC OBJECTIVES:
•To establish rapport towards the patient, significant other/s and to provide our fellow student
nurses and future aspiring nursing students informative knowledge
•To present an informative overview of the disease;
•To acquire the physical assessment for further problems to be managed;
•To discuss the anatomy and pathophysiology of the affected body part of the disease;
•To determine how CVA affects the body;
•And to recognize the effects of CVA in an individual.
OVERVIEW
A Cerebrovascular Accident (CVA), an ischemic stroke or “brain attack,” is a sudden
loss of brain function resulting from a disruption of the blood supply to a part of the brain.
It is a functional abnormality of the central nervous system. The result is an interruption in the
blood supply to the brain, causing temporary or permanent loss of movement, thought, memory,
speech, or sensation.
Classifications:
Ischemic stroke. This is the loss of function in the brain as a result of a disrupted blood supply.
Hemorrhagic stroke. Hemorrhagic strokes are caused by bleeding into the brain tissue, the
ventricles, or the subarachnoid space.
Statistics:
Globally, one in four people over age 25 will have a stroke in their lifetime. 15-49 years
1,978,946 50.29 (43.02-58.71) - Each year, over 16% of all strokes occur in people 15-49 years
of age. <70 years 7,622,088 104.79 (92.54-119.06) - Each year, over 62% of all strokes occur in
people under 70 years of age. (World Stroke Organization, 2022).
The mortality from stroke during the last 10 years remains high with an average of 63,804 deaths
per year (Figure 1) (4). In 2021, despite the COVID pandemic, the recorded annual Philippine
stroke death was 68,180 (5), increased from 64,381 in 2020.
•Motor Loss
Hemiplegia, hemiparesis
Flaccid paralysis and loss of or decrease in the deep tendon reflexes (initial clinical feature)
followed by (after 48 hours) reappearance of deep reflexes and abnormally increased muscle tone
(spasticity)
•Communication Loss
Dysarthria (difficulty speaking)
Dysphasia (impaired speech) or aphasia (loss of speech)
Apraxia (inability to perform a previously learned action)
Risk factors:
Nonmodifiable
•Advanced age (older than 55 years)
•Gender (Male)
Modifiable
•Hypertension
•Atrial fibrillation
•Hyperlipidemia
•Obesity
•Smoking
•Diabetes
•Asymptomatic carotid stenosis and valvular heart disease (eg, endocarditis, prosthetic heart
valves)
•Periodontal disease
Causes:
Large artery thrombosis. Large artery thromboses are caused by atherosclerotic plaques in the
large blood vessels of the brain.
Small penetrating artery thrombosis. Small penetrating artery thrombosis affects one or more
vessels and is the most common type of ischemic stroke.
Cardiogenic emboli. Cardiogenic emboli are associated with cardiac dysrhythmias, usually
atrial fibrillation.
Complications:
Tissue ischemia. If cerebral blood flow is inadequate, the amount of oxygen supplied to the
brain is decreased, and tissue ischemia will result.
Cardiac dysrhythmias. The heart compensates for the decreased cerebral blood flow, and with
too much pumping, dysrhythmias may occur.
Diagnostics:
CT scan. Demonstrates structural abnormalities, edema, hematomas, ischemia, and infarctions.
Demonstrates structural abnormalities, edema, hematomas, ischemia, and infarctions. Note: May
not immediately reveal all changes, e.g., ischemic infarcts are not evident on CT for 8–12 hr;
however, intracerebral hemorrhage is immediately apparent; therefore, emergency CT is always
done before administering tissue plasminogen activator (t-PA). In addition, patients with TIA
commonly have a normal CT scan
PET scan. Provides data on cerebral metabolism and blood flow changes.
MRI. Shows areas of infarction, hemorrhage, AV malformations, and areas of ischemia.
