Chronic mineralocorticoid (MC ) excess, whether due to elevated plasma aldosterone (ALDO) or deox... more Chronic mineralocorticoid (MC ) excess, whether due to elevated plasma aldosterone (ALDO) or deoxycorticosterone (DOC), is associated with a perivascular fibrosis of systemic and coronary arterioles. This remodeling of resistance vessels contributes to the appearance of hypertension. Chronic MC excess is also accompanied by cardiac myocyte necrosis, secondary to myocardial potassium depletion, and a subsequent reparative fibrosis that appears in the normotensive, nonhypertrophied right and hypertensive, hypertrophied left ventricles. Fibrosis contributes to the appearance of ventricular arrhythmias and dysfunction. Herein, clinical and experimental evidence linking chronic, inappropriate (relative to dietary sodium) elevations in circulating ALDO and DOC with these reactive and reparative forms of fibrous tissue formation in the heart and other tissues is presented. (Steroids 60:125-132, 1995)
Web Intelligence and Agent Systems: An International Journal, 2005
... 19. Manuel J. Maña López , Manuel de Buenaga Rodríguez , José María Gómez Hidalgo, Using and ... more ... 19. Manuel J. Maña López , Manuel de Buenaga Rodríguez , José María Gómez Hidalgo, Using and Evaluating User Directed ... 24. Alberto Díaz Esteban , Pablo Gervás Gómez-Navarro , Antonio García Jiménez, Evaluating a User-Model Based Personalisation Architecture for ...
Web Intelligence and Agent Systems: An International Journal, 2005
... 19. Manuel J. Maña López , Manuel de Buenaga Rodríguez , José María Gómez Hidalgo, Using and ... more ... 19. Manuel J. Maña López , Manuel de Buenaga Rodríguez , José María Gómez Hidalgo, Using and Evaluating User Directed ... 24. Alberto Díaz Esteban , Pablo Gervás Gómez-Navarro , Antonio García Jiménez, Evaluating a User-Model Based Personalisation Architecture for ...
Chronic mineralocorticoid (MC ) excess, whether due to elevated plasma aldosterone (ALDO) or deox... more Chronic mineralocorticoid (MC ) excess, whether due to elevated plasma aldosterone (ALDO) or deoxycorticosterone (DOC), is associated with a perivascular fibrosis of systemic and coronary arterioles. This remodeling of resistance vessels contributes to the appearance of hypertension. Chronic MC excess is also accompanied by cardiac myocyte necrosis, secondary to myocardial potassium depletion, and a subsequent reparative fibrosis that appears in the normotensive, nonhypertrophied right and hypertensive, hypertrophied left ventricles. Fibrosis contributes to the appearance of ventricular arrhythmias and dysfunction. Herein, clinical and experimental evidence linking chronic, inappropriate (relative to dietary sodium) elevations in circulating ALDO and DOC with these reactive and reparative forms of fibrous tissue formation in the heart and other tissues is presented. (Steroids 60:125-132, 1995)
Web Intelligence and Agent Systems: An International Journal, 2005
... 19. Manuel J. Maña López , Manuel de Buenaga Rodríguez , José María Gómez Hidalgo, Using and ... more ... 19. Manuel J. Maña López , Manuel de Buenaga Rodríguez , José María Gómez Hidalgo, Using and Evaluating User Directed ... 24. Alberto Díaz Esteban , Pablo Gervás Gómez-Navarro , Antonio García Jiménez, Evaluating a User-Model Based Personalisation Architecture for ...
Web Intelligence and Agent Systems: An International Journal, 2005
... 19. Manuel J. Maña López , Manuel de Buenaga Rodríguez , José María Gómez Hidalgo, Using and ... more ... 19. Manuel J. Maña López , Manuel de Buenaga Rodríguez , José María Gómez Hidalgo, Using and Evaluating User Directed ... 24. Alberto Díaz Esteban , Pablo Gervás Gómez-Navarro , Antonio García Jiménez, Evaluating a User-Model Based Personalisation Architecture for ...
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