DENTIN HYPERSENSITIVITY
It is characterised by short, sharp pain arising from exposed dentin in response to stimuli typically
thermal,evaporative, tactile, osmotic or chemical and which cannot be ascribed to any other form of
dental defect or pathology.
ETIOLOGY:
ENAMEL LOSS
Occlusal wear
Tooth brush abrasion
Dietary erosion
Abfraction
Parafunctional habits
CEMENTAL LOSS
Gingival recession
Periodontal disease
Root planing
Periodontal surgery
CLINICAL FEATURES:
Pain is the primary symptom.
Short, sharp pain in response to heat, cold, tactile stimuli, sweet or sour foods.
Pain is only felt on application of external stimulus.
There is no lingering discomfort once the stimulus is removed.
DIFFERENTIAL DIAGNOSIS:
Chipped teeth
Fractured restoration
Restorative treatments
Dental caries
Cracked tooth syndrome
THEORIES FOR DENTINAL HYPERSENSITIVITY
Odontoblastic transduction theory
Neural theory
hydrodynamic theory
MANAGEMENT
1.Desensitization by occluding dentinal tubules.
a) Formation of a smear layer over exposed dentin
b) Use of topical agents to occlude the exposed tubules
. Calcium hydroxide paste
. Calcium phosphate paste
. Silver nitrate
. Strontium chloride
. Fluorides
. Fluoride iontophoresis
. Potassium oxalate
. Varnishes
. Dentin adhesives
c) Placement of restorations
. Glass ionomer cements
. Composite resins
d) Use of lasers
. Carbon dioxide laser
. Nd:YAG, Er:YAG lasers
. He:Ne laser
2. Desensitization by blocking pulpal sensory nerves
Potassium nitrate toothpastes
SUBMITTED BY: SIMARPREET, SONIA, SHINA, SIMRON (INTERNS)