Cerebral angiography. Helps determine specific cause of stroke, e.g., hemorrhage or obstructed
artery, pinpoints site of occlusion or rupture. Digital subtraction angiography evaluates patency
of cerebral vessels, identifies their position in head and neck, and detects/evaluates lesions and
vascular abnormalities.
Lumbar puncture. Pressure is usually normal and CSF is clear in cerebral thrombosis,
embolism, and TIA. Pressure elevation and grossly bloody fluid suggest subarachnoid and
intracerebral hemorrhage. CSF total protein level may be elevated in cases of thrombosis because
of inflammatory process. LP should be performed if septic embolism from bacterial endocarditis
is suspected.
Transcranial Doppler ultrasonography. Evaluates the velocity of blood flow through major
intracranial vessels; identifies AV disease, e.g., problems with carotid system (blood
flow/presence of atherosclerotic plaques).
EEG. Identifies problems based on reduced electrical activity in specific areas of infarction; and
can differentiate seizure activity from CVA damage.
Skull x-ray. May show a shift of pineal gland to the opposite side from an expanding mass;
calcifications of the internal carotid may be visible in cerebral thrombosis; partial calcification of
walls of an aneurysm may be noted in subarachnoid hemorrhage.
ECG and echocardiography. To rule out cardiac origin as source of embolus (20% of strokes
are the result of blood or vegetative emboli associated with valvular disease, dysrhythmias, or
endocarditis).
Laboratory studies to rule out systemic causes: CBC, platelet and clotting studies,
VDRL/RPR, erythrocyte sedimentation rate (ESR), chemistries (glucose, sodium).
Treatments:
Recombinant tissue plasminogen activator would be prescribed unless contraindicated, and
there should be monitoring for bleeding.
Increased ICP. Management of increased ICP includes osmotic diuretics, maintenance of
PaCO2 at 30-35 mmHg, and positioning to avoid hypoxia through elevation of the head of the
bed.
Endotracheal Tube. There is a possibility of intubation to establish patent airway if necessary.
Hemodynamic monitoring. Continuous hemodynamic monitoring should be implemented to
avoid an increase in blood pressure.
Neurologic assessment to determine if the stroke is evolving and if other acute complications
are developing
Carotid endarterectomy. This is the removal of atherosclerotic plaque or thrombus from the
carotid artery to prevent stroke in patients with occlusive disease of the extracranial cerebral
arteries.
Hemicraniectomy. Hemicraniectomy may be performed for increased ICP from brain edema in
severe cases of stroke.
Prevention
Healthy lifestyle. Leading a healthy lifestyle which includes not smoking, maintaining a healthy
weight, following a healthy diet, and daily exercise can reduce the risk of having a stroke by
about one half.
DASH diet. The DASH (Dietary Approaches to Stop Hypertension) diet is high in fruits and
vegetables, moderate in low-fat dairy products, and low in animal protein and can lower the risk
of stroke.
Stroke risk screenings. Stroke risk screenings are an ideal opportunity to lower stroke risk by
identifying people or groups of people who are at high risk for stroke.
Education. Patients and the community must be educated about recognition and prevention of
stroke.
Low-dose aspirin.
Nursing Management
During the acute phase, a neurologic flow sheet is maintained to provide data about the
following important measures of the patient’s clinical status:
Change in level of consciousness or responsiveness.
Presence or absence of voluntary or involuntary movements of extremities.
Stiffness or flaccidity of the neck.
Eye opening, comparative size of pupils, and pupillary reaction to light.
Color of the face and extremities; temperature and moisture of the skin.
Ability to speak.
Presence of bleeding.
Maintenance of blood pressure.
During the postacute phase, assess the following functions:
Mental status (memory, attention span, perception, orientation, affect, speech/language).
Sensation and perception (usually the patient have decreased awareness of pain and
temperature).
Motor control (upper and lower extremity movement); swallowing ability, nutritional and
hydration status, skin integrity, activity tolerance, and bowel and bladder function.
Continue focusing nursing assessment on impairment of function in patient’s daily activities.
