TABLE 1:
PROTOZOANS SIZE NUMBER KARYOSOME PERIPHERAL CYTOPLAS
spp. “CYSTS” OF CHROMATIN M AND
NUCLEI INCLUSION
15-60 4 small fine, uniform, RBC
1. E. histolytica µm and evenly
distributed
-2.0 µm 2-4 large no peripheral insulin
2. E. coli chromatin proteins
5-10 µm 4 small, compact, uniform cyst
3. E. hartmanii centrally or
eccentrically
6–12 µm 4 large karyosome no peripheral present
4. E. nana chromatin
20-150 1 prominent clumped finely
5. E. gingivalis µm central nucleus granular
9 - 15μ 1 large coarse, RBC
6. I. butschlii granular
13-17 1 large coarse, clumped bright to dark
7. B. hominis mm uneven red
8-15 µm 1 large, dense lack numerous
8. Naegleria fowleri
15 to 25 1 large no peripheral prominent
9. Acanthamoeba spp. μm chromatin contractile
vacuole
50-70 μm 2 small, central granule ciliated
10. B. coli
PROTOZOANS SIZE MOTILITY KARYOSOME PERIPHERAL CYTOPLASM PATHOGENECITY U
spp. CHROMATIN & F
“TROPHOZOITE INCLUSION
”
15-60 sluggish small fine, RBC pathogenic ame
1. E. µm uniform, and ba la
histolytica evenly
distributed
50 µm sluggish large no peripheral insulin not
2. E. coli chromatin proteins considered path e
ogenic ka
15 µm less rapid small, uniform cyst not
3. E. hartmanii compact considered path w
ogenic t
p
6–12 sluggish large no peripheral present non-pathogenic g
4. E. nana µm karyosome chromatin amoeba
10-20 pseudopod prominent clumped finely not
5. E. µm central granular considered path k
gingivalis nucleus ogenic
8-20 sluggish large coarse, RBC not
6. I. butschlii µm granular considered path s
ogenic
n
50-70 Rotary, large coarse, coarse, unclear pathoge p
7. B. hominis mm boring clumped clumped nic te
uneven uneven
10-35 lobopodia large, dense lack lack pathogenic D
8. Naegleria µm l
fowleri
15 to cytoskeleto large no peripheral no pathogenic
9. 25 μm n-based chromatin peripheral n
Acanthamoeba chromatin
spp.
50-100 “like a small, granule granule opportunistic pat
10. B. coli μm thrown central hogen la
football”
TABLE 2:
REFERENCES:
Centers for Disease Control and Prevention
Laboratory Diagnosis of Amebiasis by Mehmet Tanyuksel, William A. Petri Jr.
Global Health, Division of Parasitic Diseases and Malaria