DEMOGRAPHIC DATA
Name: Patient AC
Sex: Female
Age: 84
Birthdate: July 5, 1939
Address: Calao east, Santiago City
Civil status: Widowed
Nationality: Filipino
Religion: Born Again
Occupation: Retired teacher
Educational Attainment: College Graduate
Admitting time: 9:14pm
Admitting date: April 8, 2024
Chief of complaints: Body Weakness and cough
Admitting diagnosis: TO CONSIDER CEREBROVASCULAR ACCIDENT
MEDICAL HISTORY
Past Medical History:
According to SO, patient AC hospitalized in year 2008 due to stroke. Since then, the
patient experienced right side paralysis. The SO also stated that patient AC has hypertension
with a maintenance of Losartan and she's also using over the counter drugs like biogesic for fever
or mild pain, Vitex Negundo L. for cough and neozep for flu. She didn't receive any COVID 19
vaccine. Had no known allergy to food and drugs.
Social History:
She lives with his son and granddaughter in-law at the rental house at Calao east. Her son
smoked every time he is at home. SO stated that she has a good relationship with them. The
patient has no vices.
Physical Assessment
General Survey: The patient AC is on semi fowler’s position wearing stripe shirt and silky
pajama. Patient is hemiplegia but she is awake and conscious with GCS of 11/15. NGT is
inserted at the left nostril, with nasal cannula and an IV line inserted on the dorsal metacarpal
gauge 22.
Vital Signs:
BP: 160/100 mmHg
Temp: 36.6°c
HR: 83 bpm
RR: 27cpm
O2: 99% via oxygen cannula
• No presence of
dandruff
No presence of
deformities and lumps
Palpation
FACE Inspection • Face is asymmetrical Patient is half
paralysis
• Right side of the face
is slightly drooping
when ask to smile
Palpation
EYES Inspection • The patient has ABNORMAL
blurry vision but not -Due to aging
using any glasses
• No discoloration in
sclera
NORMAL
• Bulbar conjunctiva -
no swelling
• Conjunctiva is
palpebral
• No presence swelling
and lesions
• Has minimal
discharge
• No discharge
• No presence of nasal
flaring
• No tenderness or
deformity upon
palpation
• No presence of NORMAL
Palpation edema
BOWEL
Patient defecate once a day, with
a soft like stool and usually
green and brown in color. The
SO stated the patient has no
difficulty while defecating.
ACTIVITY The patient can’t walk very well During hospitalization, the
EXERCISE because of her first stroke but patient’s usual activity is
according to the SO the patient is laying down on the bed
sitting or lying down while and resting.
listening or watching TV.
SLEEP REST The SO stated that the patient Since the patient's
usually slept at 8-9 pm and hospitalization, the patient
wakes up at 4-5 am to pray and always sleeps but
takes a nap at least 1hr in the sometimes wake up due to
afternoon. hospital disturbance.
COGNITIVE: COGNITIVE
SENSORY AND The patient is a secondary retired During hospitalization the
COGNITIVE teacher. She is able to speak patient can’t speak, but
PERCEPTUAL fluently in Filipino, and she is understand our question by
able to understand English. nodding as a respond.
SENSORY: SENSORY
The patient does not have any The patient right side of
issues with her hearing, feeling the body has a loss of
(touch), and smelling, tasting feeling or sensation.
and but her vision is Blurry due
to aging.
SELF NA NA
PERCEPTION/
SELF CONCEPT
ROLE According to the SO the patient NA
RELATIONSHIP has a good relationship with her
family and her neighbors. The
patient currently lives with her
children and grandchildren at
Calao east. She is kind and
talkative and prefer to be with
many people, and always order
her grandchildren to do laundry
and other household choirs.
SEXUALITY - NA NA
REPRODUCTIVE
COPING/ STRESS According to SO she noticed that During hospitalization,
TOLERANCE whenever the patient has not in The SO always place her
good mood of stress due to her cellphone on the bedside
condition the patient watch TV table while it’s playing a
and listen to radio and Christian Christian song
songs to relieve her stress.
VALUES/ BELIEF The patient is Born Again. The patient loves to listen
According to SO, the patient Christian Songs.
prays every morning and doesn’t
believe in “albularyo”
DATE: 04/08/2024
TIME: 9:57 PM
HEMATOLOGY
DATE: 04/08/224
TIME: 10:00 PM
CLINICAL CHEMISTRY
TEST RESULT REFERENCE INTERPRETATION
VALUES
Creatinine 105. 65 53.0-97.0 ABNORMAL
HIGH
May indicate underlying
kidney dysfunction or
poor kidney function
Blood Uric 10.12 2.60- 6.00 ABNORMAL
Acid HIGH
May indicate an
increased risk for
cardiovascular events
and adverse outcomes.
and may be a marker of
underlying
cardiovascular risk
factors
Sodium 141.12 135.0-150.0 NORMAL
Potassium 4.72 3.50-5.50 NORMAL
SGOT (AST) 20.19 0.00- 31.0 NORMAL
SGPT (ALT) 16.40 0.00-34.0 NORMAL
DATE: 04/10/224
TIME: 6:45 AM
CLINICAL CHEMISTRY
TEST RESULT REFERENCE INTERPRETATION
VALUES
FBS 275.54 74.00 – 100.00 mg/dl ABNORMAL
HIGH
May indicate
uncontrolled diabetes
Total 105.73 0.00 – 200.00 mg/dl NORMAL
Cholesterol
Triglycerides 81.46 0.00 – 150.0 mg/dl NORMAL
HDL- 29.30 42.00 – 88.00 mg/dl ABNORMAL
Cholesterol LOW
There is a higher
likelihood of plaque
formation, leading to
narrowed and hardened
arteries
LDL-Cholesterol 60.14 0.00 – 150.00 mg/dl NORMAL
VLDL 16.29 NORMAL
DATE: 04/10/224
TIME: 12:36 AM
URINALYSIS
ROUTINE:
Color Yellow
Transparency Clear
Specific Gravity 1.015
pH 5.0
Protein Trace
Glucose Negative
WBC 2-4/HPF
RBC 1-3/HPF
Epithelial Cells Rare
Amorphous Urates Occasional
Mucus Threads Rare
Bacteria Rare
Anatomy and Physiology
Brainstem: acts as a relay center connecting the cerebrum and cerebellum to the spinal cord. It
performs many automatic functions such as breathing, heart rate, body temperature, wake and
sleep cycles, digestion, sneezing, coughing, vomiting, and swallowing.
The cerebrum is divided into two halves: the right and left hemispheres. They are joined by a
bundle of fibers called the corpus callosum that transmits messages from one side to the other.
Each hemisphere controls the opposite side of the body. If a stroke occurs on the right side of the
brain, your left arm or leg may be weak or paralyzed.
Not all functions of the hemispheres are shared. In general, the left hemisphere controls speech,
comprehension, arithmetic, and writing. The right hemisphere controls creativity, spatial ability,
artistic, and musical skills. The left hemisphere is dominant in hand use and language in about
92% of people.
Frontal lobe
Parietal lobe
Occipital lobe
Temporal lobe
Wernicke's area: lies in the left temporal lobe. Damage to this area causes Wernicke's aphasia.
The individual may speak in long sentences that have no meaning, add unnecessary words, and
even create new words. They can make speech sounds, however they have difficulty
understanding speech and are therefore unaware of their mistakes.
Cortex
The surface of the cerebrum is called the cortex. It has a folded appearance with hills and
valleys. The cortex contains 16 billion neurons (the cerebellum has 70 billion = 86 billion total)
that are arranged in specific layers. The nerve cell bodies color the cortex grey-brown giving it
its name – gray matter. Beneath the cortex are long nerve fibers (axons) that connect brain areas
to each other — called white matter.
DISCHARGE PLAN